Neuroplasticity of sensory and sympathetic nerve fibers in a mouse model of a painful arthritic joint. - Wikidata - awgldk - TriplyDB

Neuroplasticity of sensory and sympathetic nerve fibers in a mouse model of a painful arthritic joint.

Neuroplasticity of sensory and sympathetic nerve fibers in a mouse model of a painful arthritic joint.

http://www.wikidata.org/entity/Q36067194

about

The peripheral neuronal phenotype is important in the pathogenesis of painful human tendinopathy: a systematic review Nerve growth factor-mediated regulation of pain signalling and proposed new intervention strategies in clinical pain management Bone cancer pain: from mechanism to therapy Calcitonin gene-related peptide: physiology and pathophysiology Dissociation between the relief of skeletal pain behaviors and skin hypersensitivity in a model of bone cancer pain.Chronic Monoarthritis Pain Accelerates the Processes of Cognitive Impairment and Increases the NMDAR Subunits NR2B in CA3 of Hippocampus from 5-month-old Transgenic APP/PS1 Mice.Peripheral Nerve Fibers and Their Neurotransmitters in Osteoarthritis Pathology.Chronic oral or intraarticular administration of docosahexaenoic acid reduces nociception and knee edema and improves functional outcomes in a mouse model of Complete Freund's Adjuvant-induced knee arthritis.The neurobiology of skeletal pain Role of nociceptor estrogen receptor GPR30 in a rat model of endometriosis pain.NGF blockade at early times during bone cancer development attenuates bone destruction and increases limb use A fully caninised anti-NGF monoclonal antibody for pain relief in dogs A canine-specific anti-nerve growth factor antibody alleviates pain and improves mobility and function in dogs with degenerative joint disease-associated pain Pain from intra-articular NGF or joint injury in the rat requires contributions from peptidergic joint afferents.Cartilage stem/progenitor cells are activated in osteoarthritis via interleukin-1β/nerve growth factor signaling Selective inhibition of tropomyosin-receptor-kinase A (TrkA) reduces pain and joint damage in two rat models of inflammatory arthritis.STAT3 integrates cytokine and neurotrophin signals to promote sympathetic axon regeneration Long noncoding NONRATT021972 siRNA normalized abnormal sympathetic activity mediated by the upregulation of P2X7 receptor in superior cervical ganglia after myocardial ischemia."Bedside-to-Bench" Behavioral Outcomes in Animal Models of Pain: Beyond the Evaluation of Reflexes.Control of arthritis pain with anti-nerve-growth factor: risk and benefit.Dysfunctional brain-bone marrow communication: a paradigm shift in the pathophysiology of hypertension.Repeated administration of mazindol reduces spontaneous pain-related behaviors without modifying bone density and microarchitecture in a mouse model of complete Freund's adjuvant-induced knee arthritis.Bone Pain and Muscle Weakness in Cancer Patients.Degenerative Joint Diseases and Neuroinflammation.Dynamic weight bearing as a non-reflexive method for the measurement of abdominal pain in mice.Nerve growth factor induced after temporomandibular joint inflammation decelerates chondrocyte differentiation.NGF and Its Receptors in the Regulation of Inflammatory Response.In Vitro and In Vivo Characterization of a Fully Felinized Therapeutic Anti-Nerve Growth Factor Monoclonal Antibody for the Treatment of Pain in Cats.Cancer Chemotherapy in Early Life Significantly Alters the Maturation of Pain Processing.Role of the Bone Microenvironment in the Development of Painful Complications of Skeletal Metastases.Anti-nerve growth factor therapy attenuates cutaneous hypersensitivity and musculoskeletal discomfort in mice with osteoporosis.Mechanisms of Osteoarthritis (OA) Pain

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P2860

Neuroplasticity of sensory and sympathetic nerve fibers in a mouse model of a painful arthritic joint.

http://www.wikidata.org/entity/Q36067194

description

2012 nî lūn-bûn

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Neuroplasticity of sensory and ...... of a painful arthritic joint.

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Neuroplasticity of sensory and ...... of a painful arthritic joint.

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Arthritis & Rheumatology

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Neuroplasticity of sensory and ...... of a painful arthritic joint.

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Aaron P Bloom

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Gabriela Castaneda-Corral

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Joseph R Ghilardi

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Juan M Jimenez-Andrade

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Kathleen A Coughlin

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Katie T Freeman

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Magdalena J Kaczmarska

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Michael A Kuskowski

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Patrick W Mantyh

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P2860

Does the right side know what the left is doing?

Injury-induced mechanical hypersensitivity requires C-low threshold mechanoreceptors.

A phenotypically restricted set of primary afferent nerve fibers innervate the bone versus skin: therapeutic opportunity for treating skeletal pain.

Proinflammatory mediators, stimulators of sensory neuron excitability via the expression of acid-sensing ion channels.

Blockade of nerve sprouting and neuroma formation markedly attenuates the development of late stage cancer pain.

Administration of a tropomyosin receptor kinase inhibitor attenuates sarcoma-induced nerve sprouting, neuroma formation and bone cancer pain.

The majority of myelinated and unmyelinated sensory nerve fibers that innervate bone express the tropomyosin receptor kinase A.

Topical capsaicin for pain management: therapeutic potential and mechanisms of action of the new high-concentration capsaicin 8% patch.

Burden of major musculoskeletal conditions.

New advances in musculoskeletal pain.

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2223-2232

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10.1002/ART.34385

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7

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64

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neuroplasticity

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3386465

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