Suppression of tumorigenicity in simian virus 40-transformed 3T3 cells transfected with alpha-actinin cDNA.
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The human non-muscle alpha-actinin protein encoded by the ACTN4 gene suppresses tumorigenicity of human neuroblastoma cellsDynamics of alpha-actinin in focal adhesions and stress fibers visualized with alpha-actinin-green fluorescent proteinEPLIN regulates actin dynamics by cross-linking and stabilizing filamentsCDO: an oncogene-, serum-, and anchorage-regulated member of the Ig/fibronectin type III repeat familyActinin-4, a novel actin-bundling protein associated with cell motility and cancer invasionMutant alpha-actinin-4 promotes tumorigenicity and regulates cell motility of a human lung carcinomaCytoplasmic tail regulates the intercellular adhesion function of the epithelial cell adhesion moleculeIntegrating with integrinsIncreased expression of the Ras suppressor Rsu-1 enhances Erk-2 activation and inhibits Jun kinase activationFunctional differences between L- and T-plastin isoformsDifferential nuclear translocation and transactivation potential of beta-catenin and plakoglobinHAMLET binding to α-actinin facilitates tumor cell detachmentFHL3 is an actin-binding protein that regulates alpha-actinin-mediated actin bundling: FHL3 localizes to actin stress fibers and enhances cell spreading and stress fiber disassemblyIdentification of drm, a novel gene whose expression is suppressed in transformed cells and which can inhibit growth of normal but not transformed cells in culturev-Src suppresses SHPS-1 expression via the Ras-MAP kinase pathway to promote the oncogenic growth of cells.Submembranous junctional plaque proteins include potential tumor suppressor moleculesIdentification of Nd1, a novel murine kelch family protein, involved in stabilization of actin filaments.Isoform-specific contributions of alpha-actinin to glioma cell mechanobiology.Inhibition of anchorage-independent growth of transformed NIH3T3 cells by epithelial protein lost in neoplasm (EPLIN) requires localization of EPLIN to actin cytoskeleton.A role for Rho in Ras transformation.Tight junction protein ZO-2 is differentially expressed in normal pancreatic ducts compared to human pancreatic adenocarcinoma.Polymorphism of highly cross-linked F-actin networks: probing multiple length scales.Post-transcriptional down-regulation of ROCKI/Rho-kinase through an MEK-dependent pathway leads to cytoskeleton disruption in Ras-transformed fibroblasts.Reversible tumorigenesis induced by deficiency of vasodilator-stimulated phosphoprotein.Cell motility and local viscoelasticity of fibroblastsAdhesion systems in normal breast and in invasive breast carcinoma.Expression of tropomyosin isoforms in benign and malignant human breast lesions.Regulation of beta-catenin levels and localization by overexpression of plakoglobin and inhibition of the ubiquitin-proteasome system.Suppression of tumorigenicity in breast cancer cells by the microfilament protein profilin 1.Oncogenic Ras-induced morphologic change is through MEK/ERK signaling pathway to downregulate Stat3 at a posttranslational level in NIH3T3 cells.Expression of transduced tropomyosin 1 cDNA suppresses neoplastic growth of cells transformed by the ras oncogeneActinin-4 in keratinocytes regulates motility via an effect on lamellipodia stability and matrix adhesionsIsolation and characterization of a novel mitogenic regulatory gene, 322, which is transcriptionally suppressed in cells transformed by src and rasProfilin-1 is a negative regulator of mammary carcinoma aggressivenessTumor suppressor activity of profilin requires a functional actin binding site.Forced expression of tropomyosin 2 or 3 in v-Ki-ras-transformed fibroblasts results in distinct phenotypic effects.Tropomyosin-2 cDNA lacking the 3' untranslated region riboregulator induces growth inhibition of v-Ki-ras-transformed fibroblastsThe non-muscle functions of actinins: an update.Role of phosphatidylinositol 4,5-bisphosphate in Ras/Rac-induced disruption of the cortactin-actomyosin II complex and malignant transformation.Cytoskeletal organization in tropomyosin-mediated reversion of ras-transformation: Evidence for Rho kinase pathway.
P2860
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P2860
Suppression of tumorigenicity in simian virus 40-transformed 3T3 cells transfected with alpha-actinin cDNA.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
Suppression of tumorigenicity ...... ected with alpha-actinin cDNA.
@ast
Suppression of tumorigenicity ...... ected with alpha-actinin cDNA.
@en
type
label
Suppression of tumorigenicity ...... ected with alpha-actinin cDNA.
@ast
Suppression of tumorigenicity ...... ected with alpha-actinin cDNA.
@en
prefLabel
Suppression of tumorigenicity ...... ected with alpha-actinin cDNA.
@ast
Suppression of tumorigenicity ...... ected with alpha-actinin cDNA.
@en
P2093
P2860
P356
P1476
Suppression of tumorigenicity ...... ected with alpha-actinin cDNA.
@en
P2093
Ben-Ze'ev A
Kwiatkowski DJ
P2860
P304
P356
10.1073/PNAS.90.2.383
P407
P577
1993-01-01T00:00:00Z