IRF5 controls both acute and chronic inflammation. - Wikidata - awgldk - TriplyDB

IRF5 controls both acute and chronic inflammation.

IRF5 controls both acute and chronic inflammation.

http://www.wikidata.org/entity/Q36055462

about

Macrophage in chronic kidney disease Cannabinoid CB1 receptor overactivity contributes to the pathogenesis of idiopathic pulmonary fibrosis.Specific detection of interferon regulatory factor 5 (IRF5): A case of antibody inequality.Granulocyte macrophage colony-stimulating factor receptor α expression and its targeting in antigen-induced arthritis and inflammation IRF5 governs liver macrophage activation that promotes hepatic fibrosis in mice and humans.An oligodeoxynucleotide with AAAG repeats significantly attenuates burn-induced systemic inflammatory responses via inhibiting interferon regulatory factor 5 pathway IRF5 distinguishes severe asthma in humans and drives Th1 phenotype and airway hyperreactivity in mice Macrophage heterogeneity in the context of rheumatoid arthritis.Interferon Regulatory Factor 5 Controls Necrotic Core Formation in Atherosclerotic Lesions by Impairing Efferocytosis.NOX2-dependent ATM kinase activation dictates pro-inflammatory macrophage phenotype and improves effectiveness to radiation therapy.Driving chronicity in rheumatoid arthritis - perpetuating role of myeloid cells.Synovial tissue macrophages: friend or foe?Rheumatoid arthritis patient antibodies highly recognize IL-2 in the immune response pathway involving IRF5 and EBV antigens.Medical relevance of protein-truncating variants across 337,205 individuals in the UK Biobank study.Immunogene therapy with fusogenic nanoparticles modulates macrophage response to Staphylococcus aureus.Dual role of YM1+ M2 macrophages in allergic lung inflammation.Gene Therapy for Neuropathic Pain through siRNA-IRF5 Gene Delivery with Homing Peptides to Microglia Macrophages: friend or foe in idiopathic pulmonary fibrosis?

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IRF5 controls both acute and chronic inflammation.

http://www.wikidata.org/entity/Q36055462

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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name

IRF5 controls both acute and chronic inflammation.

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IRF5 controls both acute and chronic inflammation.

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IRF5 controls both acute and chronic inflammation.

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IRF5 controls both acute and chronic inflammation.

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IRF5 controls both acute and chronic inflammation.

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IRF5 controls both acute and chronic inflammation.

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Proceedings of the National Academy of Sciences of the United States of America

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IRF5 controls both acute and chronic inflammation

@en

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Dany Perocheau

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David G Saliba

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James E Pease

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Katrina Blazek

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Marc Feldmann

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Miriam Weiss

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P2860

IRF5 promotes inflammatory macrophage polarization and TH1-TH17 responses

Integral role of IRF-5 in the gene induction programme activated by Toll-like receptors

Analysis of the cell infiltrate and expression of matrix metalloproteinases and granzyme B in paired synovial biopsy specimens from the cartilage-pannus junction in patients with RA

Impaired monocyte migration and reduced type 1 (Th1) cytokine responses in C-C chemokine receptor 2 knockout mice

Macrophage activation and polarization: nomenclature and experimental guidelines

Polymorphonuclear granulocytes in rheumatic tissue destruction. III. an electron microscopic study of PMNs at the pannus-cartilage junction in rheumatoid arthritis.

Contribution of IRF5 in B cells to the development of murine SLE-like disease through its transcriptional control of the IgG2a locus.

Beyond stem cells: self-renewal of differentiated macrophages.

The pathogenesis and prevention of joint damage in rheumatoid arthritis: advances from synovial biopsy and tissue analysis.

IL-1α and IL-1β recruit different myeloid cells and promote different stages of sterile inflammation.

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