Nutrigenomics analysis reveals that copper deficiency and dietary sucrose up-regulate inflammation, fibrosis and lipogenic pathways in a mature rat model of nonalcoholic fatty liver disease
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Molecular Pathogenesis of NASHHost Cell Copper Transporters CTR1 and ATP7A are important for Influenza A virus replication.A Nutrigenomic Approach to Non-Alcoholic Fatty Liver Disease.Fatty Acids and Breast Cancer: Make Them on Site or Have Them Delivered.The role of insufficient copper in lipid synthesis and fatty-liver disease.Development of hepatocellular cancer induced by long term low fat-high carbohydrate diet in a NAFLD/NASH mouse model.Non-Alcoholic Fatty Liver Disease and Nutritional Implications: Special Focus on Copper.Targeted inactivation of copper transporter Atp7b in hepatocytes causes liver steatosis and obesity in mice.Copper deficiency may be a leading cause of ischaemic heart diseaseCopper-dependent amino oxidase 3 governs selection of metabolic fuels in adipocytes
P2860
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P2860
Nutrigenomics analysis reveals that copper deficiency and dietary sucrose up-regulate inflammation, fibrosis and lipogenic pathways in a mature rat model of nonalcoholic fatty liver disease
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2015 nî lūn-bûn
@nan
2015年の論文
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2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Nutrigenomics analysis reveals ...... nalcoholic fatty liver disease
@ast
Nutrigenomics analysis reveals ...... nalcoholic fatty liver disease
@en
type
label
Nutrigenomics analysis reveals ...... nalcoholic fatty liver disease
@ast
Nutrigenomics analysis reveals ...... nalcoholic fatty liver disease
@en
prefLabel
Nutrigenomics analysis reveals ...... nalcoholic fatty liver disease
@ast
Nutrigenomics analysis reveals ...... nalcoholic fatty liver disease
@en
P2093
P2860
P1476
Nutrigenomics analysis reveals ...... nalcoholic fatty liver disease
@en
P2093
Martina Ralle
María Paz Cortés
Mauricio Latorre
Megan Duffy
Savannah Tallino
P2860
P304
P356
10.1016/J.JNUTBIO.2015.04.009
P577
2015-05-15T00:00:00Z