Reactive oxygen species (ROS) play a critical role in the cAMP-induced activation of Ras and the phosphorylation of ERK1/2 in Leydig cells - Wikidata - awgldk - TriplyDB

Reactive oxygen species (ROS) play a critical role in the cAMP-induced activation of Ras and the phosphorylation of ERK1/2 in Leydig cells

Reactive oxygen species (ROS) play a critical role in the cAMP-induced activation of Ras and the phosphorylation of ERK1/2 in Leydig cells

http://www.wikidata.org/entity/Q36067393

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Lactate regulates rat male germ cell function through reactive oxygen species Crosstalk between Mycobacterium tuberculosis and the host cell RiboTag analysis of actively translated mRNAs in Sertoli and Leydig cells in vivo.In utero exposure to di-(2-ethylhexyl) phthalate induces testicular effects in neonatal rats that are antagonized by genistein cotreatment The Leydig cell MEK/ERK pathway is critical for maintaining a functional population of adult Leydig cells and for fertility Aging and luteinizing hormone effects on reactive oxygen species production and DNA damage in rat Leydig cells.Mitochondrial fusion and ERK activity regulate steroidogenic acute regulatory protein localization in mitochondria GnRH Regulates Gonadotropin Gene Expression Through NADPH/Dual Oxidase-Derived Reactive Oxygen Species.Ethanol stimulates epithelial sodium channels by elevating reactive oxygen species.Mono-2-ethylhexyl phthalate stimulates androgen production but suppresses mitochondrial function in mouse leydig cells with different steroidogenic potential.Human LH and hCG stimulate differently the early signalling pathways but result in equal testosterone synthesis in mouse Leydig cells in vitro.Oxidative stress and the ageing endocrine system.Ameliorative Effect of VE, IGF-I, and hCG on the Fluoride-Induced Testosterone Release Suppression in Mice Leydig Cells.Proliferative effect of histamine on MA-10 Leydig tumor cells mediated through HRH2 activation, transient elevation in cAMP production, and increased extracellular signal-regulated kinase phosphorylation levels.Peroxiredoxin II promotes hepatic tumorigenesis through cooperation with Ras/Forkhead box M1 signaling pathway.RETRACTED: Is immunosuppression, induced by neonatal thymectomy, compatible with poor reproductive performance in adult male rats?Procyanidin B2 3,3″-di-O-gallate induces oxidative stress-mediated cell death in prostate cancer cells via inhibiting MAP kinase phosphatase activity and activating ERK1/2 and AMPK.Augmented expression of gamma-glutamyl transferase 5 (GGT5) impairs testicular steroidogenesis by deregulating local oxidative stress.Steroidogenesis in Leydig cells: effects of aging and environmental factors.

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Reactive oxygen species (ROS) play a critical role in the cAMP-induced activation of Ras and the phosphorylation of ERK1/2 in Leydig cells

http://www.wikidata.org/entity/Q36067393

description

2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

Reactive oxygen species (ROS) ...... tion of ERK1/2 in Leydig cells

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Reactive oxygen species (ROS) ...... tion of ERK1/2 in Leydig cells

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Reactive oxygen species (ROS) ...... tion of ERK1/2 in Leydig cells

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Reactive oxygen species (ROS) ...... tion of ERK1/2 in Leydig cells

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Reactive oxygen species (ROS) ...... tion of ERK1/2 in Leydig cells

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Reactive oxygen species (ROS) ...... tion of ERK1/2 in Leydig cells

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Molecular Endocrinology

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Reactive oxygen species (ROS) ...... tion of ERK1/2 in Leydig cells

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Mario Ascoli

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Ping Tai

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P2860

The mRNA encoding a high-affinity cAMP phosphodiesterase is regulated by hormones and cAMP

The NOX family of ROS-generating NADPH oxidases: physiology and pathophysiology

Antioxidant protective mechanisms against reactive oxygen species (ROS) generated by mitochondrial P450 systems in steroidogenic cells

Mitochondrial respiratory chain is involved in insulin-stimulated hydrogen peroxide production and plays an integral role in insulin receptor autophosphorylation in neurons

A co-culture system reveals the involvement of intercellular pathways as mediators of the lutropin receptor (LHR)-stimulated ERK1/2 phosphorylation in Leydig cells.

Activation of the lutropin/choriogonadotropin receptor in MA-10 cells stimulates tyrosine kinase cascades that activate ras and the extracellular signal regulated kinases (ERK1/2).

Molecular mechanisms mediating the effect of mono-(2-ethylhexyl) phthalate on hormone-stimulated steroidogenesis in MA-10 mouse tumor Leydig cells

Effect of glutathione redox state on Leydig cell susceptibility to acute oxidative stress.

Epac: defining a new mechanism for cAMP action.

Activation of the lutropin/choriogonadotropin receptor in MA-10 cells leads to the tyrosine phosphorylation of the focal adhesion kinase by a pathway that involves Src family kinases

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885-893

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10.1210/ME.2010-0489

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5

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25

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2011-02-17T00:00:00Z

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21330403

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phosphorylation

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3386528

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