Myeloid cell-derived hypoxia-inducible factor attenuates inflammation in unilateral ureteral obstruction-induced kidney injury - Wikidata - awgldk - TriplyDB

Myeloid cell-derived hypoxia-inducible factor attenuates inflammation in unilateral ureteral obstruction-induced kidney injury

Myeloid cell-derived hypoxia-inducible factor attenuates inflammation in unilateral ureteral obstruction-induced kidney injury

http://www.wikidata.org/entity/Q35936151

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Recent advances in understanding of chronic kidney disease Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes Molecular mechanisms of ischemic preconditioning in the kidney Silencing of hypoxia-inducible factor-1α gene attenuates chronic ischemic renal injury in two-kidney, one-clip rats.Hypoxia, HIF, and Associated Signaling Networks in Chronic Kidney Disease.Renal Oxygenation in the Pathophysiology of Chronic Kidney Disease.Endothelial HIF-2 mediates protection and recovery from ischemic kidney injury.Prohibitin attenuates oxidative stress and extracellular matrix accumulation in renal interstitial fibrosis disease.Inflammation and hypoxia linked to renal injury by CCAAT/enhancer-binding protein δ.Inflammation and hypoxia in the kidney: friends or foes?The transcription of the alarmin cytokine interleukin-1 alpha is controlled by hypoxia inducible factors 1 and 2 alpha in hypoxic cells TGF-β/Smad3 activates mammalian target of rapamycin complex-1 to promote collagen production by increasing HIF-1α expression.Hypoxia inducible factors are dispensable for myeloid cell migration into the inflamed mouse eye.Renal erythropoietin-producing cells in health and disease.Renal injury is accelerated by global hypoxia-inducible factor 1 alpha deficiency in a mouse model of STZ-induced diabetes.HIF prolyl hydroxylase inhibitors for the treatment of renal anaemia and beyond.Expanding roles of the hypoxia-response network in chronic kidney disease.Hypoxia and Dysregulated Angiogenesis in Kidney Disease.Hypoxia-inducible factors: key regulators of myeloid cells during inflammation.HIF-mediated innate immune responses: cell signaling and therapeutic implications.Renal epithelium regulates erythropoiesis via HIF-dependent suppression of erythropoietin.Distinct subpopulations of FOXD1 stroma-derived cells regulate renal erythropoietin.Hypoxia-inducible factor-1α promotes glomerulosclerosis and regulates COL1A2 expression through interactions with Smad3.Erythropoietin Synthesis in Renal Myofibroblasts Is Restored by Activation of Hypoxia Signaling.Deletion of phd2 in myeloid lineage attenuates hypertensive cardiovascular remodeling.Prohibitin is associated with antioxidative protection in hypoxia/reoxygenation-induced renal tubular epithelial cell injury.Cyclosporin a induces renal episodic hypoxia.Association of prohibitin-1 and 2 with oxidative stress in rats with renal interstitial fibrosis.Hypoxia Induced Factor in Chronic Kidney Disease: Friend or Foe?Summary of the 2018 ISN Frontiers Meeting: Kidney Disease and Cardiovascular Disease.

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Myeloid cell-derived hypoxia-inducible factor attenuates inflammation in unilateral ureteral obstruction-induced kidney injury

http://www.wikidata.org/entity/Q35936151

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2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

@zh-tw

2012年论文

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2012年论文

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2012年论文

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name

Myeloid cell-derived hypoxia-i ...... truction-induced kidney injury

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Myeloid cell-derived hypoxia-i ...... truction-induced kidney injury

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type

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Myeloid cell-derived hypoxia-i ...... truction-induced kidney injury

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Myeloid cell-derived hypoxia-i ...... truction-induced kidney injury

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Myeloid cell-derived hypoxia-i ...... truction-induced kidney injury

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JIMMUNOL.1103377

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Journal of Immunology

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Myeloid cell-derived hypoxia-i ...... truction-induced kidney injury

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Detlef Schlöndorff

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Hanako Kobayashi

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Pinelopi P Kapitsinou

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Qingdu Liu

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Travis L Unger

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Victoria Gilbert

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P2860

Hypoxia promotes fibrogenesis in vivo via HIF-1 stimulation of epithelial-to-mesenchymal transition

FoxA1 translates epigenetic signatures into enhancer-driven lineage-specific transcription

Galectin-3 expression and secretion links macrophages to the promotion of renal fibrosis

Generalized lacZ expression with the ROSA26 Cre reporter strain

HIF-1: mediator of physiological and pathophysiological responses to hypoxia

Regulation of immune cells by local-tissue oxygen tension: HIF1 alpha and adenosine receptors

Deletion of the developmentally essential gene ATR in adult mice leads to age-related phenotypes and stem cell loss

Hypoxia-inducible factors in the kidney

Abnormal B lymphocyte development and autoimmunity in hypoxia-inducible factor 1alpha -deficient chimeric mice.

A mouse model for beta 0-thalassemia

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5106-5115

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10.4049/JIMMUNOL.1103377

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Stefano Rivella

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