Endogenous interleukin-18 improves the early antimicrobial host response in severe melioidosis.
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Development of Burkholderia mallei and pseudomallei vaccinesGlyburide reduces bacterial dissemination in a mouse model of melioidosisGlobal and gene-specific translational regulation in rat lung developmentMyD88 dependent signaling contributes to protective host defense against Burkholderia pseudomalleiExpression and function of macrophage migration inhibitory factor (MIF) in melioidosis.Osteopontin impairs host defense during established gram-negative sepsis caused by Burkholderia pseudomallei (melioidosis).Caspase-6 mediates resistance against Burkholderia pseudomallei infection and influences the expression of detrimental cytokines.Cytokine-mediated regulation of antimicrobial proteins.Neutrophil elastase causes tissue damage that decreases host tolerance to lung infection with burkholderia species.Inflammasome-dependent pyroptosis and IL-18 protect against Burkholderia pseudomallei lung infection while IL-1β is deleteriousDiabetes-independent increase of factor VII-activating protease activation in patients with Gram-negative sepsis (melioidosis).Survey of innate immune responses to Burkholderia pseudomallei in human blood identifies a central role for lipopolysaccharide.Development of vaccines against burkholderia pseudomallei.Caspase-1 mediates resistance in murine melioidosis.Therapeutic Administration of a Monoclonal Anti-Il-1β Antibody Protects Against Experimental Melioidosis.The molecular and cellular basis of pathogenesis in melioidosis: how does Burkholderia pseudomallei cause disease?Role of Canonical and Non-canonical Inflammasomes During Burkholderia Infection.Heme Oxygenase-1 and Carbon Monoxide Promote Burkholderia pseudomallei Infection.Pathogenesis of percutaneous infection of goats with Burkholderia pseudomallei: clinical, pathologic, and immunological responses in chronic melioidosis.Interleukin 18 participates in the early inflammatory response and bacterial clearance during pneumonia caused by nontypeable Haemophilus influenzae.Expression and function of transforming growth factor β in melioidosis.Caspase-11-dependent pyroptosis of lung epithelial cells protects from melioidosis while caspase-1 mediates macrophage pyroptosis and production of IL-18.
P2860
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P2860
Endogenous interleukin-18 improves the early antimicrobial host response in severe melioidosis.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Endogenous interleukin-18 impr ...... esponse in severe melioidosis.
@ast
Endogenous interleukin-18 impr ...... esponse in severe melioidosis.
@en
type
label
Endogenous interleukin-18 impr ...... esponse in severe melioidosis.
@ast
Endogenous interleukin-18 impr ...... esponse in severe melioidosis.
@en
prefLabel
Endogenous interleukin-18 impr ...... esponse in severe melioidosis.
@ast
Endogenous interleukin-18 impr ...... esponse in severe melioidosis.
@en
P2093
P2860
P50
P356
P1476
Endogenous interleukin-18 impr ...... esponse in severe melioidosis.
@en
P2093
Anita de Boer
Arjen Dondorp
Catharina W Wieland
Gerritje J W van der Windt
W Joost Wiersinga
P2860
P304
P356
10.1128/IAI.00080-07
P407
P577
2007-05-21T00:00:00Z