Obesogens, stem cells and the developmental programming of obesity.
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Environmental Chemical Assessment in Clinical Practice: Unveiling the Elephant in the RoomRisk Evaluation of Endocrine-Disrupting Chemicals: Effects of Developmental Exposure to Low Doses of Bisphenol A on Behavior and Physiology in Mice (Mus musculus)Zebrafish as a Model to Study the Role of Peroxisome Proliferating-Activated Receptors in Adipogenesis and ObesityThe Role of Epigenetics in the Latent Effects of Early Life Exposure to Obesogenic Endocrine Disrupting ChemicalsThe epigenetic lorax: gene-environment interactions in human healthEDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting ChemicalsChallenges and future directions to evaluating the association between prenatal exposure to endocrine disrupting chemicals and childhood obesityEditor's Highlight: Screening ToxCast Prioritized Chemicals for PPARG Function in a Human Adipose-Derived Stem Cell Model of AdipogenesisGene-Lifestyle Interactions in ObesityObesity and diabetes: from genetics to epigenetics.Altered Adipogenesis in Zebrafish Larvae Following High Fat Diet and Chemical Exposure Is Visualised by Stimulated Raman Scattering MicroscopyThe pathogenesis of obesity from a genomic and systems biology perspective.Halogenated bisphenol-A analogs act as obesogens in zebrafish larvae (Danio rerio).Diosgenin relieves goiter via the inhibition of thyrocyte proliferation in a mouse model of Graves' disease.Early-life bisphenol a exposure and child body mass index: a prospective cohort study.Low dietary calcium and obesity: a comparative study in genetically obese and normal rats during early growth.Ancestral dichlorodiphenyltrichloroethane (DDT) exposure promotes epigenetic transgenerational inheritance of obesity.Evolution of DOHaD: the impact of environmental health sciences.The expression of selected genes linked to metabolic homeostasis in obese pansteatitis-suffering Mozambique tilapia, Oreochromis mossambicus (Peters).Metabolic outcome of female mice exposed to a mixture of low-dose pollutants in a diet-induced obesity model.Reporter Cell Lines for the Characterization of the Interactions between Human Nuclear Receptors and Endocrine Disruptors.Reporter cell lines to evaluate the selectivity of chemicals for human and zebrafish estrogen and peroxysome proliferator activated γ receptorsParma consensus statement on metabolic disruptorsPrenatal perfluoroalkyl substance exposure and child adiposity at 8 years of age: The HOME study.Developmental origins of obesity: programmed adipogenesisEpigenetic influences in the aetiology of cancers arising from breast and prostate: a hypothesised transgenerational evolution in chromatin accessibilityAdipocytes under assault: environmental disruption of adipose physiology.Metabolic disruption in male mice due to fetal exposure to low but not high doses of bisphenol A (BPA): evidence for effects on body weight, food intake, adipocytes, leptin, adiponectin, insulin and glucose regulation.Transcriptional and epigenetic mechanisms underlying enhanced in vitro adipocyte differentiation by the brominated flame retardant BDE-47.Epigenetics in the development, modification, and prevention of cardiovascular disease.Childhood obesity: a (re) programming disease?Individual and Joint Effects of Early-Life Ambient Exposure and Maternal Prepregnancy Obesity on Childhood Overweight or Obesity.Environmental signaling: from environmental estrogens to endocrine-disrupting chemicals and beyond.Obesogens and male fertility.An evaluation of the endocrine disruptive potential of crude oil water accommodated fractions and crude oil contaminated surface water to freshwater organisms using in vitro and in vivo approaches.A structural perspective on nuclear receptors as targets of environmental compounds.Prenatal exposure to persistent organic pollutants and rapid weight gain and overweight in infancy.The effects of the obesogen tributyltin on the metabolism of Sertoli cells cultured ex vivo.Chemical and non-chemical stressors affecting childhood obesity: a systematic scoping review.Endocrine Disruptors and Developmental Origins of Nonalcoholic Fatty Liver Disease.
P2860
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P2860
Obesogens, stem cells and the developmental programming of obesity.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Obesogens, stem cells and the developmental programming of obesity.
@ast
Obesogens, stem cells and the developmental programming of obesity.
@en
type
label
Obesogens, stem cells and the developmental programming of obesity.
@ast
Obesogens, stem cells and the developmental programming of obesity.
@en
prefLabel
Obesogens, stem cells and the developmental programming of obesity.
@ast
Obesogens, stem cells and the developmental programming of obesity.
@en
P2860
P1476
Obesogens, stem cells and the developmental programming of obesity.
@en
P2093
A Janesick
B Blumberg
P2860
P304
P356
10.1111/J.1365-2605.2012.01247.X
P577
2012-02-28T00:00:00Z