Disseminated intravascular coagulation or acute coagulopathy of trauma shock early after trauma? An observational study. - Wikidata - awgldk - TriplyDB

Disseminated intravascular coagulation or acute coagulopathy of trauma shock early after trauma? An observational study.

Disseminated intravascular coagulation or acute coagulopathy of trauma shock early after trauma? An observational study.

http://www.wikidata.org/entity/Q36073044

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Management of bleeding and coagulopathy following major trauma: an updated European guideline.Sympathoadrenal Activation is Associated with Acute Traumatic Coagulopathy and Endotheliopathy in Isolated Brain Injury The European guideline on management of major bleeding and coagulopathy following trauma: fourth edition Coagulation complications following trauma Tranexamic acid: from trauma to routine perioperative use The current understanding of trauma-induced coagulopathy (TIC): a focused review on pathophysiology Fibrinogen degradation product levels on arrival for trauma patients requiring a transfusion even without head injury Fibrinogen depletion in trauma: early, easy to estimate and central to trauma-induced coagulopathy.Acute coagulopathy in trauma: with or without shock? That is the question.Differentiating disseminated intravascular coagulation (DIC) with the fibrinolytic phenotype from coagulopathy of trauma and acute coagulopathy of trauma-shock (COT/ACOTS).High levels of soluble VEGF receptor 1 early after trauma are associated with shock, sympathoadrenal activation, glycocalyx degradation and inflammation in severely injured patients: a prospective study Overwhelming tPA release, not PAI-1 degradation, is responsible for hyperfibrinolysis in severely injured trauma patients.Mechanistic determinates of the acute coagulopathy of trauma (ACoT) in patients requiring emergency surgery.Interventional Algorithms for the Control of Coagulopathic Bleeding in Surgical, Trauma, and Postpartum Settings: Recommendations From the Share Network Group THE GLYCOCALYX AND TRAUMA: A REVIEW.Blood levels of histone-complexed DNA fragments are associated with coagulopathy, inflammation and endothelial damage early after trauma.Activated Protein C Drives the Hyperfibrinolysis of Acute Traumatic Coagulopathy.Shock induced endotheliopathy (SHINE) in acute critical illness - a unifying pathophysiologic mechanism.Plasma transfusion for patients with severe hemorrhage: what is the evidence?Current management of massive hemorrhage in trauma.Hypothermia in trauma.New developments in the area of factor XIII.The Pathophysiology and Management of Acute Traumatic Coagulopathy.Traumatic coagulopathy--part 1: Pathophysiology and diagnosis.The pathogenesis of traumatic coagulopathy.Modern resuscitation of hemorrhagic shock: what is on the horizon?Progresses in understanding trauma-induced coagulopathy and the underlying mechanism.Coagulation and complement system in critically ill patients.Activated protein C does not increase in the early phase of trauma with disseminated intravascular coagulation: comparison with acute coagulopathy of trauma-shock.Fibrinolysis in trauma: a review.Low hemorrhage-related mortality in trauma patients in a Level I trauma center employing transfusion packages and early thromboelastography-directed hemostatic resuscitation with plasma and platelets.Lyophilized plasma attenuates vascular permeability, inflammation and lung injury in hemorrhagic shock.Prospective multicenter evaluation of coagulation abnormalities in dogs following severe acute trauma.7.5% NaCl Resuscitation Leads to Abnormal Clot Fibrinolysis after Severe Hemorrhagic Shock and its Correction with 7.5% NaCl Adenosine, Lidocaine, and Mg2.Acute Traumatic Coagulopathy: The Value of Histone in Pediatric Trauma Patients.Activated protein C plays no major roles in the inhibition of coagulation or increased fibrinolysis in acute coagulopathy of trauma-shock: a systematic review.

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P2860

Disseminated intravascular coagulation or acute coagulopathy of trauma shock early after trauma? An observational study.

http://www.wikidata.org/entity/Q36073044

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Anne Marie Sørensen

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Claus F Larsen

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Karen Lise Welling

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P2860

Epidemiology of trauma deaths: a reassessment

Extracellular RNA constitutes a natural procoagulant cofactor in blood coagulation.

Differential clot stabilising effects of rFVIIa and rFXIII-A2 in whole blood from thrombocytopenic patients and healthy volunteers.

Early coagulopathy after isolated severe traumatic brain injury: relationship with hypoperfusion challenged.

Disseminated intravascular coagulation in trauma patients.

Pathophysiology of early trauma-induced coagulopathy: emerging evidence for hemodilution and coagulation factor depletion.

Contribution of fibrin stabilization to clot strength. Supplementation of factor XIII-deficient plasma with the purified zymogen.

Acute coagulopathy of trauma: mechanism, identification and effect.

The coagulopathy of trauma: a review of mechanisms.

Early evaluation of acute traumatic coagulopathy by thrombelastography.

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