PIP4kγ is a substrate for mTORC1 that maintains basal mTORC1 signaling during starvation
about
The Mechanistic Target of Rapamycin: The Grand ConducTOR of Metabolism and AgingDeletion of the gene Pip4k2c, a novel phosphatidylinositol kinase, results in hyperactivation of the immune systemPrevention of Proliferative Vitreoretinopathy by Suppression of Phosphatidylinositol 5-Phosphate 4-Kinases.Inhibition of PIP4Kγ ameliorates the pathological effects of mutant huntingtin protein.Dual blockade of the lipid kinase PIP4Ks and mitotic pathways leads to cancer-selective lethality.
P2860
PIP4kγ is a substrate for mTORC1 that maintains basal mTORC1 signaling during starvation
description
2014 nî lūn-bûn
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PIP4kγ is a substrate for mTORC1 that maintains basal mTORC1 signaling during starvation
@ast
PIP4kγ is a substrate for mTORC1 that maintains basal mTORC1 signaling during starvation
@en
type
label
PIP4kγ is a substrate for mTORC1 that maintains basal mTORC1 signaling during starvation
@ast
PIP4kγ is a substrate for mTORC1 that maintains basal mTORC1 signaling during starvation
@en
prefLabel
PIP4kγ is a substrate for mTORC1 that maintains basal mTORC1 signaling during starvation
@ast
PIP4kγ is a substrate for mTORC1 that maintains basal mTORC1 signaling during starvation
@en
P2093
P2860
P1433
P1476
PIP4kγ is a substrate for mTORC1 that maintains basal mTORC1 signaling during starvation
@en
P2093
Ashley M Mackey
Deborah A Sarkes
Ian Bettencourt
John M Asara
Lucia E Rameh
P2860
P356
10.1126/SCISIGNAL.2005191
P577
2014-11-04T00:00:00Z