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New Developments in Chronic Myeloid Leukemia: Implications for TherapyChronic myeloid leukemia: mechanisms of blastic transformationThe elusive chronic myeloid leukemia stem cell: does it matter and how do we eliminate it?Tailoring tyrosine kinase inhibitors to fit the lung cancer genome.Immune modulation of minimal residual disease in early chronic phase chronic myelogenous leukemia: a randomized trial of frontline high-dose imatinib mesylate with or without pegylated interferon alpha-2b and granulocyte-macrophage colony-stimulatinTargeting multiple kinase pathways in leukemic progenitors and stem cells is essential for improved treatment of Ph+ leukemia in mice.Stem cell and kinase activity-independent pathway in resistance of leukaemia to BCR-ABL kinase inhibitorsBcr-abl signals to desensitize chronic myeloid leukemia cells to IFNα via accelerating the degradation of its receptorChronic myeloid leukemia stem cells in the era of targeted therapies: resistance, persistence and long-term dormancy.CD34(+)/CD38(-) stem cells in chronic myeloid leukemia express Siglec-3 (CD33) and are responsive to the CD33-targeting drug gemtuzumab/ozogamicinImmunotherapy in multiple myeloma--possibility or probability?FoxO tumor suppressors and BCR-ABL-induced leukemia: a matter of evasion of apoptosis.Target for cancer therapy: proliferating cells or stem cells.Tetrandrine citrate eliminates imatinib-resistant chronic myeloid leukemia cells in vitro and in vivo by inhibiting Bcr-Abl/β-catenin axis.Synergistic effects of p53 activation via MDM2 inhibition in combination with inhibition of Bcl-2 or Bcr-Abl in CD34+ proliferating and quiescent chronic myeloid leukemia blast crisis cells.Emerging drugs in chronic myelogenous leukaemia.Immunotherapy of multiple myeloma: the start of a long and tortuous journey.Small-molecule inhibition of BRD4 as a new potent approach to eliminate leukemic stem- and progenitor cells in acute myeloid leukemia AML.BCR/ABL, DNA damage and DNA repair: implications for new treatment concepts.Immunotherapeutic approaches in chronic myelogenous leukemia.The development of novel immunotherapeutic approaches in multiple myeloma.Triptolide induces cell death independent of cellular responses to imatinib in blast crisis chronic myelogenous leukemia cells including quiescent CD34+ primitive progenitor cells.Combined targeting of BCL-2 and BCR-ABL tyrosine kinase eradicates chronic myeloid leukemia stem cells.Understanding the cancer stem cell.Chronic myeloid leukemia cells refractory/resistant to tyrosine kinase inhibitors are genetically unstable and may cause relapse and malignant progression to the terminal disease state.Mechanisms controlling hematopoietic stem cell functions during normal hematopoiesis and hematological malignancies.Stem cells, quiescence and rectal carcinoma: an unexplored relationship and potential therapeutic target.Genetic mechanisms of chronic myeloid leukemia blastic transformation.Mechanisms and novel approaches in overriding tyrosine kinase inhibitor resistance in chronic myeloid leukemia.Kinase-inhibitor-insensitive cancer stem cells in chronic myeloid leukemia.The hemopoietic stem cell niche versus the microenvironment of the multiple myeloma-tumor initiating cell.Isolation of colorectal cancer stem-like cells.The BCR-ABL/NF-κB signal transduction network: a long lasting relationship in Philadelphia positive LeukemiasBCR-ABL1 tyrosine kinase sustained MECOM expression in chronic myeloid leukaemia.The bone marrow microenvironment as niche retreats for hematopoietic and leukemic stem cells.Imatinib increases cytotoxicity of melphalan and their combination allows an efficient killing of chronic myeloid leukemia cells.Identification of CD133(-)/telomerase(low) progenitor cells in glioblastoma-derived cancer stem cell lines.Inhibition of stromal cell-derived factor-1/CXCR4 axis for the cure of BCR-ABL positive chronic myeloid leukemia.Nilotinib concentration in cell lines and primary CD34(+) chronic myeloid leukemia cells is not mediated by active uptake or efflux by major drug transporters.BCR-ABL1-induced expression of HSPA8 promotes cell survival in chronic myeloid leukaemia.
P2860
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P2860
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Punish the parent not the progeny.
@ast
Punish the parent not the progeny.
@en
type
label
Punish the parent not the progeny.
@ast
Punish the parent not the progeny.
@en
prefLabel
Punish the parent not the progeny.
@ast
Punish the parent not the progeny.
@en
P50
P1433
P1476
Punish the parent not the progeny
@en
P2093
Lucy J Elrick
P304
P356
10.1182/BLOOD-2004-08-3373
P407
P577
2004-11-04T00:00:00Z