DNA repair is indispensable for survival after acute inflammation. - Wikidata - awgldk - TriplyDB

DNA repair is indispensable for survival after acute inflammation.

DNA repair is indispensable for survival after acute inflammation.

http://www.wikidata.org/entity/Q36068346

about

Lipidome in colorectal cancer A role for Saccharomyces cerevisiae Tpa1 protein in direct alkylation repair Base excision repair Noncanonical regulation of alkylation damage resistance by the OTUD4 deubiquitinase.A novel role for transcription-coupled nucleotide excision repair for the in vivo repair of 3,N4-ethenocytosine.Adaptive Response Enzyme AlkB Preferentially Repairs 1-Methylguanine and 3-Methylthymine Adducts in Double-Stranded DNA Oncometabolites d- and l-2-Hydroxyglutarate Inhibit the AlkB Family DNA Repair Enzymes under Physiological Conditions PRL-3 promotes telomere deprotection and chromosomal instability.O6-methylguanine-DNA methyltransferase in the defense against N-nitroso compounds and colorectal cancer.Aag-initiated base excision repair promotes ischemia reperfusion injury in liver, brain, and kidney.Base excision repair capacity in informing healthspan.Interplay between DNA repair and inflammation, and the link to cancer Kinetic mechanism for the flipping and excision of 1,N(6)-ethenoadenine by AlkA.Chemistry meets biology in colitis-associated carcinogenesis Protozoan ALKBH8 oxygenases display both DNA repair and tRNA modification activities.Differential repair of etheno-DNA adducts by bacterial and human AlkB proteins The AlkB Family of Fe(II)/α-Ketoglutarate-dependent Dioxygenases: Repairing Nucleic Acid Alkylation Damage and Beyond.Neil2-null Mice Accumulate Oxidized DNA Bases in the Transcriptionally Active Sequences of the Genome and Are Susceptible to Innate Inflammation Mechanisms of glycosylase induced genomic instability Base excision repair in physiology and pathology of the central nervous system Influenza infection induces host DNA damage and dynamic DNA damage responses during tissue regeneration.Nucleic acid oxidation in DNA damage repair and epigenetics Non-homologous functions of the AlkB homologs.Regulation of DNA Alkylation Damage Repair: Lessons and Therapeutic Opportunities.The MUTYH base excision repair gene protects against inflammation-associated colorectal carcinogenesis The Mbd4 DNA glycosylase protects mice from inflammation-driven colon cancer and tissue injury.Slow repair of lipid peroxidation-induced DNA damage at p53 mutation hotspots in human cells caused by low turnover of a DNA glycosylase Mitochondrial DNA Integrity Is Maintained by APE1 in Carcinogen-Induced Colorectal Cancer.

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P2860

DNA repair is indispensable for survival after acute inflammation.

http://www.wikidata.org/entity/Q36068346

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

DNA repair is indispensable for survival after acute inflammation.

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DNA repair is indispensable for survival after acute inflammation.

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type

label

DNA repair is indispensable for survival after acute inflammation.

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DNA repair is indispensable for survival after acute inflammation.

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prefLabel

DNA repair is indispensable for survival after acute inflammation.

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DNA repair is indispensable for survival after acute inflammation.

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Journal of Clinical Investigation

P1476

DNA repair is indispensable for survival after acute inflammation.

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P2093

Catherine A Moroski-Erkul

http://www.w3.org/2001/XMLSchema#string

Chun-Yue I Lee

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Jennifer A Calvo

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Koli Taghizadeh

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Leona D Samson

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Lindsey W Eichinger

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Line M Nordstrand

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Lisiane B Meira

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Nona Abolhassani

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Sureshkumar Muthupalani

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P2860

Hallmarks of Cancer: The Next Generation

Defective LPS Signaling in C3H/HeJ and C57BL/10ScCr Mice: Mutations in Tlr4 Gene

DNA unwinding by ASCC3 helicase is coupled to ALKBH3-dependent DNA alkylation repair and cancer cell proliferation

The inflammasomes

Repair deficient mice reveal mABH2 as the primary oxidative demethylase for repairing 1meA and 3meC lesions in DNA

Mutagenesis, genotoxicity, and repair of 1-methyladenine, 3-alkylcytosines, 1-methylguanine, and 3-methylthymine in alkB Escherichia coli

Cerulein pancreatitis: oxidative stress, inflammation, and apoptosis

Inflammation and cancer

The risk of colorectal cancer in ulcerative colitis: a meta-analysis

Structural Basis for the Inhibition of Human Alkyladenine DNA Glycosylase (AAG) by 3,N4-Ethenocytosine-containing DNA

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2680-2689

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scholarly article

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10.1172/JCI63338

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7

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122

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22684101

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3386829

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