Epstein-Barr virus oncoprotein super-enhancers control B cell growth - Wikidata - awgldk - TriplyDB

Epstein-Barr virus oncoprotein super-enhancers control B cell growth

Epstein-Barr virus oncoprotein super-enhancers control B cell growth

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Epigenetic Impact on EBV Associated B-Cell Lymphomagenesis EBNA2 Drives Formation of New Chromosome Binding Sites and Target Genes for B-Cell Master Regulatory Transcription Factors RBP-jκ and EBF1 The Epstein-Barr Virus (EBV) in T Cell and NK Cell Lymphomas: Time for a Reassessment.An ensemble of regulatory elements controls Runx3 spatiotemporal expression in subsets of dorsal root ganglia proprioceptive neurons Higher-Order Chromatin Regulation of Inflammatory Gene Expression Chemical probes targeting epigenetic proteins: Applications beyond oncology.Mice deficient of Myc super-enhancer region reveal differential control mechanism between normal and pathological growth.Selective inhibition of protein arginine methyltransferase 5 blocks initiation and maintenance of B-cell transformation EBV Nuclear Antigen 3C Mediates Regulation of E2F6 to Inhibit E2F1 Transcription and Promote Cell Proliferation.Role of NSC319726 in ovarian cancer based on the bioinformatics analyses Super Enhancers in Cancers, Complex Disease, and Developmental Disorders.Induction of Epstein-Barr Virus Oncoprotein LMP1 by Transcription Factors AP-2 and Early B Cell Factor Utility of Lymphoblastoid Cell Lines for Induced Pluripotent Stem Cell Generation Hierarchy within the mammary STAT5-driven Wap super-enhancer.MYC activation and BCL2L11 silencing by a tumour virus through the large-scale reconfiguration of enhancer-promoter hubs.Epstein-Barr virus super-enhancer eRNAs are essential for MYC oncogene expression and lymphoblast proliferation Epstein-Barr virus latency: current and future perspectives.EBV epigenetically suppresses the B cell-to-plasma cell differentiation pathway while establishing long-term latency.Nasopharyngeal carcinoma super-enhancer-driven ETV6 correlates with prognosis.Therapeutics Targeting Protein Acetylation Perturb Latency of Human Viruses.Molecular mechanisms of human papillomavirus-related carcinogenesis in head and neck cancer.RUNX super-enhancer control through the Notch pathway by Epstein-Barr virus transcription factors regulates B cell growth.The role of metabolic reprogramming in γ-herpesvirus-associated oncogenesis.LMP1 and Dynamic Progressive Telomere Dysfunction: A Major Culprit in EBV-Associated Hodgkin's Lymphoma.Super-Enhancer-Driven Transcriptional Dependencies in Cancer.JQ1 reduces Epstein-Barr virus-associated lymphoproliferative disease in mice without sustained oncogene repression.Epstein-Barr Virus LMP1 Mediated Oncogenicity.Regulation of tumour related genes by dynamic epigenetic alteration at enhancer regions in gastric epithelial cells infected by Epstein-Barr virus.Epstein-Barr virus: a master epigenetic manipulator.Roles of RUNX in B Cell Immortalisation.The Epstein-Barr Virus Regulome in Lymphoblastoid Cells.EBF1 binds to EBNA2 and promotes the assembly of EBNA2 chromatin complexes in B cells.Integrated analysis of whole-genome ChIP-Seq and RNA-Seq data of primary head and neck tumor samples associates HPV integration sites with open chromatin marks.c-Myc Represses Transcription of the Epstein-Barr Virus Latent Membrane Protein 1 Early After Primary B Cell Infection.CRISPR-Cas9 Genetic Analysis of Virus-Host Interactions.Current Progress in EBV-Associated B-Cell Lymphomas.LIMD1 is induced by and required for LMP1 signaling, and protects EBV-transformed cells from DNA damage-induced cell death.Transcription factors operate across disease loci, with EBNA2 implicated in autoimmunity.Epstein–Barr Virus-Induced Metabolic Rearrangements in Human B-Cell Lymphomas.Enhancer control of miR-155 expression in Epstein-Barr virus infected B cells

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Epstein-Barr virus oncoprotein super-enhancers control B cell growth

http://www.wikidata.org/entity/Q35963272

description

2015 nî lūn-bûn

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2015年论文

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Epstein-Barr virus oncoprotein super-enhancers control B cell growth

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Epstein-Barr virus oncoprotein super-enhancers control B cell growth

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Epstein-Barr virus oncoprotein super-enhancers control B cell growth

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Epstein-Barr virus oncoprotein super-enhancers control B cell growth

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Epstein-Barr virus oncoprotein super-enhancers control B cell growth

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Epstein-Barr virus oncoprotein super-enhancers control B cell growth

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J.CHOM.2014.12.013

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Cell Host & Microbe

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Epstein-Barr virus oncoprotein super-enhancers control B cell growth

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Benjamin E Gewurz

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Bo Zhao

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Bradford Willox

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Eric C Johannsen

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Hufeng Zhou

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Jun Liang

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Katharina Bernhardt

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Peter Kharchenko

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Sizun Jiang

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Stefanie C S Schmidt

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The viral and cellular microRNA targetome in lymphoblastoid cell lines

Model-based analysis of ChIP-Seq (MACS)

NF-kappa B inhibition causes spontaneous apoptosis in Epstein-Barr virus-transformed lymphoblastoid cells

Selective inhibition of BET bromodomains

Epstein-Barr virus nuclear antigen 3C and prothymosin alpha interact with the p300 transcriptional coactivator at the CH1 and CH3/HAT domains and cooperate in regulation of transcription and histone acetylation.

The 62- and 80-kDa subunits of transcription factor IIH mediate the interaction with Epstein-Barr virus nuclear protein 2

An atlas of the Epstein-Barr virus transcriptome and epigenome reveals host-virus regulatory interactions

Simple combinations of lineage-determining transcription factors prime cis-regulatory elements required for macrophage and B cell identities

Epstein-Barr virus nuclear protein 2 (EBNA2) binds to a component of the human SNF-SWI complex, hSNF5/Ini1

Orchestrating B cell lymphopoiesis through interplay of IL-7 receptor and pre-B cell receptor signalling

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205-216

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scholarly article

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10.1016/J.CHOM.2014.12.013

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2

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17

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Elliott D. Kieff

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2015-01-29T00:00:00Z

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271073802

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25639793

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Epstein–Barr virus

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4539236

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