Lipopolysaccharide induces delayed FosB/DeltaFosB immunostaining within the mouse extended amygdala, hippocampus and hypothalamus, that parallel the expression of depressive-like behavior.
about
From inflammation to sickness and depression: when the immune system subjugates the brainCytokine, sickness behavior, and depressionTwenty years of research on cytokine-induced sickness behaviorLinking Activation of Microglia and Peripheral Monocytic Cells to the Pathophysiology of Psychiatric DisordersInflammation Models of Depression in Rodents: Relevance to Psychotropic Drug DiscoveryNeuroinflammation and Depression: Microglia Activation, Extracellular Microvesicles and microRNA DysregulationResponse to peripheral immune stimulation within the brain: magnetic resonance imaging perspective of treatment successRole of neuroinflammation in the emotional and cognitive alterations displayed by animal models of obesityDepression and sickness behavior are Janus-faced responses to shared inflammatory pathways.Neuropsychiatric comorbidity in obesity: role of inflammatory processesCytokine effects on the basal ganglia and dopamine function: the subcortical source of inflammatory malaiseBrain-derived neurotrophic factor: a bridge between inflammation and neuroplasticityAlcoholism and inflammation: neuroimmunology of behavioral and mood disordersInfluenza virus- and cytokine-immunoreactive cells in the murine olfactory and central autonomic nervous systems before and after illness onsetThe pharmacokinetic-pharmacodynamic model of azithromycin for lipopolysaccharide-induced depressive-like behavior in miceInduction of IDO by bacille Calmette-Guérin is responsible for development of murine depressive-like behaviorPKR deficiency alters E. coli-induced sickness behaviors but does not exacerbate neuroimmune responses or bacterial loadAscending caudal medullary catecholamine pathways drive sickness-induced deficits in exploratory behavior: brain substrates for fatigue?Lipopolysaccharide-induced depressive-like behavior is associated with α₁-adrenoceptor dependent downregulation of the membrane GluR1 subunit in the mouse medial prefrontal cortex and ventral tegmental areaBehavioral and monoamine perturbations in adult male mice with chronic inflammation induced by repeated peripheral lipopolysaccharide administration.Neurotoxic kynurenine metabolism is increased in the dorsal hippocampus and drives distinct depressive behaviors during inflammation.The puzzle of depression and acute coronary syndrome: reviewing the role of acute inflammation.Deficient adolescent social behavior following early-life inflammation is ameliorated by augmentation of anandamide signaling.Prolonged depression-like behavior caused by immune challenge: influence of mouse strain and social environment.Inoculation of Bacillus Calmette-Guerin to mice induces an acute episode of sickness behavior followed by chronic depressive-like behavior.Cognitive and emotional alterations are related to hippocampal inflammation in a mouse model of metabolic syndrome.Central administration of lipopolysaccharide induces depressive-like behavior in vivo and activates brain indoleamine 2,3 dioxygenase in murine organotypic hippocampal slice culturesInflammation-associated depression: from serotonin to kynurenineLipopolysaccharide reduces incentive motivation while boosting preference for high reward in miceLong-term exercise is a potent trigger for ΔFosB induction in the hippocampus along the dorso-ventral axis.Fractalkine receptor (CX3CR1) deficiency sensitizes mice to the behavioral changes induced by lipopolysaccharideImmune suppression and immune activation in depressionCancer induces inflammation and depressive-like behavior in the mouse: modulation by social housing.A biological pathway linking inflammation and depression: activation of indoleamine 2,3-dioxygenase.Lipopolysaccharide-induced depressive-like behavior is mediated by indoleamine 2,3-dioxygenase activation in mice.Immune system to brain signaling: neuropsychopharmacological implications.Neurochemical Changes in the Mouse Hippocampus Underlying the Antidepressant Effect of Genetic Deletion of P2X7 ReceptorsNeuroimmune pharmacology as a sub-discipline of medical neuroscience in the medical school curriculum.Evidence from knockout mice that neuropeptide-Y Y2 and Y4 receptor signalling prevents long-term depression-like behaviour caused by immune challengeImplication of neuropeptide-Y Y2 receptors in the effects of immune stress on emotional, locomotor and social behavior of mice.
P2860
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P2860
Lipopolysaccharide induces delayed FosB/DeltaFosB immunostaining within the mouse extended amygdala, hippocampus and hypothalamus, that parallel the expression of depressive-like behavior.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Lipopolysaccharide induces del ...... n of depressive-like behavior.
@ast
Lipopolysaccharide induces del ...... n of depressive-like behavior.
@en
type
label
Lipopolysaccharide induces del ...... n of depressive-like behavior.
@ast
Lipopolysaccharide induces del ...... n of depressive-like behavior.
@en
prefLabel
Lipopolysaccharide induces del ...... n of depressive-like behavior.
@ast
Lipopolysaccharide induces del ...... n of depressive-like behavior.
@en
P2093
P2860
P50
P1476
Lipopolysaccharide induces del ...... on of depressive-like behavior
@en
P2093
Charlotte Micon
François Frenois
Jacques Lestage
Jason O'Connor
Marc Lawson
Maïté Moreau
P2860
P304
P356
10.1016/J.PSYNEUEN.2007.03.005
P577
2007-05-04T00:00:00Z