Leukemia stem cells in T-ALL require active Hif1α and Wnt signaling - Wikidata - awgldk - TriplyDB

Leukemia stem cells in T-ALL require active Hif1α and Wnt signaling

Leukemia stem cells in T-ALL require active Hif1α and Wnt signaling

http://www.wikidata.org/entity/Q35993601

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Aberrant Wnt Signaling in Leukemia Targeting leukemia stem cells: which pathways drive self-renewal activity in T-cell acute lymphoblastic leukemia?Hypoxia and Hypoxia-Inducible Factors in Leukemias Single-cell transcriptional analysis of normal, aberrant, and malignant hematopoiesis in zebrafish Tumor-Initiating Cells: a criTICal review of isolation approaches and new challenges in targeting strategies.Antineoplastic effects and mechanisms of micheliolide in acute myelogenous leukemia stem cells.Hypoxia regulates the hematopoietic stem cell niche.Effect of hypoxia-inducible factors in normal and leukemic stem cell regulation and their potential therapeutic impact.The Wnt signaling pathway in cancer.Conserved IKAROS-regulated genes associated with B-progenitor acute lymphoblastic leukemia outcome.Control of amino acid transport coordinates metabolic reprogramming in T-cell malignancy.Integration of hypoxic HIF-α signaling in blood cancers.β-Catenin is required for T-cell leukemia initiation and MYC transcription downstream of Notch1.EZH2 mediates ATO-induced apoptosis in acute myeloid leukemia cell lines through the Wnt signaling pathway.Molecular Pathways: Hypoxia-Activated Prodrugs in Cancer Therapy.The hypoxia signalling pathway in haematological malignancies.CBP/Catenin antagonists: Targeting LSCs' Achilles heel.Aberrant Signaling Pathways in T-Cell Acute Lymphoblastic Leukemia.Priming of the Cells: Hypoxic Preconditioning for Stem Cell Therapy A positive feedback loop involving the Wnt/β-catenin/MYC/Sox2 axis defines a highly tumorigenic cell subpopulation in ALK-positive anaplastic large cell lymphoma Pharmacologic Manipulation of Wnt Signaling and Cancer Stem Cells.Molecular Pathogenesis of Radiation-Induced Cell Toxicity in Stem Cells.TOX Regulates Growth, DNA Repair, and Genomic Instability in T-cell Acute Lymphoblastic Leukemia.A Regulatory Role for RUNX1, RUNX3 in the Maintenance of Genomic Integrity.Wnt signaling in cancer.The Ins and Outs of Autophagy and Metabolism in Hematopoietic and Leukemic Stem Cells: Food for Thought

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Leukemia stem cells in T-ALL require active Hif1α and Wnt signaling

http://www.wikidata.org/entity/Q35993601

description

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2015年論文

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2015年論文

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2015年論文

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name

Leukemia stem cells in T-ALL require active Hif1α and Wnt signaling

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Leukemia stem cells in T-ALL require active Hif1α and Wnt signaling

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type

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Leukemia stem cells in T-ALL require active Hif1α and Wnt signaling

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Leukemia stem cells in T-ALL require active Hif1α and Wnt signaling

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prefLabel

Leukemia stem cells in T-ALL require active Hif1α and Wnt signaling

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Leukemia stem cells in T-ALL require active Hif1α and Wnt signaling

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Leukemia stem cells in T-ALL require active Hif1α and Wnt signaling

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Andrew P Weng

http://www.w3.org/2001/XMLSchema#string

Catherine E Jenkins

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Catherine Hoofd

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Deanne Gracias

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Miriam Belmonte

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Sam Gusscott

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Sonya H Lam

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Vincenzo Giambra

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Xuehai Wang

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P2860

The ubiquitin ligase FBXW7 modulates leukemia-initiating cell activity by regulating MYC stability

Tankyrase inhibition stabilizes axin and antagonizes Wnt signalling

Wnt/β-catenin signaling and disease

Wnt signaling strength regulates normal hematopoiesis and its deregulation is involved in leukemia development

Beta-catenin regulates expression of cyclin D1 in colon carcinoma cells

Stem cells, cancer, and cancer stem cells

Cancer stem cells--perspectives on current status and future directions: AACR Workshop on cancer stem cells

Lentiviral vectors to probe and manipulate the Wnt signaling pathway

Inactivation of the beta-catenin gene by Wnt1-Cre-mediated deletion results in dramatic brain malformation and failure of craniofacial development

Pronounced hypoxia in models of murine and human leukemia: high efficacy of hypoxia-activated prodrug PR-104

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3917-3927

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10.1182/BLOOD-2014-10-609370

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