Identification of genetic variants contributing to cisplatin-induced cytotoxicity by use of a genomewide approach - Wikidata - awgldk - TriplyDB

Identification of genetic variants contributing to cisplatin-induced cytotoxicity by use of a genomewide approach

Identification of genetic variants contributing to cisplatin-induced cytotoxicity by use of a genomewide approach

http://www.wikidata.org/entity/Q35946327

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Genome-wide association study of d-amphetamine response in healthy volunteers identifies putative associations, including cadherin 13 (CDH13)Chemotherapeutic drug susceptibility associated SNPs are enriched in expression quantitative trait loci In vitro human cell line models to predict clinical response to anticancer drugs Co-administration of cisplatin and furosemide causes rapid and massive loss of cochlear hair cells in mice.Pharmacogenomic discovery using cell-based models Comprehensive survey of SNPs in the Affymetrix exon array using the 1000 Genomes dataset Genetic analysis of human traits in vitro: drug response and gene expression in lymphoblastoid cell lines Mixed effects modeling of proliferation rates in cell-based models: consequence for pharmacogenomics and cancer Protein quantitative trait loci identify novel candidates modulating cellular response to chemotherapy Exon-specific QTLs skew the inferred distribution of expression QTLs detected using gene expression array data Genome-wide association study of survival in non-small cell lung cancer patients receiving platinum-based chemotherapy Polymorphic variation in TPMT is the principal determinant of TPMT phenotype: A meta-analysis of three genome-wide association studies.Heat shock protein-mediated protection against Cisplatin-induced hair cell death.Integrative genomics: quantifying significance of phenotype-genotype relationships from multiple sources of high-throughput data ATHENA: a tool for meta-dimensional analysis applied to genotypes and gene expression data to predict HDL cholesterol levels Integrative "omic" analysis for tamoxifen sensitivity through cell based models.Concordant gene expression in leukemia cells and normal leukocytes is associated with germline cis-SNPs HapMap filter 1.0: a tool to preprocess the HapMap genotypic data for association studies An eQTL-based method identifies CTTN and ZMAT3 as pemetrexed susceptibility markers.Data-driven methods to discover molecular determinants of serious adverse drug events.Unsupervised assessment of microarray data quality using a Gaussian mixture model Knowledge-based data analysis comes of age.Beyond the HapMap Genotypic Data: Prospects of Deep Resequencing Projects Integrating cell-based and clinical genome-wide studies to identify genetic variants contributing to treatment failure in neuroblastoma patients Impact of the 1000 genomes project on the next wave of pharmacogenomic discovery Genome-wide local ancestry approach identifies genes and variants associated with chemotherapeutic susceptibility in African Americans.Integrating Epigenomics into Pharmacogenomic Studies Testicular cancer survivorship: research strategies and recommendations.Identification of quantitative trait loci underlying proteome variation in human lymphoblastoid cells.Integrating heterogeneous high-throughput data for meta-dimensional pharmacogenomics and disease-related studies Heritable and non-genetic factors as variables of pharmacologic phenotypes in lymphoblastoid cell lines.Meta-analysis on pharmacogenetics of platinum-based chemotherapy in non small cell lung cancer (NSCLC) patients.Theory on the coupled stochastic dynamics of transcription and splice-site recognition Genome-wide analysis correlates Ayurveda Prakriti.Cell-based Models for Discovery of Pharmacogenomic Markers of Anticancer Agent Toxicity.The use of genomic information to optimize cancer chemotherapy Comprehensive evaluation of the contribution of X chromosome genes to platinum sensitivity Germline polymorphisms discovered via a cell-based, genome-wide approach predict platinum response in head and neck cancers A genome-wide approach identifies that the aspartate metabolism pathway contributes to asparaginase sensitivity.Prediction of individual response to anticancer therapy: historical and future perspectives

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Identification of genetic variants contributing to cisplatin-induced cytotoxicity by use of a genomewide approach

http://www.wikidata.org/entity/Q35946327

description

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Identification of genetic vari ...... y use of a genomewide approach

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Identification of genetic vari ...... y use of a genomewide approach

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American Journal of Human Genetics

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Identification of genetic vari ...... y use of a genomewide approach

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P2093

Anthony C Schweitzer

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Emily O Kistner

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John E Blume

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M Eileen Dolan

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Shiwei Duan

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Sunita J Shukla

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Tina X Chen

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P2860

A haplotype map of the human genome

Nek2A kinase stimulates centrosome disjunction and is required for formation of bipolar mitotic spindles

Exploration, normalization, and summaries of high density oligonucleotide array probe level data

Cisplatin: mode of cytotoxic action and molecular basis of resistance

Cellular processing of platinum anticancer drugs

Analysis of differential protein expression by cisplatin treatment in cervical carcinoma cells.

Relation between cytokine promoter gene polymorphism and toxicity of 5-fluorouracil plus cisplatin chemotherapy.

Cellular response to oxidative stress: signaling for suicide and survival.

A general test of association for quantitative traits in nuclear families.

Curing metastatic cancer: lessons from testicular germ-cell tumours.

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R Stephanie Huang

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