Inflammatory signaling in human tuberculosis granulomas is spatially organized. - Wikidata - awgldk - TriplyDB

Inflammatory signaling in human tuberculosis granulomas is spatially organized.

Inflammatory signaling in human tuberculosis granulomas is spatially organized.

http://www.wikidata.org/entity/Q35979217

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Granulomas and Inflammation: Host-Directed Therapies for Tuberculosis.Dissection of the host-pathogen interaction in human tuberculosis using a bioengineered 3-dimensional model Intracellular growth of Mycobacterium tuberculosis after macrophage cell death leads to serial killing of host cells.Hypervirulent Mycobacterium tuberculosis strain triggers necrotic lung pathology associated with enhanced recruitment of neutrophils in resistant C57BL/6 mice A Bioengineered Three-Dimensional Cell Culture Platform Integrated with Microfluidics To Address Antimicrobial Resistance in Tuberculosis.The bacillary and macrophage response to hypoxia in tuberculosis and the consequences for T cell antigen recognition.Chemical activation of adenylyl cyclase Rv1625c inhibits growth of Mycobacterium tuberculosis on cholesterol and modulates intramacrophage signaling.Protein kinase C-delta (PKCδ), a marker of inflammation and tuberculosis disease progression in humans, is important for optimal macrophage killing effector functions and survival in mice.Nitric oxide prevents a pathogen-permissive granulocytic inflammation during tuberculosis.Macrophage form, function, and phenotype in mycobacterial infection: lessons from tuberculosis and other diseases.The Wnt Blows: On the Functional Role of Wnt Signaling in Mycobacterium tuberculosis Infection and Beyond Unravelling the networks dictating host resistance versus tolerance during pulmonary infections.Crystal nephropathies: mechanisms of crystal-induced kidney injury.Integrating Lung Physiology, Immunology, and Tuberculosis.Mycobacterium tuberculosis in the Face of Host-Imposed Nutrient Limitation.Kunkel Lecture: Fundamental immunodeficiency and its correction.NOS2-deficient mice with hypoxic necrotizing lung lesions predict outcomes of tuberculosis chemotherapy in humans.The Capacity of Mycobacterium tuberculosis To Survive Iron Starvation Might Enable It To Persist in Iron-Deprived Microenvironments of Human Granulomas.HIV-Associated Cardiovascular Disease: Role of Connexin 43.Mycobacterium tuberculosis DosR Regulon Gene Rv2004c Encodes a Novel Antigen with Pro-inflammatory Functions and Potential Diagnostic Application for Detection of Latent Tuberculosis.Mass spectrometry-based proteomic exploration of the human immune system: focus on the inflammasome, global protein secretion, and T cells.Macrophage Epithelial Reprogramming Underlies Mycobacterial Granuloma Formation and Promotes Infection.Utility of Macrophage-activated Marker CD163 for Diagnosis and Prognosis in Pulmonary Tuberculosis.The ins and outs of the Mycobacterium tuberculosis-containing vacuole.Comparison of Surface Proteomes of Adherence Variants of Listeria Monocytogenes Using LC-MS/MS for Identification of Potential Surface Adhesins.Mycobacterial infection induces higher interleukin-1β and dysregulated lung inflammation in mice with defective leukocyte NADPH oxidase.The Troika Host-Pathogen-Extrinsic Factors in Tuberculosis: Modulating Inflammation and Clinical Outcomes.Tuberculosis Infectiousness and Host Susceptibility.Immunological roulette: Luck or something more? Considering the connections between host and environment in TB.Deletion of TGF-β1 Increases Bacterial Clearance by Cytotoxic T Cells in a Tuberculosis Granuloma Model.Spatial Tissue Proteomics Quantifies Inter- and Intratumor Heterogeneity in Hepatocellular Carcinoma (HCC).Gene Enrichment Analysis Reveals Major Regulators of Mycobacterium tuberculosis Gene Expression in Two Models of Antibiotic Tolerance.Growth of Mycobacterium tuberculosis in vivo segregates with host macrophage metabolism and ontogeny.Macrophage Heterogeneity in the Immunopathogenesis of Tuberculosis.A Beneficial Effect of Low-Dose Aspirin in a Murine Model of Active Tuberculosis.Transferring knowledge of bacterial protein interaction networks to predict pathogen targeted human genes and immune signaling pathways: a case study on M. tuberculosis.Storage lipid studies in tuberculosis reveal that foam cell biogenesis is disease-specific Human Monocytic Suppressive Cells Promote Replication of and Alter Stability of Generated Granulomas The Immune Mechanisms of Lung Parenchymal Damage in Tuberculosis and the Role of Host-Directed Therapy Combining discovery and targeted proteomics reveals a prognostic signature in oral cancer

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Inflammatory signaling in human tuberculosis granulomas is spatially organized.

http://www.wikidata.org/entity/Q35979217

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2016 nî lūn-bûn

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2016年の論文

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2016年论文

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2016年论文

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name

Inflammatory signaling in human tuberculosis granulomas is spatially organized.

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Inflammatory signaling in human tuberculosis granulomas is spatially organized.

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Inflammatory signaling in human tuberculosis granulomas is spatially organized.

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Inflammatory signaling in human tuberculosis granulomas is spatially organized.

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Inflammatory signaling in human tuberculosis granulomas is spatially organized.

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Inflammatory signaling in human tuberculosis granulomas is spatially organized.

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Nature Medicine

P1476

Inflammatory signaling in human tuberculosis granulomas is spatially organized.

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Brendan Prideaux

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Clifton E Barry

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Eliseo A Eugenin

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Eric J Rubin

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Jin Hee Kim

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Kirti Sharma

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Laura E Via

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Matthias Mann

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Pei-Yu Chen

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Ravikiran M Raju

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P2860

TB: the Yin and Yang of lipid mediators

Variability in tuberculosis granuloma T cell responses exists, but a balance of pro- and anti-inflammatory cytokines is associated with sterilization

Type I interferons in infectious disease

Resolving inflammation: dual anti-inflammatory and pro-resolution lipid mediators

Andromeda: a peptide search engine integrated into the MaxQuant environment

Tumor necrosis factor-alpha is required in the protective immune response against Mycobacterium tuberculosis in mice

The lta4h locus modulates susceptibility to mycobacterial infection in zebrafish and humans

Multiscale computational modeling reveals a critical role for TNF-α receptor 1 dynamics in tuberculosis granuloma formation.

The role of the granuloma in expansion and dissemination of early tuberculous infection.

Tumor necrosis factor signaling mediates resistance to mycobacteria by inhibiting bacterial growth and macrophage death.

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531-538

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10.1038/NM.4073

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5

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22

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Matthew G. Booty

Samuel M. Behar

Mohlopheni Marakalala

Isaac B Daudelin

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2016-04-04T00:00:00Z

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1003867873

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27043495

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Akafuba bulwadde

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4860068

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