Mast cells modulate the pathogenesis of elastase-induced abdominal aortic aneurysms in mice - Wikidata - awgldk - TriplyDB

Mast cells modulate the pathogenesis of elastase-induced abdominal aortic aneurysms in mice

Mast cells modulate the pathogenesis of elastase-induced abdominal aortic aneurysms in mice

http://www.wikidata.org/entity/Q36025276

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New developments in mast cell biology Mast Cell: A Multi-Functional Master Cell Emerging role of mast cells and macrophages in cardiovascular and metabolic diseases Mast cells modulate acute toxoplasmosis in murine models Deficiency of cathepsin S attenuates angiotensin II-induced abdominal aortic aneurysm formation in apolipoprotein E-deficient mice Infection of mast cells with live streptococci causes a toll-like receptor 2- and cell-cell contact-dependent cytokine and chemokine response Fibulin-2 and fibulin-5 cooperatively function to form the internal elastic lamina and protect from vascular injury.Toll-like receptor 7 deficiency protects apolipoprotein E-deficient mice from diet-induced atherosclerosis Immunomodulatory mast cells: negative, as well as positive, regulators of immunity Regulation of endothelial cell adhesion molecule expression by mast cells, macrophages, and neutrophils.Non-coding RNA Contribution to Thoracic and Abdominal Aortic Aneurysm Disease Development and Progression.Increased neovascularization in mice lacking tissue inhibitor of metalloproteinases-3.Nanoparticle targeting to diseased vasculature for imaging and therapy.Cystatin C deficiency promotes inflammation in angiotensin II-induced abdominal aortic aneurisms in atherosclerotic mice.Development of mast cells and importance of their tryptase and chymase serine proteases in inflammation and wound healing.IgE actions on CD4+ T cells, mast cells, and macrophages participate in the pathogenesis of experimental abdominal aortic aneurysms.Vascular smooth muscle cell peroxisome proliferator-activated receptor-γ deletion promotes abdominal aortic aneurysms Kit (W-sh) mice develop earlier and more severe experimental autoimmune encephalomyelitis due to absence of immune suppression.Lack of EP4 receptors on bone marrow-derived cells enhances inflammation in atherosclerotic lesions.Assessment of elastase-induced murine abdominal aortic aneurysms: comparison of ultrasound imaging with in situ video microscopy.Animal models of cardiovascular diseases Complement regulator CD59 protects against angiotensin II-induced abdominal aortic aneurysms in mice Cathepsin G deficiency reduces periaortic calcium chloride injury-induced abdominal aortic aneurysms in mice.Identification of Hic-5 as a novel scaffold for the MKK4/p54 JNK pathway in the development of abdominal aortic aneurysms.Mast cell tryptase deficiency attenuates mouse abdominal aortic aneurysm formation Tumor necrosis factor-like weak inducer of apoptosis or Fn14 deficiency reduce elastase perfusion-induced aortic abdominal aneurysm in mice.Receptor-interacting protein kinase 3 contributes to abdominal aortic aneurysms via smooth muscle cell necrosis and inflammation Cysteinyl cathepsins and mast cell proteases in the pathogenesis and therapeutics of cardiovascular diseases.Mast cells and metabolic syndrome.Non coding RNAs in aortic aneurysmal disease.Mast Cell Proteases and Inflammation Cathepsin K deficiency reduces elastase perfusion-induced abdominal aortic aneurysms in mice.Cathepsins: a new culprit behind abdominal aortic aneurysm.Thrombospondin-1 (TSP1) contributes to the development of vascular inflammation by regulating monocytic cell motility in mouse models of abdominal aortic aneurysm.Cathepsin L activity is essential to elastase perfusion-induced abdominal aortic aneurysms in mice Mast cell stabilization: novel medication for obesity and diabetes.Cysteine Protease Cathepsins in Atherosclerosis and Abdominal Aortic Aneurysm.Inflammatory cell phenotypes in AAAs: their role and potential as targets for therapy.Functional alterations of myeloid cell subsets in hyperlipidaemia: relevance for atherosclerosis.Different roles of mast cells in obesity and diabetes: lessons from experimental animals and humans.

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Mast cells modulate the pathogenesis of elastase-induced abdominal aortic aneurysms in mice

http://www.wikidata.org/entity/Q36025276

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2007年論文

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2007年论文

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name

Mast cells modulate the pathog ...... minal aortic aneurysms in mice

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Mast cells modulate the pathog ...... minal aortic aneurysms in mice

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Mast cells modulate the pathog ...... minal aortic aneurysms in mice

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Mast cells modulate the pathog ...... minal aortic aneurysms in mice

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Mast cells modulate the pathog ...... minal aortic aneurysms in mice

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Mast cells modulate the pathog ...... minal aortic aneurysms in mice

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Mast cells modulate the pathog ...... minal aortic aneurysms in mice

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B Timothy Baxter

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Chongxiu Sun

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Galina K Sukhova

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Guo-Ping Shi

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Jian Liu

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Jiusong Sun

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Lindsey A MacFarlane

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Min Yang

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Paul J Wolters

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Rebecca Knispel

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P2860

Targeted gene disruption of matrix metalloproteinase-9 (gelatinase B) suppresses development of experimental abdominal aortic aneurysms

Molecular cloning and expression of human alveolar macrophage cathepsin S, an elastinolytic cysteine protease

Mast cell-deficient W-sash c-kit mutant Kit W-sh/W-sh mice as a model for investigating mast cell biology in vivo

A key role for mast cell chymase in the activation of pro-matrix metalloprotease-9 and pro-matrix metalloprotease-2.

Mast cells enhance T cell activation: Importance of mast cell-derived TNF.

MCP-1 and RANTES are mediators of acute and chronic inflammation.

The Wsh and Ph mutations affect the c-kit expression profile: c-kit misexpression in embryogenesis impairs melanogenesis in Wsh and Ph mutant mice.

Disruption of the cathepsin K gene reduces atherosclerosis progression and induces plaque fibrosis but accelerates macrophage foam cell formation.

Compound 48/80: a potent histamine liberator.

Deficiency of cathepsin S reduces atherosclerosis in LDL receptor-deficient mice

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17932568

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