Predominant activation of MAP kinases and pro-destructive/pro-inflammatory features by TNF alpha in early-passage synovial fibroblasts via TNF receptor-1: failure of p38 inhibition to suppress matrix metalloproteinase-1 in rheumatoid arthritis

Predominant activation of MAP kinases and pro-destructive/pro-inflammatory features by TNF alpha in early-passage synovial fibroblasts via TNF receptor-1: failure of p38 inhibition to suppress matrix metalloproteinase-1 in rheumatoid arthritis

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http://www.wikidata.org/entity/Q35953531
A crucial role for tumor necrosis factor receptor 1 in synovial lining cells and the reticuloendothelial system in mediating experimental arthritisMechanisms of non-opioid analgesics beyond cyclooxygenase enzyme inhibition.Activated protein C inhibits proliferation and tumor necrosis factor α-stimulated activation of p38, c-Jun NH2-terminal kinase (JNK) and Akt in rheumatoid synovial fibroblastsSelective involvement of ERK and JNK mitogen-activated protein kinases in early rheumatoid arthritis (1987 ACR criteria compared to 2010 ACR/EULAR criteria): a prospective study aimed at identification of diagnostic and prognostic biomarkers as wellVisfatin/pre-B-cell colony-enhancing factor (PBEF), a proinflammatory and cell motility-changing factor in rheumatoid arthritisCells of the synovium in rheumatoid arthritis. Macrophages.Cells of the synovium in rheumatoid arthritis. Synovial fibroblasts.IL-33 regulates TNF-α dependent effects in synovial fibroblasts.Metalloproteases as potential therapeutic targets in arthritis treatment.The Ras guanine nucleotide exchange factor RasGRF1 promotes matrix metalloproteinase-3 production in rheumatoid arthritis synovial tissue.Itraconazole inhibits TNF-α-induced CXCL10 expression in oral fibroblasts.Cellular electrophysiological principles that modulate secretion from synovial fibroblasts.ERK2 and JNK1 contribute to TNF-α-induced IL-8 expression in synovial fibroblasts.Tumor necrosis factor causes persistent sensitization of joint nociceptors to mechanical stimuli in rats.Apoptosis signal-regulating kinase 1 is mediated in TNF-α-induced CCL2 expression in human synovial fibroblasts.Inhibition of MAP kinase in synovium by treatment with tocilizumab in rheumatoid arthritis.Inhibition of inflammatory mediators and related signaling pathways by macrophage-stimulating protein in rheumatoid arthritis synovial fibroblasts.
P2860

Predominant activation of MAP kinases and pro-destructive/pro-inflammatory features by TNF alpha in early-passage synovial fibroblasts via TNF receptor-1: failure of p38 inhibition to suppress matrix metalloproteinase-1 in rheumatoid arthritis

Item
http://www.wikidata.org/entity/Q35953531
description
2007 nî lūn-bûn
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2007年の論文
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2007年論文
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2007年論文
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2007年論文
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2007年論文
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2007年論文
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2007年论文
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2007年论文
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2007年论文
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Predominant activation of MAP ...... nase-1 in rheumatoid arthritis
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Predominant activation of MAP ...... nase-1 in rheumatoid arthritis
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Predominant activation of MAP ...... nase-1 in rheumatoid arthritis
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Predominant activation of MAP ...... nase-1 in rheumatoid arthritis
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Predominant activation of MAP ...... nase-1 in rheumatoid arthritis
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Predominant activation of MAP ...... nase-1 in rheumatoid arthritis
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Predominant activation of MAP ...... nase-1 in rheumatoid arthritis
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P2093
P2860
P304
P31
P356
P407
P433
P478
P577
P5875
P698
P921
P932