Chop deficiency prevents UUO-induced renal fibrosis by attenuating fibrotic signals originated from Hmgb1/TLR4/NFκB/IL-1β signaling.
about
Vascular Remodelling and Mesenchymal Transition in Systemic SclerosisNupr1/Chop signal axis is involved in mitochondrion-related endothelial cell apoptosis induced by methamphetamineHMGB1 exacerbates bronchiolitis obliterans syndrome via RAGE/NF-κB/HPSE signaling to enhance latent TGF-β release from ECM.C/EBP homologous protein (CHOP) deficiency ameliorates renal fibrosis in unilateral ureteral obstructive kidney disease.Endoplasmic reticulum stress in bone marrow-derived cells prevents acute cardiac inflammation and injury in response to angiotensin II.Endoplasmic reticulum stress, a new wrestler, in the pathogenesis of idiopathic pulmonary fibrosis.Proinflammatory effect of high-mobility group protein B1 on keratinocytes: an autocrine mechanism underlying psoriasis development.Bacteroides fragilis Enterotoxin Induces Formation of Autophagosomes in Endothelial Cells but Interferes with Fusion with Lysosomes for Complete Autophagic Flux through a Mitogen-Activated Protein Kinase-, AP-1-, and C/EBP Homologous Protein-DependeHMGB1 induces lung fibroblast to myofibroblast differentiation through NF‑κB‑mediated TGF‑β1 release.Endoplasmic reticulum stress inhibition attenuates hypertensive chronic kidney disease through reduction in proteinuria.Both conditional ablation and overexpression of E2 SUMO-conjugating enzyme (UBC9) in mouse pancreatic beta cells result in impaired beta cell function.Transcription Factor C/EBP Homologous Protein in Health and Diseases.Endogenous ligands of TLR4 promote unresolving tissue fibrosis: Implications for systemic sclerosis and its targeted therapy.High Uric Acid-Induced Epithelial-Mesenchymal Transition of Renal Tubular Epithelial Cells via the TLR4/NF-kB Signaling Pathway.
P2860
Q26749091-ABDD10A5-57E1-4AF0-A3B9-9D82596C76E6Q36775049-C781C596-7130-4AA9-A4A8-E3A0F7D2CCB4Q36964047-9FA098DF-B862-4101-B234-E12E07EDC0D1Q37225434-1C2FBD34-2FA6-418E-91BB-EEDDD42A8B08Q37482793-5AF8D455-25A8-45C1-96ED-4E5D161D5A3BQ37685292-3C6FCBF6-735F-4501-9C89-D88744048F14Q38794037-2C890286-7BCB-475C-883A-33DC4618F882Q40140848-EA6AA94D-095D-40F9-AF15-5BA054FE1BF7Q41848319-84C1ED3A-3A87-41F6-84B8-F468E83F8A3BQ42170580-843BD7B2-9D8E-4DAB-B77F-CA2F14A724C0Q47233632-9CCD8E54-A7B0-482F-A983-113DE3271A67Q47304202-4C0364A3-E466-44DC-855D-AB090C7F3EFFQ47753846-3E4B938C-377B-42E0-9E68-A498BACBF30CQ47826501-5BBC23D0-3494-4A77-B262-823C635E7237
P2860
Chop deficiency prevents UUO-induced renal fibrosis by attenuating fibrotic signals originated from Hmgb1/TLR4/NFκB/IL-1β signaling.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@ast
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@en
type
label
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@ast
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@en
prefLabel
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@ast
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@en
P2093
P2860
P356
P1476
Chop deficiency prevents UUO-i ...... gb1/TLR4/NFκB/IL-1β signaling.
@en
P2093
P2860
P2888
P356
10.1038/CDDIS.2015.206
P577
2015-08-06T00:00:00Z
P5875
P6179
1001706538