Oncostatin M promotes mucosal epithelial barrier dysfunction, and its expression is increased in patients with eosinophilic mucosal disease - Wikidata - awgldk - TriplyDB

Oncostatin M promotes mucosal epithelial barrier dysfunction, and its expression is increased in patients with eosinophilic mucosal disease

Oncostatin M promotes mucosal epithelial barrier dysfunction, and its expression is increased in patients with eosinophilic mucosal disease

http://www.wikidata.org/entity/Q36037454

about

Pathogenic CD4+ T cells in patients with asthma.Immunopathogenesis of Chronic Rhinosinusitis and Nasal Polyposis.The Etiology and Pathogenesis of Chronic Rhinosinusitis: a Review of Current Hypotheses.Barrier function of the nasal mucosa in health and type-2 biased airway diseases.Chronic rhinosinusitis pathogenesis.Age-Related Decline of Neutrophilic Inflammation Is Associated with Better Postoperative Prognosis in Non-eosinophilic Nasal Polyps.Fstl1 Promotes Asthmatic Airway Remodeling by Inducing Oncostatin M.Transcriptome Analysis Reveals Distinct Gene Expression Profiles in Eosinophilic and Noneosinophilic Chronic Rhinosinusitis with Nasal Polyps.Chronic Rhinosinusitis with Nasal Polyps.International Consensus Statement on Allergy and Rhinology: Rhinosinusitis.Chronic rhinosinusitis: the rationale for current treatments.Classical complement pathway activation in the nasal tissue of patients with chronic rhinosinusitis.The barrier hypothesis and Oncostatin M: Restoration of epithelial barrier function as a novel therapeutic strategy for the treatment of type 2 inflammatory disease.Airway epithelial barrier dysfunction in the pathogenesis and prognosis of respiratory tract diseases in childhood and adulthood.Potential Involvement of the Epidermal Growth Factor Receptor Ligand Epiregulin and Matrix Metalloproteinase-1 in Pathogenesis of Chronic Rhinosinusitis.Airborne Particulate Matter Induces Nonallergic Eosinophilic Sinonasal Inflammation in Mice.Microparticles in nasal lavage fluids in chronic rhinosinusitis: Potential biomarkers for diagnosis of aspirin-exacerbated respiratory disease.Neutrophils are a major source of the epithelial barrier disrupting cytokine oncostatin M in patients with mucosal airways disease.Allergic Aspergillus Rhinosinusitis.Etiology of epithelial barrier dysfunction in patients with type 2 inflammatory diseases.Pathogenesis of Recalcitrant Chronic Rhinosinusitis: The Emerging Role of Innate Immune Cells.Recent advances in biologic therapy of asthma and the role in therapy of chronic rhinosinusitis.OSMR gene effect on the pathogenesis of chronic autoimmune Urticaria via the JAK/STAT3 pathway.Inflammatory Endotypes and Microbial Associations in Chronic Rhinosinusitis Biomarkers in Chronic Rhinosinusitis with Nasal Polyps Fibroblast-to-myofibroblast transition in bronchial asthma

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P2860

Oncostatin M promotes mucosal epithelial barrier dysfunction, and its expression is increased in patients with eosinophilic mucosal disease

http://www.wikidata.org/entity/Q36037454

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

Oncostatin M promotes mucosal ...... h eosinophilic mucosal disease

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Oncostatin M promotes mucosal ...... h eosinophilic mucosal disease

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type

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Oncostatin M promotes mucosal ...... h eosinophilic mucosal disease

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Oncostatin M promotes mucosal ...... h eosinophilic mucosal disease

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Oncostatin M promotes mucosal ...... h eosinophilic mucosal disease

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Oncostatin M promotes mucosal ...... h eosinophilic mucosal disease

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J.JACI.2015.01.043

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The Journal of Allergy and Clinical Immunology

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Oncostatin M promotes mucosal ...... h eosinophilic mucosal disease

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Anju T Peters

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Bruce K Tan

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David B Conley

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Erin Rocci

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James E Norton

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Kathleen E Harris

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Kathryn L Pothoven

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Lydia A Suh

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Mark C Liu

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Paul J Bryce

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P2860

Oncostatin M is a member of a cytokine family that includes leukemia-inhibitory factor, granulocyte colony-stimulating factor, and interleukin 6

Influence of subunit combinations on signaling by receptors for oncostatin M, leukemia inhibitory factor, and interleukin-6

The tight junction protein ZO-1 establishes a link between the transmembrane protein occludin and the actin cytoskeleton

Regulation of fibrinogen biosynthesis by cytokines, consequences on the vascular risk

Common loss-of-function variants of the epidermal barrier protein filaggrin are a major predisposing factor for atopic dermatitis

Soluble gp130 is the natural inhibitor of soluble interleukin-6 receptor transsignaling responses

Mucosal immunity and inflammation. V. Innate mechanisms of mucosal defense and repair: the best offense is a good defense.

Evidence of a role for B cell-activating factor of the TNF family in the pathogenesis of chronic rhinosinusitis with nasal polyps.

Evidence for altered activity of the IL-6 pathway in chronic rhinosinusitis with nasal polyps.

Evidence for diminished levels of epithelial psoriasin and calprotectin in chronic rhinosinusitis

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Nirmala Gonsalves

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