Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury. - Wikidata - awgldk - TriplyDB

Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury.

Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury.

http://www.wikidata.org/entity/Q36013685

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Plasmacytoid dendritic cells control lung inflammation and monocyte recruitment in indirect acute lung injury in mice Differential role of the Fas/Fas ligand apoptotic pathway in inflammation and lung fibrosis associated with reovirus 1/L-induced bronchiolitis obliterans organizing pneumonia and acute respiratory distress syndrome.Milk fat globule epidermal growth factor 8 attenuates acute lung injury in mice after intestinal ischemia and reperfusion Ciglitazone, a novel inhibitor of lung apoptosis following hemorrhagic shock.Febrile-range hyperthermia augments lipopolysaccharide-induced lung injury by a mechanism of enhanced alveolar epithelial apoptosis Effects of Fas-ligand overexpression on alveolar type II cell growth kinetics in perinatal murine lungs.Fas (CD95) induces rapid, TLR4/IRAK4-dependent release of pro-inflammatory HMGB1 from macrophages Protective effect of sodium tanshinone IIA sulfonate on injury of small intestine in rats with sepsis and its mechanism.Clinical pathology of the shock syndromes.Pathogenesis of indirect (secondary) acute lung injury.Death receptors mediate the adverse effects of febrile-range hyperthermia on the outcome of lipopolysaccharide-induced lung injury.Experimental models and emerging hypotheses for acute lung injury.Fas determines differential fates of resident and recruited macrophages during resolution of acute lung injury.Fas activation in alveolar epithelial cells induces KC (CXCL1) release by a MyD88-dependent mechanism.Disruption of fas-fas ligand signaling, apoptosis, and innate immunity by bacterial pathogens Role of the Fas/FasL system in a model of RSV infection in mechanically ventilated mice.AICAR attenuates organ injury and inflammatory response after intestinal ischemia and reperfusion.Mechanisms of indirect acute lung injury: a novel role for the coinhibitory receptor, programmed death-1.Involvement of Rho kinase (ROCK) in sepsis-induced acute lung injury.Kallistatin protects against sepsis-related acute lung injury via inhibiting inflammation and apoptosis.The role and source of tumor necrosis factor-α in hemorrhage-induced priming for septic lung injury.Role of Complement C5 in Experimental Blunt Chest Trauma-Induced Septic Acute Lung Injury (ALI).HMGB1 release triggered by the interaction of live retinal cells and uveitogenic T cells is Fas/FasL activation-dependent.Association of FAS -670A/G and FASL -843C/T Gene Polymorphisms on Allograft Nephropathy in Pediatric Renal Transplant Patients.The tyrosine kinase inhibitor imatinib prevents lung injury and death after intravenous LPS in mice.Therapeutic accessibility of caspase-mediated cell death as a key pathomechanism in indirect acute lung injury Depletion of resident alveolar macrophages does not prevent Fas-mediated lung injury in mice.Lung epithelial apoptosis in influenza virus pneumonia: the role of macrophage-expressed TNF-related apoptosis-inducing ligand.Apoptosis in the lung: induction, clearance and detection.Epithelial cell apoptosis and neutrophil recruitment in acute lung injury-a unifying hypothesis? What we have learned from small interfering RNAs.Update in critical care 2007.Trauma-hemorrhagic shock-induced pulmonary epithelial and endothelial cell injury utilizes different programmed cell death signaling pathways.Soluble programmed cell death receptor-1 (sPD-1): a potential biomarker with anti-inflammatory properties in human and experimental acute respiratory distress syndrome (ARDS).Contribution of neutrophils to acute lung injury Neutrophils in local and systemic antibody-dependent inflammatory and anaphylactic reactions.Life in the Fas lane: differential outcomes of Fas signaling.Mustard Gas Inhalation Injury: Therapeutic Strategy.Inhibition of Prolyl Hydroxylase Attenuates Fas Ligand-Induced Apoptosis and Lung Injury in Mice.Annexin A5 promotes macrophage activation and contributes to pulmonary fibrosis induced by silica particles.NF-κB RNAi decreases the Bax/Bcl-2 ratio and inhibits TNF-α-induced apoptosis in human alveolar epithelial cells.

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Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury.

http://www.wikidata.org/entity/Q36013685

description

2007 nî lūn-bûn

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name

Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury.

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Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury.

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type

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Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury.

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Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury.

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Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury.

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Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury.

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American Journal of Respiratory and Critical Care Medicine

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Fas-induced pulmonary apoptosis and inflammation during indirect acute lung injury.

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Chun-Shiang Chung

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Joanne Lomas-Neira

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Mario Perl

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Monique de Paepe

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Ulrike Perl

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William G Cioffi

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P2860

Fas and fas ligand are up-regulated in pulmonary edema fluid and lung tissue of patients with acute lung injury and the acute respiratory distress syndrome

The caspase-8 inhibitor FLIP promotes activation of NF-kappaB and Erk signaling pathways

Cellular FLICE-inhibitory protein splice variants inhibit different steps of caspase-8 activation at the CD95 death-inducing signaling complex

A synthetic inhibitor of the mitogen-activated protein kinase cascade

Resolution of lung inflammation by CD44

Macrophages that have ingested apoptotic cells in vitro inhibit proinflammatory cytokine production through autocrine/paracrine mechanisms involving TGF-beta, PGE2, and PAF

Hyperoxia-induced apoptosis and Fas/FasL expression in lung epithelial cells.

Consensus conference definitions for sepsis, septic shock, acute lung injury, and acute respiratory distress syndrome: time for a reevaluation.

Immunocytochemical detection and mapping of a cytokeratin 18 neo-epitope exposed during early apoptosis.

The death domain superfamily in intracellular signaling of apoptosis and inflammation.

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apoptotic process

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1994224

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