Luteinizing hormone reduces the activity of the NPR2 guanylyl cyclase in mouse ovarian follicles, contributing to the cyclic GMP decrease that promotes resumption of meiosis in oocytes.
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Selective Regulation of Oocyte Meiotic Events Enhances Progress in Fertility Preservation MethodsThe Ovarian Antral Follicle: Living on the Edge of Hypoxia or Not?Epidermal growth factor-mediated mitogen-activated protein kinase3/1 pathway is conducive to in vitro maturation of sheep oocytesThe Local CNP/GC-B system in growth plate is responsible for physiological endochondral bone growth.Biologically active substances-enriched diet regulates gonadotrope cell activation pathway in liver of adult and old rats.Dephosphorylation and inactivation of NPR2 guanylyl cyclase in granulosa cells contributes to the LH-induced decrease in cGMP that causes resumption of meiosis in rat oocytes.Two classes of gap junction channels mediate soma-germline interactions essential for germline proliferation and gametogenesis in Caenorhabditis elegansA tudor domain protein SPINDLIN1 interacts with the mRNA-binding protein SERBP1 and is involved in mouse oocyte meiotic resumption.Cyclic guanosine monophosphate does not inhibit gonadotropin-induced activation of mitogen-activated protein kinase 3/1 in pig cumulus-oocyte complexes.Guanylyl cyclases A and B are asymmetric dimers that are allosterically activated by ATP binding to the catalytic domain.Intercellular signaling via cyclic GMP diffusion through gap junctions restarts meiosis in mouse ovarian follicles.Multiple pathways mediate luteinizing hormone regulation of cGMP signaling in the mouse ovarian follicleEffect of C-Type Natriuretic Peptide on Maturation and Developmental Competence of Goat Oocytes Matured In Vitro.Ontogeny of the ovary in polycystic ovary syndrome.Regulation of mitogen-activated protein kinase 3/1 activity during meiosis resumption in mammals.Hormonal coordination of natriuretic peptide type C and natriuretic peptide receptor 3 expression in mouse granulosa cells.Dephosphorylation of juxtamembrane serines and threonines of the NPR2 guanylyl cyclase is required for rapid resumption of oocyte meiosis in response to luteinizing hormone.Mitochondria Synthesize Melatonin to Ameliorate Its Function and Improve Mice Oocyte's Quality under in Vitro Conditions.Luteinizing Hormone Causes Phosphorylation and Activation of the cGMP Phosphodiesterase PDE5 in Rat Ovarian Follicles, Contributing, Together with PDE1 Activity, to the Resumption of MeiosisBidirectional communication between oocytes and ovarian follicular somatic cells is required for meiotic arrest of mammalian oocytesNatriuretic peptide type C induces sperm attraction for fertilization in mouse.cyclic GMP Mediated Inhibition of Spontaneous Germinal Vesicle Breakdown Both with and without Cumulus in Mouse Oocyte.Estrogen receptors in granulosa cells govern meiotic resumption of pre-ovulatory oocytes in mammals.Control of Oocyte Growth and Development by Intercellular Communication Within the Follicular Niche.Transcriptome analysis of human cumulus cells reveals hypoxia as the main determinant of follicular senescence.MAPK3/1 is conducive to luteinizing hormone-mediated C-type natriuretic peptide decrease in bovine granulosa cells.Macrophage colony-stimulating factor (M-CSF) is an intermediate in the process of luteinizing hormone-induced decrease in natriuretic peptide receptor 2 (NPR2) and resumption of oocyte meiosis.A pre-in vitro maturation medium containing cumulus oocyte complex ligand-receptor signaling molecules maintains meiotic arrest, supports the cumulus oocyte complex and improves oocyte developmental competence.Brain natriuretic peptide and C-type natriuretic peptide maintain porcine oocyte meiotic arrest.Role of Mitogen Activated Protein Kinase and Maturation Promoting Factor During the Achievement of Meiotic Competency in Mammalian Oocytes.Effect of C-type natriuretic peptide pretreatment on in vitro bovine oocyte maturation.Regulation of germ cell development by intercellular signaling in the mammalian ovarian follicle.Transporting cumulus complexes using novel meiotic arresting conditions permits maintenance of oocyte developmental competence.Luteinizing hormone signaling phosphorylates and activates the cyclic GMP phosphodiesterase PDE5 in mouse ovarian follicles, contributing an additional component to the hormonally induced decrease in cyclic GMP that reinitiates meiosis.Significance of epidermal growth factor receptor signaling for acquisition of meiotic and developmental competence in mammalian oocytes.Regulation of Mammalian Oocyte Meiosis by Intercellular Communication Within the Ovarian Follicle.CREB activity is required for luteinizing hormone-induced the expression of EGF-like factors.Beneficial effects of melatonin on bovine oocytes maturation: a mechanistic approach.CNP/NPR2 signaling maintains oocyte meiotic arrest in early antral follicles and is suppressed by EGFR-mediated signaling in preovulatory follicles.MPF-based meiotic cell cycle control: Half a century of lessons from starfish oocytes.
P2860
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P2860
Luteinizing hormone reduces the activity of the NPR2 guanylyl cyclase in mouse ovarian follicles, contributing to the cyclic GMP decrease that promotes resumption of meiosis in oocytes.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Luteinizing hormone reduces th ...... umption of meiosis in oocytes.
@ast
Luteinizing hormone reduces th ...... umption of meiosis in oocytes.
@en
type
label
Luteinizing hormone reduces th ...... umption of meiosis in oocytes.
@ast
Luteinizing hormone reduces th ...... umption of meiosis in oocytes.
@en
prefLabel
Luteinizing hormone reduces th ...... umption of meiosis in oocytes.
@ast
Luteinizing hormone reduces th ...... umption of meiosis in oocytes.
@en
P2093
P2860
P1476
Luteinizing hormone reduces th ...... umption of meiosis in oocytes.
@en
P2093
Andreas Geerts
Frank Wunder
Jerid W Robinson
John J Eppig
Laurinda A Jaffe
Leia C Shuhaibar
Lincoln R Potter
Meijia Zhang
Rachael P Norris
P2860
P304
P356
10.1016/J.YDBIO.2012.04.019
P407
P577
2012-04-21T00:00:00Z