Identification of App1 as a regulator of phagocytosis and virulence of Cryptococcus neoformans - Wikidata - awgldk - TriplyDB

Identification of App1 as a regulator of phagocytosis and virulence of Cryptococcus neoformans

Identification of App1 as a regulator of phagocytosis and virulence of Cryptococcus neoformans

http://www.wikidata.org/entity/Q36001291

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Molecular mechanisms of cryptococcal meningitis Cryptococcal cell morphology affects host cell interactions and pathogenicity Anti-Immune Strategies of Pathogenic Fungi Role of sphingomyelin synthase in controlling the antimicrobial activity of neutrophils against Cryptococcus neoformans Atf2 transcription factor binds to the APP1 promoter in Cryptococcus neoformans: stimulatory effect of diacylglycerol Cryptococcal interactions with the host immune system.Direct cell-to-cell spread of a pathogenic yeast.Phagocytosis of Cryptococcus neoformans by, and nonlytic exocytosis from, Acanthamoeba castellanii.Exploiting amoeboid and non-vertebrate animal model systems to study the virulence of human pathogenic fungi.Cryptococcus neoformans Kin1 protein kinase homologue, identified through a Caenorhabditis elegans screen, promotes virulence in mammals.Insights into the mechanisms of protective immunity against Cryptococcus neoformans infection using a mouse model of pulmonary cryptococcosis Elucidation of the Role of 3-Hydroxy Fatty Acids in Cryptococcus-amoeba Interactions.Paradoxical roles of alveolar macrophages in the host response to Cryptococcus neoformans.RNA biology and the adaptation of Cryptococcus neoformans to host temperature and stress The absence of serum IgM enhances the susceptibility of mice to pulmonary challenge with Cryptococcus neoformans.Cryptococcus neoformans modulates extracellular killing by neutrophils.Role of sphingolipids in microbial pathogenesis In vivo role of dendritic cells in a murine model of pulmonary cryptococcosis The glucose sensor-like protein Hxs1 is a high-affinity glucose transporter and required for virulence in Cryptococcus neoformans Role of glucose in the expression of Cryptococcus neoformans antiphagocytic protein 1, App1.A major role for capsule-independent phagocytosis-inhibitory mechanisms in mammalian infection by Cryptococcus neoformans.Evidence of a role for monocytes in dissemination and brain invasion by Cryptococcus neoformans.Cryptococcus neoformans glucuronoxylomannan fractions of different molecular masses are functionally distinct.Cytokine signaling regulates the outcome of intracellular macrophage parasitism by Cryptococcus neoformans.The cryptococcal enzyme inositol phosphosphingolipid-phospholipase C confers resistance to the antifungal effects of macrophages and promotes fungal dissemination to the central nervous system Capsule structure changes associated with Cryptococcus neoformans crossing of the blood-brain barrier Role of sphingosine-1-phosphate (S1P) and S1P receptor 2 in the phagocytosis of Cryptococcus neoformans by alveolar macrophages Dynamics of Cryptococcus neoformans-macrophage interactions reveal that fungal background influences outcome during cryptococcal meningoencephalitis in humans Human immunoglobulin G2 (IgG2) and IgG4, but not IgG1 or IgG3, protect mice against Cryptococcus neoformans infection.Adaptation of Cryptococcus neoformans to mammalian hosts: integrated regulation of metabolism and virulence.Role of phagocytosis in the virulence of Cryptococcus neoformans.Morphology and its underlying genetic regulation impact the interaction between Cryptococcus neoformans and its hosts Biosynthesis and immunogenicity of glucosylceramide in Cryptococcus neoformans and other human pathogens.Depletion of alveolar macrophages decreases the dissemination of a glucosylceramide-deficient mutant of Cryptococcus neoformans in immunodeficient mice Deciphering the model pathogenic fungus Cryptococcus neoformans.Aimless mutants of Cryptococcus neoformans: failure to disseminate.CNLAC1 is required for extrapulmonary dissemination of Cryptococcus neoformans but not pulmonary persistence.Loss of allergen 1 confers a hypervirulent phenotype that resembles mucoid switch variants of Cryptococcus neoformans Complement and fungal pathogens: an update.The elastic properties of the Cryptococcus neoformans capsule.

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Identification of App1 as a regulator of phagocytosis and virulence of Cryptococcus neoformans

http://www.wikidata.org/entity/Q36001291

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Identification of App1 as a re ...... nce of Cryptococcus neoformans

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Journal of Clinical Investigation

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Identification of App1 as a re ...... nce of Cryptococcus neoformans

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Beatriz Martinez-Mariño

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Benjamin Bolaños

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Chiara Luberto

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Daniel Taraskiewicz

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Dena L Toffaletti

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Edward Balish

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Gary M Cox

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Maurizio Del Poeta

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Pasquale Chitano

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Yusuf A Hannun

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Idiopathic CD4+ T-lymphocytopenia--four patients with opportunistic infections and no evidence of HIV infection

Cryptococcus neoformans resides in an acidic phagolysosome of human macrophages

Combined histochemical stains in the differential diagnosis of Cryptococcus neoformans

The PROSITE database, its status in 2002

MUC7 20-Mer: investigation of antimicrobial activity, secondary structure, and possible mechanism of antifungal action

Replication of Cryptococcus neoformans in macrophages is accompanied by phagosomal permeabilization and accumulation of vesicles containing polysaccharide in the cytoplasm

Genetic control of susceptibility to Cryptococcus neoformans in mice.

In vitro assessment of antifungal therapeutic potential of salivary histatin-5, two variants of histatin-5, and salivary mucin (MUC7) domain 1.

Intracellular parasitism of macrophages by Cryptococcus neoformans.

Growth of Cryptococcus neoformans within human macrophages in vitro

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7

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14523045

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antiphagocytic protein 1

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198528

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