Inhibition of DPP-4 reduces acute mortality after myocardial infarction with restoration of autophagic response in type 2 diabetic rats.

Inhibition of DPP-4 reduces acute mortality after myocardial infarction with restoration of autophagic response in type 2 diabetic rats.

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http://www.wikidata.org/entity/Q35936441
The Role of Oxidative Stress in Myocardial Ischemia and Reperfusion Injury and Remodeling: RevisitedPrognostic value of plasma DPP4 activity in ST-elevation myocardial infarctionThe regulatory role of DPP4 in atherosclerotic disease.The impact of DPP-4 inhibitors on long-term survival among diabetic patients after first acute myocardial infarctionSuppressed autophagic response underlies augmentation of renal ischemia/reperfusion injury by type 2 diabetes.Inhibition of dynamin-related protein 1 protects against myocardial ischemia-reperfusion injury in diabetic mice.SGLT-2 Inhibition with Dapagliflozin Reduces the Activation of the Nlrp3/ASC Inflammasome and Attenuates the Development of Diabetic Cardiomyopathy in Mice with Type 2 Diabetes. Further Augmentation of the Effects with Saxagliptin, a DPP4 Inhibitor.DPP-4 inhibitors in diabetic complications: role of DPP-4 beyond glucose control.Alterations of voltage-dependent K(+) channels in the mesenteric artery during the early and chronic phases of diabetes.Concise Review: Challenges in Regenerating the Diabetic Heart: A Comprehensive Review.The effects of vildagliptin compared with metformin on vascular endothelial function and metabolic parameters: a randomized, controlled trial (Sapporo Athero-Incretin Study 3).Sitagliptin-mediated preservation of endothelial progenitor cell function via augmenting autophagy enhances ischaemic angiogenesis in diabetes.Restoring diabetes-induced autophagic flux arrest in ischemic/reperfused heart by ADIPOR (adiponectin receptor) activation involves both AMPK-dependent and AMPK-independent signaling.Diabetes increases the susceptibility to acute kidney injury after myocardial infarction through augmented activation of renal Toll-like receptors in rats.Empagliflozin normalizes the size and number of mitochondria and prevents reduction in mitochondrial size after myocardial infarction in diabetic hearts.Combination of Vildagliptin and Ischemic Postconditioning in Diabetic Hearts as a Working Strategy to Reduce Myocardial Reperfusion Injury by Restoring Mitochondrial Function and Autophagic Activity.
P2860

Inhibition of DPP-4 reduces acute mortality after myocardial infarction with restoration of autophagic response in type 2 diabetic rats.

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http://www.wikidata.org/entity/Q35936441
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2015 nî lūn-bûn
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2015年の論文
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2015年論文
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2015年論文
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2015年論文
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2015年論文
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name
Inhibition of DPP-4 reduces ac ...... ponse in type 2 diabetic rats.
@ast
Inhibition of DPP-4 reduces ac ...... ponse in type 2 diabetic rats.
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type
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Inhibition of DPP-4 reduces ac ...... ponse in type 2 diabetic rats.
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Inhibition of DPP-4 reduces ac ...... ponse in type 2 diabetic rats.
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Inhibition of DPP-4 reduces ac ...... ponse in type 2 diabetic rats.
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Inhibition of DPP-4 reduces ac ...... ponse in type 2 diabetic rats.
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P1476
P2093
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P2888
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P5875
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P1476
Inhibition of DPP-4 reduces ac ...... sponse in type 2 diabetic rats
@en
P2093
P2860
P2888
P304
P31
P356
P407
P478
P50
P577
P5875
P6179
P698
P921
P932