TRAIL+ monocytes and monocyte-related cells cause lung damage and thereby increase susceptibility to influenza-Streptococcus pneumoniae coinfection. - Wikidata - awgldk - TriplyDB

TRAIL+ monocytes and monocyte-related cells cause lung damage and thereby increase susceptibility to influenza-Streptococcus pneumoniae coinfection.

TRAIL+ monocytes and monocyte-related cells cause lung damage and thereby increase susceptibility to influenza-Streptococcus pneumoniae coinfection.

http://www.wikidata.org/entity/Q36078577

about

Role of Autophagy and Apoptosis in the Postinfluenza Bacterial Pneumonia Highly pathogenic avian influenza H5N1 virus delays apoptotic responses via activation of STAT3.Exogenous Activation of Invariant Natural Killer T Cells by α-Galactosylceramide Reduces Pneumococcal Outgrowth and Dissemination Postinfluenza Inflammatory Responses Regulating Alveolar Ion Transport during Pulmonary Infections.Macrophage-epithelial paracrine crosstalk inhibits lung edema clearance during influenza infection.Influenza A Virus Infection Predisposes Hosts to Secondary Infection with Different Streptococcus pneumoniae Serotypes with Similar Outcome but Serotype-Specific Manifestation.Hierarchical effects of pro-inflammatory cytokines on the post-influenza susceptibility to pneumococcal coinfection.Type I Interferons as Regulators of Lung Inflammation.The Roles of Type I Interferon in Bacterial Infection.The inflammatory response triggered by Influenza virus: a two edged sword.Protection against Streptococcus pneumoniae Invasive Pathogenesis by a Protein-Based Vaccine Is Achieved by Suppression of Nasopharyngeal Bacterial Density during Influenza A Virus Coinfection.The Impact of the Interferon/TNF-Related Apoptosis-Inducing Ligand Signaling Axis on Disease Progression in Respiratory Viral Infection and Beyond.Influenza A virus-induced release of interleukin-10 inhibits the anti-microbial activities of invariant natural killer T cells during invasive pneumococcal superinfection.Influenza A virus infection impacts systemic microbiota dynamics and causes quantitative enteric dysbiosis.Cytokine-Ion Channel Interactions in Pulmonary Inflammation.Neutrophil extracellular traps in immunity and disease.The Murine Natural Cytotoxic Receptor NKp46/NCR1 Controls TRAIL Protein Expression in NK Cells and ILC1s.Interleukin-22 immunotherapy during severe influenza enhances lung tissue integrity and reduces secondary bacterial systemic invasion.Lethal Synergism between Influenza and Treating Influenza Infection, From Now and Into the Future Back to the Future: Lessons Learned From the 1918 Influenza Pandemic Alteration of Flt3-Ligand-dependent de novo generation of conventional dendritic cells during influenza infection contributes to respiratory bacterial superinfection Role of Inflammatory Risk Factors in the Pathogenesis of Coinfection With Influenza A Virus and : An Underrecognized Impact on Host Resistance and Tolerance to Pulmonary Infections

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P2860

TRAIL+ monocytes and monocyte-related cells cause lung damage and thereby increase susceptibility to influenza-Streptococcus pneumoniae coinfection.

http://www.wikidata.org/entity/Q36078577

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

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2015年論文

@zh-hant

2015年論文

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2015年論文

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2015年論文

@zh-tw

2015年论文

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2015年论文

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2015年论文

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name

TRAIL+ monocytes and monocyte- ...... coccus pneumoniae coinfection.

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TRAIL+ monocytes and monocyte- ...... coccus pneumoniae coinfection.

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TRAIL+ monocytes and monocyte- ...... coccus pneumoniae coinfection.

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TRAIL+ monocytes and monocyte- ...... coccus pneumoniae coinfection.

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TRAIL+ monocytes and monocyte- ...... coccus pneumoniae coinfection.

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TRAIL+ monocytes and monocyte- ...... coccus pneumoniae coinfection.

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TRAIL+ monocytes and monocyte- ...... coccus pneumoniae coinfection.

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Gregory T Ellis

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Sophia Davidson

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Venizelos Papayannopoulos

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P2860

Predominant role of bacterial pneumonia as a cause of death in pandemic influenza: implications for pandemic influenza preparedness

Neutrophil-Mediated Phagocytosis of Staphylococcus aureus

Macrophage-expressed IFN-β contributes to apoptotic alveolar epithelial cell injury in severe influenza virus pneumonia

The immunology of influenza virus-associated bacterial pneumonia

Influenza: old and new threats

Flow cytometric analysis of macrophages and dendritic cell subsets in the mouse lung

Neutrophils and immunity: challenges and opportunities

Monocyte emigration from bone marrow during bacterial infection requires signals mediated by chemokine receptor CCR2

Protecting against post-influenza bacterial pneumonia by increasing phagocyte recruitment and ROS production.

The co-pathogenesis of influenza viruses with bacteria in the lung.

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1203-1218

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scholarly article

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10.15252/EMBR.201540473

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9

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16

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Stefania Crotta

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2015-08-11T00:00:00Z

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280999665

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