Constitutive TL1A expression under colitogenic conditions modulates the severity and location of gut mucosal inflammation and induces fibrostenosis.
about
Cytokine and anti-cytokine therapies in prevention or treatment of fibrosis in IBDImmunopathology of inflammatory bowel diseaseUnique gene expression and MR T2 relaxometry patterns define chronic murine dextran sodium sulphate colitis as a model for connective tissue changes in human Crohn's diseaseInhibition of a novel fibrogenic factor Tl1a reverses established colonic fibrosis.Perturbations of mucosal homeostasis through interactions of intestinal microbes with myeloid cells.Central role of IL-17/Th17 immune responses and the gut microbiota in the pathogenesis of intestinal fibrosisTNFSF15 Modulates Neovascularization and InflammationSUSTAINED TL1A (TNFSF15) EXPRESSION ON BOTH LYMPHOID AND MYELOID CELLS LEADS TO MILD SPONTANEOUS INTESTINAL INFLAMMATION AND FIBROSIS.Multiple activating and repressive cis-promoter regions regulate TNFSF15 expression in human primary mononuclear cells.Cellular and Molecular Mediators of Intestinal Fibrosis.Tumor Necrosis Factor-like Cytokine TL1A and Its Receptors DR3 and DcR3: Important New Factors in Mucosal Homeostasis and Inflammation.Biologics beyond TNF-α inhibitors and the effect of targeting the homologues TL1A-DR3 pathway in chronic inflammatory disorders.An Overview of the Innate and Adaptive Immune System in Inflammatory Bowel Disease.The protection role of Atg16l1 in CD11c+dendritic cells in murine colitis.The Role of Cytokines in the Fibrotic Responses in Crohn's Disease.Association of Ribonuclease T2 Gene Polymorphisms With Decreased Expression and Clinical Characteristics of Severity in Crohn's Disease.Myeloid ATG16L1 Facilitates Host-Bacteria Interactions in Maintaining Intestinal Homeostasis.Death Receptor 3 regulates distinct pathological attributes of acute versus chronic murine allergic lung inflammation.Combining TNFSF15 and ASCA IgA can be used as a predictor for the stenosis/perforating phenotype of Crohn's disease.
P2860
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P2860
Constitutive TL1A expression under colitogenic conditions modulates the severity and location of gut mucosal inflammation and induces fibrostenosis.
description
2011 nî lūn-bûn
@nan
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
2011年论文
@zh
2011年论文
@zh-cn
name
Constitutive TL1A expression u ...... ion and induces fibrostenosis.
@ast
Constitutive TL1A expression u ...... ion and induces fibrostenosis.
@en
type
label
Constitutive TL1A expression u ...... ion and induces fibrostenosis.
@ast
Constitutive TL1A expression u ...... ion and induces fibrostenosis.
@en
prefLabel
Constitutive TL1A expression u ...... ion and induces fibrostenosis.
@ast
Constitutive TL1A expression u ...... ion and induces fibrostenosis.
@en
P2093
P2860
P1476
Constitutive TL1A expression u ...... ion and induces fibrostenosis.
@en
P2093
Charalabos Pothoulakis
David Q Shih
Dror Berel
Hon Wai Koon
Jyh-Yau Chang
Kathrin S Michelsen
Mary Ann Nguyen
Michelle Vu
Nicole Yeager
Robert Barrett
P2860
P304
P356
10.1016/J.AJPATH.2011.10.026
P407
P577
2011-12-02T00:00:00Z