Norepinephrine causes epigenetic repression of PKCε gene in rodent hearts by activating Nox1-dependent reactive oxygen species production. - Wikidata - awgldk - TriplyDB

Norepinephrine causes epigenetic repression of PKCε gene in rodent hearts by activating Nox1-dependent reactive oxygen species production.

Norepinephrine causes epigenetic repression of PKCε gene in rodent hearts by activating Nox1-dependent reactive oxygen species production.

http://www.wikidata.org/entity/Q36055530

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Hepatoprotective Effects of Chinese Medicinal Herbs: A Focus on Anti-Inflammatory and Anti-Oxidative Activities Cardiovascular and Hepatic Toxicity of Cocaine: Potential Beneficial Effects of Modulators of Oxidative Stress Mechanisms of superoxide signaling in epigenetic processes: relation to aging and cancer Early developmental conditioning of later health and disease: physiology or pathophysiology?Transcriptional regulation of oncogenic protein kinase Cϵ (PKCϵ) by STAT1 and Sp1 proteins.Perinatal nicotine exposure increases angiotensin II receptor-mediated vascular contractility in adult offspring.Gestational hypoxia and epigenetic programming of brain development disorders.Chronic hypoxia during gestation enhances uterine arterial myogenic tone via heightened oxidative stress.Redox-regulated suppression of splenic T-lymphocyte activation in a model of sympathoexcitation.Hypoxia Represses ER-α Expression and Inhibits Estrogen-Induced Regulation of Ca2+-Activated K+ Channel Activity and Myogenic Tone in Ovine Uterine Arteries: Causal Role of DNA Methylation Norepinephrine-induced apoptotic and hypertrophic responses in H9c2 cardiac myoblasts are characterized by different repertoire of reactive oxygen species generation Structural impact of complete CpG methylation within target DNA on specific complex formation of the inducible transcription factor Egr-1.Protective Effect of Antenatal Antioxidant on Nicotine-Induced Heart Ischemia-Sensitive Phenotype in Rat Offspring Perinatal nicotine exposure increases vulnerability of hypoxic-ischemic brain injury in neonatal rats: role of angiotensin II receptors.Angiotensin II stimulates superoxide production in the thick ascending limb by activating NOX4.Antenatal hypoxia induces epigenetic repression of glucocorticoid receptor and promotes ischemic-sensitive phenotype in the developing heart.Role of the hypothalamic-pituitary-adrenal axis in developmental programming of health and disease.Hypoxia inhibits cardiomyocyte proliferation in fetal rat hearts via upregulating TIMP-4 Promoter methylation represses AT2R gene and increases brain hypoxic-ischemic injury in neonatal rats Zif268/Egr1 gain of function facilitates hippocampal synaptic plasticity and long-term spatial recognition memory.New nucleophilic mechanisms of ros-dependent epigenetic modifications: comparison of aging and cancer.Inhibition of DNA methylation reverses norepinephrine-induced cardiac hypertrophy in rats.Endothelin-1 promotes cardiomyocyte terminal differentiation in the developing heart via heightened DNA methylation.Fetal hypoxia increases vulnerability of hypoxic-ischemic brain injury in neonatal rats: role of glucocorticoid receptors.From cell phenotype to epigenetic mechanisms: new insights into regenerating myocardium.Developmental programming of cardiovascular disease by prenatal hypoxia.Oxidative stress and DNA methylation regulation in the metabolic syndrome.Dopamine induces lipid accumulation, NADPH oxidase-related oxidative stress, and a proinflammatory status of the plasma membrane in H9c2 cells.The epigenetic landscape related to reactive oxygen species formation in the cardiovascular system.Direct effect of chronic hypoxia in suppressing large conductance Ca(2+)-activated K(+) channel activity in ovine uterine arteries via increasing oxidative stress.Chronic hypoxia upregulates DNA methyltransferase and represses large conductance Ca2+-activated K+ channel function in ovine uterine arteries.Albumin overload down-regulates integrin-β1 through reactive oxygen species-endoplasmic reticulum stress pathway in podocytes.A polymorphism in the norepinephrine transporter gene is associated with affective and cardiovascular disease through a microRNA mechanism.Apocynin Prevents Abnormal Megakaryopoiesis and Platelet Activation Induced by Chronic Stress.

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Norepinephrine causes epigenetic repression of PKCε gene in rodent hearts by activating Nox1-dependent reactive oxygen species production.

http://www.wikidata.org/entity/Q36055530

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2012 nî lūn-bûn

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Norepinephrine causes epigenet ...... ive oxygen species production.

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Fuxia Xiong

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P2860

Heart Disease and Stroke Statistics--2010 Update: A Report From the American Heart Association

Functional proteomic analysis of protein kinase C epsilon signaling complexes in the normal heart and during cardioprotection

Nox1 is involved in angiotensin II-mediated hypertension: a study in Nox1-deficient mice

Sympathetic nervous system overactivity and its role in the development of cardiovascular disease

Subcellular localization of Nox4 and regulation in diabetes

Novel gp91(phox) homologues in vascular smooth muscle cells : nox1 mediates angiotensin II-induced superoxide formation and redox-sensitive signaling pathways

Effect of in utero and early-life conditions on adult health and disease

Opposing cardioprotective actions and parallel hypertrophic effects of delta PKC and epsilon PKC.

Chronic prenatal hypoxia induces epigenetic programming of PKC{epsilon} gene repression in rat hearts.

NADPH oxidase 4 (Nox4) is a major source of oxidative stress in the failing heart.

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