Conditional deletion of β-catenin in mammary epithelial cells of Ron receptor, Mst1r, overexpressing mice alters mammary tumorigenesis.
about
Ron receptor tyrosine kinase signaling as a therapeutic targetFatty acid synthase is required for mammary gland development and milk production during lactationTumor cell migration screen identifies SRPK1 as breast cancer metastasis determinant.Vitamin D3-dependent VDR signaling delays ron-mediated breast tumorigenesis through suppression of β-catenin activity.HGFL supports mammary tumorigenesis by enhancing tumor cell intrinsic survival and influencing macrophage and T-cell responses.HGFL-mediated RON signaling supports breast cancer stem cell phenotypes via activation of non-canonical β-catenin signaling.RON Signaling Is a Key Mediator of Tumor Progression in Many Human Cancers.mTORC1 is a key mediator of RON-dependent breast cancer metastasis with therapeutic potential
P2860
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P2860
Conditional deletion of β-catenin in mammary epithelial cells of Ron receptor, Mst1r, overexpressing mice alters mammary tumorigenesis.
description
2012 nî lūn-bûn
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年论文
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2012年论文
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2012年论文
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name
Conditional deletion of β-cate ...... alters mammary tumorigenesis.
@ast
Conditional deletion of β-cate ...... alters mammary tumorigenesis.
@en
type
label
Conditional deletion of β-cate ...... alters mammary tumorigenesis.
@ast
Conditional deletion of β-cate ...... alters mammary tumorigenesis.
@en
prefLabel
Conditional deletion of β-cate ...... alters mammary tumorigenesis.
@ast
Conditional deletion of β-cate ...... alters mammary tumorigenesis.
@en
P2093
P2860
P356
P1433
P1476
Conditional deletion of β-cate ...... e alters mammary tumorigenesis
@en
P2093
Archana Shrestha
Glendon M Zinser
Jerilyn K Gray
Purnima K Wagh
P2860
P304
P356
10.1210/EN.2011-1543
P407
P577
2012-04-02T00:00:00Z