Viral targeting of fibroblastic reticular cells contributes to immunosuppression and persistence during chronic infection. - Wikidata - awgldk - TriplyDB

Viral targeting of fibroblastic reticular cells contributes to immunosuppression and persistence during chronic infection.

Viral targeting of fibroblastic reticular cells contributes to immunosuppression and persistence during chronic infection.

http://www.wikidata.org/entity/Q36024206

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Modes of Antigen Presentation by Lymph Node Stromal Cells and Their Immunological Implications Molecular and cellular insights into T cell exhaustion Stromal cell-based immunotherapy in transplantation The effects of TLR activation on T-cell development and differentiation Dynamic Imaging of CD8(+) T cells and dendritic cells during infection with Toxoplasma gondii Stromal cell regulation of homeostatic and inflammatory lymphoid organogenesis Stromal Fibroblasts in Tertiary Lymphoid Structures: A Novel Target in Chronic Inflammation Lymph node fibroblastic reticular cells in health and disease Inhibition of receptor tyrosine kinases restores immunocompetence and improves immune-dependent chemotherapy against experimental leishmaniasis in mice.Type I interferon suppresses virus-specific B cell responses by modulating CD8(+) T cell differentiation Platelets support a protective immune response to LCMV by preventing splenic necrosis.Anatomy of viral persistence.Short-term inhibition of p53 combined with keratinocyte growth factor improves thymic epithelial cell recovery and enhances T-cell reconstitution after murine bone marrow transplantation Replicating viral vector platform exploits alarmin signals for potent CD8+ T cell-mediated tumour immunotherapy.Lymph node fibroblastic reticular cells directly present peripheral tissue antigen under steady-state and inflammatory conditions.PD-L1 has distinct functions in hematopoietic and nonhematopoietic cells in regulating T cell responses during chronic infection in mice.Regulation of T cell priming by lymphoid stroma.Fibroblastic reticular cells from lymph nodes attenuate T cell expansion by producing nitric oxide.Interplay between regulatory T cells and PD-1 in modulating T cell exhaustion and viral control during chronic LCMV infection.Minimal effect of CD103 expression on the control of a chronic antiviral immune response.IFN-λ exerts opposing effects on T cell responses depending on the chronicity of the virus infection.Normalization of the lymph node T cell stromal microenvironment in lpr/lpr mice is associated with SU5416-induced reduction in autoantibodies.Influence of the fibroblastic reticular network on cell-cell interactions in lymphoid organs.Nonhematopoietic antigen blocks memory programming of alloreactive CD8+ T cells and drives their eventual exhaustion in mouse models of bone marrow transplantation.Stromal and hematopoietic cells in secondary lymphoid organs: partners in immunity.Cell-intrinsic transforming growth factor-beta signaling mediates virus-specific CD8+ T cell deletion and viral persistence in vivo Decoding arenavirus pathogenesis: essential roles for alpha-dystroglycan-virus interactions and the immune response.NK cells and their ability to modulate T cells during virus infections Early virus-host interactions dictate the course of a persistent infection Cell-mediated immune responses in tuberculosis.Stromal cell contributions to the homeostasis and functionality of the immune system.Lymphocytic choriomeningitis virus persistence promotes effector-like memory differentiation and enhances mucosal T cell distribution Enhanced CD8 T cell responses through GITR-mediated costimulation resolve chronic viral infection.Blockade of PD-1/PD-L1 promotes adoptive T-cell immunotherapy in a tolerogenic environment.Follicular dendritic cells: dynamic antigen libraries.Of mice, macaques and men: scaling of virus dynamics and immune responses Lymph node fibroblastic reticular cell transplants show robust therapeutic efficacy in high-mortality murine sepsis Tight regulation of memory CD8(+) T cells limits their effectiveness during sustained high viral load Microbiota-Dependent Sequelae of Acute Infection Compromise Tissue-Specific Immunity Prostaglandin E2 and programmed cell death 1 signaling coordinately impair CTL function and survival during chronic viral infection.

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P2860

Viral targeting of fibroblastic reticular cells contributes to immunosuppression and persistence during chronic infection.

http://www.wikidata.org/entity/Q36024206

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Viral targeting of fibroblasti ...... ence during chronic infection.

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Arlene H Sharpe

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Christian P Larsen

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Daniel M Clemens

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Gordon J Freeman

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Mehrdad Matloubian

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Rafi Ahmed

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Scott N Mueller

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Shivaprakash Gangappa

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P2860

Viral immune evasion due to persistence of activated T cells without effector function

Specific remodeling of splenic architecture by cytomegalovirus.

Restoring function in exhausted CD8 T cells during chronic viral infection

Lymph node fibroblastic reticular cells construct the stromal reticulum via contact with lymphocytes

Distribution of dystroglycan in normal adult mouse tissues

Stromal cell networks regulate lymphocyte entry, migration, and territoriality in lymph nodes

Characterization and cloning of a novel glycoprotein expressed by stromal cells in T-dependent areas of peripheral lymphoid tissues.

General and specific immunosuppression caused by antiviral T-cell responses.

Generalized immunosuppression: how viruses undermine the immune response.

Spatial and molecular organization of lymph node T cell cortex: a labyrinthine cavity bounded by an epithelium-like monolayer of fibroblastic reticular cells anchored to basement membrane-like extracellular matrix.

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104

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17878315

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immunosuppression

viral persistence

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2000533

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