EGF prevents the neuroendocrine differentiation of LNCaP cells induced by serum deprivation: the modulator role of PI3K/Akt - Wikidata - awgldk - TriplyDB

EGF prevents the neuroendocrine differentiation of LNCaP cells induced by serum deprivation: the modulator role of PI3K/Akt

EGF prevents the neuroendocrine differentiation of LNCaP cells induced by serum deprivation: the modulator role of PI3K/Akt

http://www.wikidata.org/entity/Q35945055

about

Knockdown of protein tyrosine phosphatase SHP-1 inhibits G1/S progression in prostate cancer cells through the regulation of components of the cell-cycle machinery Neuroendocrine differentiation in prostate cancer: a mechanism of radioresistance and treatment failure Neoplasia: the second decade.In vitro model of metastasis to bone marrow mediates prostate cancer castration resistant growth through paracrine and extracellular matrix factors.Highly efficient capture and enumeration of low abundance prostate cancer cells using prostate-specific membrane antigen aptamers immobilized to a polymeric microfluidic device.Altered fibroblast growth factor receptor 4 stability promotes prostate cancer progression.The War on Cancer rages on.IGFBP3 and MAPK/ERK signaling mediates melatonin-induced antitumor activity in prostate cancer.FOXA1 inhibits prostate cancer neuroendocrine differentiation.EGF promotes neuroendocrine-like differentiation of prostate cancer cells in the presence of LY294002 through increased ErbB2 expression independent of the phosphatidylinositol 3-kinase-AKT pathway.Regulation of neuroendocrine differentiation by AKT/hnRNPK/AR/β-catenin signaling in prostate cancer cells.Osteoblast-induced EGFR/ERBB2 signaling in androgen-sensitive prostate carcinoma cells characterized by multiplex kinase activity profiling.Alterations of androgen receptor-regulated enhancer RNAs (eRNAs) contribute to enzalutamide resistance in castration-resistant prostate cancer.Induction of Neuroendocrine Differentiation in Prostate Cancer Cells by Dovitinib (TKI-258) and its Therapeutic Implications.Involvement of Src in the Adaptation of Cancer Cells under Microenvironmental Stresses.SRPK1 contributes to malignancy of hepatocellular carcinoma through a possible mechanism involving PI3K/Akt.Neoplasia: An Anniversary of Progress.Combined analysis of EGF+61G>A and TGFB1+869T>C functional polymorphisms in the time to androgen independence and prostate cancer susceptibility.MicroRNA-652 induces NED in LNCaP and EMT in PC3 prostate cancer cells.

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P2860

EGF prevents the neuroendocrine differentiation of LNCaP cells induced by serum deprivation: the modulator role of PI3K/Akt

http://www.wikidata.org/entity/Q35945055

description

2007 nî lūn-bûn

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2007年の論文

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2007年論文

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2007年論文

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2007年論文

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2007年論文

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2007年论文

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name

EGF prevents the neuroendocrin ...... the modulator role of PI3K/Akt

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EGF prevents the neuroendocrin ...... the modulator role of PI3K/Akt

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type

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EGF prevents the neuroendocrin ...... the modulator role of PI3K/Akt

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EGF prevents the neuroendocrin ...... the modulator role of PI3K/Akt

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EGF prevents the neuroendocrin ...... the modulator role of PI3K/Akt

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EGF prevents the neuroendocrin ...... the modulator role of PI3K/Akt

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EGF prevents the neuroendocrin ...... the modulator role of PI3K/Akt

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Begoña Colás

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Carmén Almaraz-Pro

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F Javier Rodríguez-Ubreva

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M Alicia Cortés

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Pilar López-Ruiz

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Rosa M Martín-Orozco

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Santiago Ropero

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P2860

From HER2/Neu signal cascade to androgen receptor and its coactivators: a novel pathway by induction of androgen target genes through MAP kinase in prostate cancer cells

Interleukin-6- and cyclic AMP-mediated signaling potentiates neuroendocrine differentiation of LNCaP prostate tumor cells.

PTEN activation contributes to tumor inhibition by trastuzumab, and loss of PTEN predicts trastuzumab resistance in patients.

Untangling the ErbB signalling network

The phosphatidylinositol 3'-kinase pathway is a dominant growth factor-activated cell survival pathway in LNCaP human prostate carcinoma cells

Heterodimerization of the epidermal-growth-factor (EGF) receptor and ErbB2 and the affinity of EGF binding are regulated by different mechanisms

The ErbB signaling network: receptor heterodimerization in development and cancer

ERBB receptors and cancer: the complexity of targeted inhibitors

Acquisition of neuroendocrine characteristics by prostate tumor cells is reversible: implications for prostate cancer progression.

Neuroendocrine differentiation in prostatic carcinoma.

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614-624

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10.1593/NEO.07337

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8

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9

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5945568

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17898861

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1950431

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