Glutathione/thioredoxin systems modulate mitochondrial H2O2 emission: an experimental-computational study. - Wikidata - awgldk - TriplyDB

Glutathione/thioredoxin systems modulate mitochondrial H2O2 emission: an experimental-computational study.

Glutathione/thioredoxin systems modulate mitochondrial H2O2 emission: an experimental-computational study.

http://www.wikidata.org/entity/Q35995736

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Mitochondrial Redox Dysfunction and Environmental Exposures Computational modeling analysis of mitochondrial superoxide production under varying substrate conditions and upon inhibition of different segments of the electron transport chain.Computational Models of Reactive Oxygen Species as Metabolic Byproducts and Signal-Transduction Modulators A computational model of reactive oxygen species and redox balance in cardiac mitochondria Protective mechanisms of mitochondria and heart function in diabetes Mitochondrial dynamics in diabetic cardiomyopathy Functional and morphological changes in endocrine pancreas following cola drink consumption in rats Pathogenic role of oxidative and nitrosative stress in primary biliary cirrhosis.A mechanistic mathematical model for the catalytic action of glutathione peroxidase.Hydrogen gas protects IP3Rs by reducing disulfide bridges in human keratinocytes under oxidative stress Mitochondrial respiration and ROS emission during β-oxidation in the heart: An experimental-computational study.Complex oscillatory redox dynamics with signaling potential at the edge between normal and pathological mitochondrial function.Mitochondrial reactive oxygen species (ROS) and ROS-induced ROS release.Mitochondrial ion channels/transporters as sensors and regulators of cellular redox signaling.Mitochondrial and cellular mechanisms for managing lipid excess.The impact of age-related dysregulation of the angiotensin system on mitochondrial redox balance Possible involvement of nitric oxide and reactive oxygen species in glucose deprivation-induced activation of transcription factor rst2.Restoring redox balance enhances contractility in heart trabeculae from type 2 diabetic rats exposed to high glucose.Can endurance exercise preconditioning prevention disuse muscle atrophy?Distinct mPTP activation mechanisms in ischaemia-reperfusion: contributions of Ca2+, ROS, pH, and inorganic polyphosphate.Dynamic Redox Regulation of IL-4 Signaling.Quantitative analyses of the hepatic proteome of methylmercury-exposed Atlantic cod (Gadus morhua) suggest oxidative stress-mediated effects on cellular energy metabolism Redox mechanisms of cardiomyocyte mitochondrial protection.Reperfusion injury and reactive oxygen species: The evolution of a concept.ErbB2 overexpression upregulates antioxidant enzymes, reduces basal levels of reactive oxygen species, and protects against doxorubicin cardiotoxicity.GSH or palmitate preserves mitochondrial energetic/redox balance, preventing mechanical dysfunction in metabolically challenged myocytes/hearts from type 2 diabetic mice O-GlcNAcomic Profiling Identifies Widespread O-Linked β-N-Acetylglucosamine Modification (O-GlcNAcylation) in Oxidative Phosphorylation System Regulating Cardiac Mitochondrial Function OXPHOS mutations and neurodegeneration Integrating mitochondrial energetics, redox and ROS metabolic networks: a two-compartment model Catalytic Coupling of Oxidative Phosphorylation, ATP Demand, and Reactive Oxygen Species Generation Compartment-specific Control of Reactive Oxygen Species Scavenging by Antioxidant Pathway Enzymes.Analysis of the kinetics and bistability of ubiquinol:cytochrome c oxidoreductase A biophysically based mathematical model for the catalytic mechanism of glutathione reductase.Synergy in free radical generation is blunted by high-fat diet induced alterations in skeletal muscle mitochondrial metabolism.An integrated mitochondrial ROS production and scavenging model: implications for heart failure.Redox-optimized ROS balance and the relationship between mitochondrial respiration and ROS.The glutathione system: a new drug target in neuroimmune disorders.Role of endothelial cells in bovine mammary gland health and disease.Modelling the molecular mechanisms of aging.An Advanced Omic Approach to Identify Co-Regulated Clusters and Transcription Regulation Network with AGCT and SHOE Methods.

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P2860

Glutathione/thioredoxin systems modulate mitochondrial H2O2 emission: an experimental-computational study.

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Glutathione/thioredoxin system ...... erimental-computational study.

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Brian Alan Stanley

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Brian O'Rourke

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Jackelyn Melissa Kembro

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Miguel Antonio Aon

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Nazareno Paolocci

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Sonia Cortassa

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Vidhya Sivakumaran

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P2860

Mitochondrial respiratory chain-dependent generation of superoxide anion and its release into the intermembrane space

How mitochondria produce reactive oxygen species

The Nrf2-antioxidant response element signaling pathway and its activation by oxidative stress

Oxidative stress and diabetes: what can we learn about insulin resistance from antioxidant mutant mouse models?

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

The putative glutathione peroxidase gene of Plasmodium falciparum codes for a thioredoxin peroxidase

Peroxiredoxin, a novel family of peroxidases

Enzymes or redox couples? The kinetics of thioredoxin and glutaredoxin reactions in a systems biology context

The role of oxidized cytochrome c in regulating mitochondrial reactive oxygen species production and its perturbation in ischaemia

Mitochondrial respiratory chain and thioredoxin reductase regulate intermembrane Cu,Zn-superoxide dismutase activity: implications for mitochondrial energy metabolism and apoptosis

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