about
Antibiotics for treating infected burn woundsIntestinal alkaline phosphatase is a gut mucosal defense factor maintained by enteral nutritionNrf2 plays a pivotal role in protection against burn trauma-induced intestinal injury and deathThe Emerging Therapeutic Role of Medical Foods for Gastrointestinal Disorders.Alcohol, inflammation, and gut-liver-brain interactions in tissue damage and disease development.Membership and behavior of ultra-low-diversity pathogen communities present in the gut of humans during prolonged critical illness.An alteration of the gut-liver axis drives pulmonary inflammation after intoxication and burn injury in mice.Recombinant factor XIII diminishes multiple organ dysfunction in rats caused by gut ischemia-reperfusion injury.Burn wound infections.Valproic acid treatment inhibits hypoxia-inducible factor 1α accumulation and protects against burn-induced gut barrier dysfunction in a rodent modelInflammatory response in multiple organs in a mouse model of acute alcohol intoxication and burn injury.The dynamics of the early inflammatory response in double-hit burn and sepsis animal modelsmicroRNA-98 mediated microvascular hyperpermeability during burn shock phase via inhibiting FIH-1.Liver disease in burn injury: evidence from a national sample of 31,338 adult patients.High-dose ascorbate with low-dose amphotericin B attenuates severity of disease in a model of the reappearance of candidemia during sepsis in the mouse.Long-term dynamic profiling of inflammatory mediators in double-hit burn and sepsis animal models.Burn-induced gut barrier injury is attenuated by phosphodiesterase inhibition: effects on tight junction structural proteins.Immunologic responses to critical injury and sepsis.Cellular basis of burn-induced cardiac dysfunction and prevention by mesenteric lymph duct ligation.Noninvasive measurement of intestinal inflammation after burn injury.Role of non-muscle myosin light chain kinase in neutrophil-mediated intestinal barrier dysfunction during thermal injuryOrexigenic hormone ghrelin ameliorates gut barrier dysfunction in sepsis in rats.Effects of Pediatric Burns on Gastrointestinal Diseases: A Population-Based Study.Regional variation in expression of pro-inflammatory mediators in the intestine following a combined insult of alcohol and burn injury.Toll-like receptor-4 mediates intestinal barrier breakdown after thermal injury.TNF-α induces vectorial secretion of IL-8 in Caco-2 cells.Bactericidal/permeability increasing protein attenuates the myocardial inflammation/dysfunction that occurs with burn complicated by subsequent infection.Intestinal dipeptide absorption is preserved during thermal injury and cytokine treatment.Dysregulation of microRNA biogenesis in the small intestine after ethanol and burn injury.Alcohol Modulation of the Postburn Hepatic Response.
P2860
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P2860
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Role of the gastrointestinal tract in burn sepsis.
@ast
Role of the gastrointestinal tract in burn sepsis.
@en
type
label
Role of the gastrointestinal tract in burn sepsis.
@ast
Role of the gastrointestinal tract in burn sepsis.
@en
prefLabel
Role of the gastrointestinal tract in burn sepsis.
@ast
Role of the gastrointestinal tract in burn sepsis.
@en
P1476
Role of the gastrointestinal tract in burn sepsis.
@en
P2093
Ankush Gosain
Richard L Gamelli
P356
10.1097/01.BCR.0000150212.21651.79
P577
2005-01-01T00:00:00Z