AM-36 modulates the neutrophil inflammatory response and reduces breakdown of the blood brain barrier after endothelin-1 induced focal brain ischaemia.
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Inflammatory mechanisms in ischemic stroke: role of inflammatory cells.Cinnamaldehyde inhibits inflammation and brain damage in a mouse model of permanent cerebral ischaemia.Tracking the inflammatory response in stroke in vivo by sensing the enzyme myeloperoxidaseThrombosis, Neuroinflammation, and Poststroke Infection: The Multifaceted Role of Neutrophils in Stroke.NADPH Oxidase as a Therapeutic Target for Neuroprotection against Ischaemic Stroke: Future Perspectives.Osthole, a natural coumarin improves cognitive impairments and BBB dysfunction after transient global brain ischemia in C57 BL/6J mice: involvement of Nrf2 pathway.Pharmacokinetics and brain uptake of AM-36, a novel neuroprotective agent, following intravenous administration to rats
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P2860
AM-36 modulates the neutrophil inflammatory response and reduces breakdown of the blood brain barrier after endothelin-1 induced focal brain ischaemia.
description
2006 nî lūn-bûn
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2006年の論文
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2006年学术文章
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name
AM-36 modulates the neutrophil ...... induced focal brain ischaemia.
@ast
AM-36 modulates the neutrophil ...... induced focal brain ischaemia.
@en
type
label
AM-36 modulates the neutrophil ...... induced focal brain ischaemia.
@ast
AM-36 modulates the neutrophil ...... induced focal brain ischaemia.
@en
prefLabel
AM-36 modulates the neutrophil ...... induced focal brain ischaemia.
@ast
AM-36 modulates the neutrophil ...... induced focal brain ischaemia.
@en
P2093
P2860
P356
P1476
AM-36 modulates the neutrophil ...... induced focal brain ischaemia.
@en
P2093
J K Callaway
R M Weston
Y Ishizuka
P2860
P304
P356
10.1038/SJ.BJP.0706918
P407
P577
2006-10-03T00:00:00Z