Centrosome amplification and the origin of chromosomal instability in breast cancer.
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Centrosome dysfunction contributes to chromosome instability, chromoanagenesis, and genome reprograming in cancerGenotoxicity of multi-walled carbon nanotubes at occupationally relevant dosesSingle-walled carbon nanotube-induced mitotic disruptionPotential pulmonary effects of engineered carbon nanotubes: in vitro genotoxic effectsGenotoxicity and carcinogenic potential of carbon nanomaterialsDifferential regulation of centrosome integrity by DNA damage response proteins.Laser microsurgery in the GFP era: a cell biologist's perspectiveVariation in genes required for normal mitosis and risk of breast cancerDelocalization of gamma-tubulin due to increased solubility in human breast cancer cell linesE2F activators signal and maintain centrosome amplification in breast cancer cells.Nuclear CSPP1 expression defined subtypes of basal-like breast cancer.Causes and consequences of centrosome abnormalities in cancer.Formation of extra centrosomal structures is dependent on beta-catenin.Abnormal centrosome amplification in cells through the targeting of Ran-binding protein-1 by the human T cell leukemia virus type-1 Tax oncoproteinCan the centrosome be a marker for DNA ploidy in breast cancer?Pin1 regulates centrosome duplication, and its overexpression induces centrosome amplification, chromosome instability, and oncogenesisA gene signature predictive for outcome in advanced ovarian cancer identifies a survival factor: microfibril-associated glycoprotein 2.Cdk4 and nek2 signal binucleation and centrosome amplification in a her2+ breast cancer model.Mouse embryonic fibroblasts null for the Krüppel-like factor 4 gene are genetically unstable.Differential expression of arrestins is a predictor of breast cancer progression and survival.A clinical overview of centrosome amplification in human cancers.Centrosome amplification induces high grade features and is prognostic of worse outcomes in breast cancer.Tubulin targets in the pathobiology and therapy of glioblastoma multiforme. II. gamma-Tubulin.Implications of genomic instability in the diagnosis and treatment of breast cancer.Therapeutic applications of carbon nanotubes: opportunities and challenges.The 3D nuclear organization of telomeres marks the transition from Hodgkin to Reed-Sternberg cells.Geometric approach to segmentation and protein localization in cell culture assays.A comparison of folic acid deficiency-induced genomic instability in lymphocytes of breast cancer patients and normal non-cancer controls from a Chinese population in Yunnan.Structural centrosome aberrations promote non-cell-autonomous invasiveness.Chromosomal instability induced by mammography X-rays in primary human fibroblasts from BRCA1 and BRCA2 mutation carriers.
P2860
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P2860
Centrosome amplification and the origin of chromosomal instability in breast cancer.
description
2004 nî lūn-bûn
@nan
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
2004年论文
@zh
2004年论文
@zh-cn
name
Centrosome amplification and the origin of chromosomal instability in breast cancer.
@ast
Centrosome amplification and the origin of chromosomal instability in breast cancer.
@en
type
label
Centrosome amplification and the origin of chromosomal instability in breast cancer.
@ast
Centrosome amplification and the origin of chromosomal instability in breast cancer.
@en
prefLabel
Centrosome amplification and the origin of chromosomal instability in breast cancer.
@ast
Centrosome amplification and the origin of chromosomal instability in breast cancer.
@en
P2093
P1476
Centrosome amplification and the origin of chromosomal instability in breast cancer.
@en
P2093
Antonino B D'Assoro
Jeffrey L Salisbury
Wilma L Lingle
P304
P356
10.1023/B:JOMG.0000048774.27697.30
P577
2004-07-01T00:00:00Z
P6179
1019587443