about
Cytokines as key players in the pathophysiology of preeclampsiaImmunosuppression improves blood pressure and endothelial function in a rat model of pregnancy-induced hypertensionCluster analysis of placental inflammatory proteins can distinguish preeclampsia from preterm labor and premature membrane rupture in singleton deliveries less than 28 weeks of gestationPlacental expression profiling in preeclampsia: local overproduction of hemoglobin may drive pathological changes.Circulating cytokines, chemokines and adhesion molecules in normal pregnancy and preeclampsia determined by multiplex suspension arrayMaternal plasma soluble TRAIL is decreased in preeclampsia.Autoantibody-mediated complement C3a receptor activation contributes to the pathogenesis of preeclampsia.Plasma concentrations of angiogenic/anti-angiogenic factors have prognostic value in women presenting with suspected preeclampsia to the obstetrical triage area: a prospective study.Elevated maternal C-reactive protein and increased risk of schizophrenia in a national birth cohort.The possible role of extravillous trophoblast-derived exosomes on the uterine spiral arterial remodeling under both normal and pathological conditionsTrophoblast expression of the minor histocompatibility antigen HA-1 is regulated by oxygen and is increased in placentas from preeclamptic women.Maternal gene expression profiling during pregnancy and preeclampsia in human peripheral blood mononuclear cellsEndocan, a putative endothelial cell marker, is elevated in preeclampsia, decreased in acute pyelonephritis, and unchanged in other obstetrical syndromesChronic air pollution exposure during pregnancy and maternal and fetal C-reactive protein levels: the Generation R Study.Nicotine inhibits cytokine production by placenta cells via NFkappaB: potential role in pregnancy-induced hypertension.Moderate Exercise Attenuates Lipopolysaccharide-Induced Inflammation and Associated Maternal and Fetal Morbidities in Pregnant Rats.Diabetic angiopathy and angiogenic defects.Quantification of preeclampsia-related microRNAs in maternal serum.Elevated maternal C-reactive protein and autism in a national birth cohortA combination of single nucleotide polymorphisms in the 3'untranslated region of HLA-G is associated with preeclampsia.Pathogenesis and promising non-invasive markers for preeclampsiaCirculating C19MC microRNAs in preeclampsia, gestational hypertension, and fetal growth restriction.Evidence of sexual dimorphism in the placental function with severe preeclampsia.Circulating heat shock protein mRNA profile in gestational hypertension, pre-eclampsia & foetal growth restriction.Lower Serum Vitamin D Metabolite Levels in Relation to Circulating Cytokines/Chemokines and Metabolic Hormones in Pregnant Women with Hypertensive Disorders.TREM-1 expression is increased in human placentas from severe early-onset preeclamptic pregnancies where it may be involved in syncytializationPreeclampsia is associated with decreased serum alpha(2)-HS glycoprotein (fetuin-A) concentration.Leukocytosis might precede in-hospital eclampsia in preeclamptic women who do not receive magnesium sulfate.The profiles of soluble adhesion molecules in the "great obstetrical syndromes".Placental inflammation in pre-eclampsia by Nod-like receptor protein (NLRP)3 inflammasome activation in trophoblasts.Mining the human tissue proteome for protein citrullination.Maternal plasma fetuin-A concentration is lower in patients who subsequently developed preterm preeclampsia than in uncomplicated pregnancy: a longitudinal study.The direct and sustained consequences of severe placental hypoxia on vascular contractilityP-Glycoprotein (P-gp)/ABCB1 plays a functional role in extravillous trophoblast (EVT) invasion and is decreased in the pre-eclamptic placenta
P2860
Q26830707-F6651B62-8D3A-4C13-9E98-DB1568342737Q28582225-409B1978-1CA8-4607-9341-161E458961D3Q30503917-428A93FE-2E87-4A6F-A9C6-9423C26AF9AFQ33312878-A4DE47D0-B326-4B69-8336-8003D4F38341Q33761815-80C6BB8D-11C7-4F09-88DA-8A87B67BAFD6Q33909572-FF088CE4-F3B8-4855-A957-E1CC226C688BQ34040357-172C5998-8383-41C6-9696-0B958875CB6BQ34119026-7938A36C-6005-40B7-8E13-B6D46C251A60Q34155481-5AE42846-CD37-47E8-89F3-273104E94A24Q34267611-1CCE957D-EA8B-4101-B3F2-73A4BB1D6907Q34481622-3CCEFAEB-5E79-414B-9EAB-F4A03FB190E2Q34582266-0BF5B4BD-765F-42BF-B435-AE927F45A7E6Q35547367-13904AD9-99C8-4D69-99D8-115D1F743827Q35941841-0637DA1A-E18B-417F-AA09-2D5AB9609F62Q35995193-2FA697D9-BAFF-42B8-A3E9-0D823BB9F743Q36002112-E0383781-5696-4651-A2E1-D807999E908FQ36301365-E3A46136-F143-4EDF-B0D7-B26FB00A5EB8Q36321403-00BEA124-F9F4-4922-9FB9-5933ECFC8531Q36787986-4F595AB1-098F-4711-8A01-30F4FB82E39CQ37075173-70489AE0-C12D-4ACC-ABFA-1F26C3D3A52CQ37199922-B179BB99-8503-460A-85AE-22EFAF7E76ABQ37358895-A8237F28-D5FD-4241-A094-4A50BCDD97A1Q37404418-8E425810-C398-4166-B621-45098D0F235EQ37550896-072EB892-5C37-4883-9432-6D46555B4F6AQ37694496-F60162C5-F831-416B-973D-2E5DD64AB6F0Q37699280-7ED3EE0B-8A87-4697-BF0C-B86BE3EC5B6BQ47699767-A3AAD45B-7570-455E-A146-109CCA22EEAAQ48664058-B8D3555F-8FE4-4B78-8906-DD4A9DB2B60DQ50102668-837E832F-5B3A-44B6-91F4-B8F8EAC7B3BEQ52312934-98F8DF68-0F14-44B6-9958-22C67CE78824Q52611944-CDB99339-6362-47B1-8713-EF32A0BA8612Q55004295-FAF4F805-3037-4009-85B5-3786830E25C0Q58708934-9A9A6592-2E50-4AA8-B84A-FEC99F56D40EQ58762258-BB5394E6-B56E-4BD3-A185-C09307E87779
P2860
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Preeclampsia and the systemic inflammatory response.
@ast
Preeclampsia and the systemic inflammatory response.
@en
type
label
Preeclampsia and the systemic inflammatory response.
@ast
Preeclampsia and the systemic inflammatory response.
@en
prefLabel
Preeclampsia and the systemic inflammatory response.
@ast
Preeclampsia and the systemic inflammatory response.
@en
P1476
Preeclampsia and the systemic inflammatory response.
@en
P2093
Christopher W G Redman
Ian L Sargent
P304
P356
10.1016/S0270-9295(04)00127-5
P577
2004-11-01T00:00:00Z