Identification and characterization of CPS1 as a hyaluronic acid synthase contributing to the pathogenesis of Cryptococcus neoformans infection. - Wikidata - awgldk - TriplyDB

Identification and characterization of CPS1 as a hyaluronic acid synthase contributing to the pathogenesis of Cryptococcus neoformans infection.

Identification and characterization of CPS1 as a hyaluronic acid synthase contributing to the pathogenesis of Cryptococcus neoformans infection.

http://www.wikidata.org/entity/Q35947140

about

The Cryptococcus neoformans capsule: a sword and a shield.Genetic basis for hyper production of hyaluronic acid in natural and engineered microorganisms Virulence-Associated Enzymes of Cryptococcus neoformans Can host receptors for fungi be targeted for treatment of fungal infections?Surface-associated plasminogen binding of Cryptococcus neoformans promotes extracellular matrix invasion Binding of the wheat germ lectin to Cryptococcus neoformans suggests an association of chitinlike structures with yeast budding and capsular glucuronoxylomannan.Invasion of the central nervous system by Cryptococcus neoformans requires a secreted fungal metalloprotease.How sweet it is! Cell wall biogenesis and polysaccharide capsule formation in Cryptococcus neoformans.The Neurospora crassa CPS-1 polysaccharide synthase functions in cell wall biosynthesis Cryptococcus inositol utilization modulates the host protective immune response during brain infection Clustered Conserved Cysteines in Hyaluronan Synthase Mediate Cooperative Activation by Mg(2+) Ions and Severe Inhibitory Effects of Divalent Cations Identification of genes from the fungal pathogen Cryptococcus neoformans related to transmigration into the central nervous system Induction of brain microvascular endothelial cell urokinase expression by Cryptococcus neoformans facilitates blood-brain barrier invasion Brain inositol is a novel stimulator for promoting Cryptococcus penetration of the blood-brain barrier Host cell invasion by medically important fungi 4-methylumbelliferone treatment and hyaluronan inhibition as a therapeutic strategy in inflammation, autoimmunity, and cancer.Latent homology and convergent regulatory evolution underlies the repeated emergence of yeasts.Invasion of Cryptococcus neoformans into human brain microvascular endothelial cells is mediated through the lipid rafts-endocytic pathway via the dual specificity tyrosine phosphorylation-regulated kinase 3 (DYRK3)Alpha7 nicotinic acetylcholine receptor is required for blood-brain barrier injury-related CNS disorders caused by Cryptococcus neoformans and HIV-1 associated comorbidity factors Hyaluronic acid receptor CD44 deficiency is associated with decreased Cryptococcus neoformans brain infection cpsA regulates mycotoxin production, morphogenesis and cell wall biosynthesis in the fungus Aspergillus nidulans.Aimless mutants of Cryptococcus neoformans: failure to disseminate.CD44-mediated monocyte transmigration across Cryptococcus neoformans-infected brain microvascular endothelial cells is enhanced by HIV-1 gp41-I90 ectodomain Invasion of Cryptococcus neoformans into human brain microvascular endothelial cells requires protein kinase C-alpha activation.Immortalized human brain endothelial cell line HCMEC/D3 as a model of the blood-brain barrier facilitates in vitro studies of central nervous system infection by Cryptococcus neoformans.Real-time in vivo imaging of fungal migration to the central nervous system.Hypotheses on the evolution of hyaluronan: a highly ironic acid.Opportunistic yeast pathogens: reservoirs, virulence mechanisms, and therapeutic strategies.How Cryptococcus interacts with the blood-brain barrier.Production of extracellular polysaccharides by CAP mutants of Cryptococcus neoformans.Blood-brain barrier invasion by Cryptococcus neoformans is enhanced by functional interactions with plasmin.Lipid raft/caveolae signaling is required for Cryptococcus neoformans invasion into human brain microvascular endothelial cells.The Human Blood-Brain Barrier Internalizes Cryptococcus neoformans via the EphA2-Tyrosine Kinase Receptor.A conserved fungal glycosyltransferase facilitates pathogenesis of plants by enabling hyphal growth on solid surfaces.Innate immune evasion strategies against Cryptococcal meningitis caused by Cryptococcus neoformans.Biotechnological production of hyaluronic acid: a mini review.Production Methods for Hyaluronan

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P2860

Identification and characterization of CPS1 as a hyaluronic acid synthase contributing to the pathogenesis of Cryptococcus neoformans infection.

http://www.wikidata.org/entity/Q35947140

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Identification and characteriz ...... tococcus neoformans infection.

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Ambrose Jong

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Anna Plaas

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Chun-Hua Wu

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Craig W Lamunyon

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Feng Luo

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Han-Min Chen

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Kyung J Kwon-Chung

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Sheng-He Huang

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Yun C Chang

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P2860

The essential role of Saccharomyces cerevisiae CDC6 nucleotide-binding site in cell growth, DNA synthesis, and Orc1 association.

Adaptation of FACE methodology for microanalysis of total hyaluronan and chondroitin sulfate composition from cartilage

Isolation, characterization, and localization of a capsule-associated gene, CAP10, of Cryptococcus neoformans.

The active streptococcal hyaluronan synthases (HASs) contain a single HAS monomer and multiple cardiolipin molecules.

The cell biology of the blood-brain barrier.

Isolation of the third capsule-associated gene, CAP60, required for virulence in Cryptococcus neoformans

Fungal meningitis.

Functional cloning and characterization of a UDP- glucuronic acid decarboxylase: the pathogenic fungus Cryptococcus neoformans elucidates UDP-xylose synthesis.

Enhanced adhesion of Pasteurella multocida to cultured turkey peripheral blood monocytes

Cellular mechanisms of microbial proteins contributing to invasion of the blood-brain barrier.

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8

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