Macrophage dectin-1 expression is controlled by leukotriene B4 via a GM-CSF/PU.1 axis. - Wikidata - awgldk - TriplyDB

Macrophage dectin-1 expression is controlled by leukotriene B4 via a GM-CSF/PU.1 axis.

Macrophage dectin-1 expression is controlled by leukotriene B4 via a GM-CSF/PU.1 axis.

http://www.wikidata.org/entity/Q36083704

about

Pivotal roles of GM-CSF in autoimmunity and inflammation The pivotal role of 5-lipoxygenase-derived LTB4 in controlling pulmonary paracoccidioidomycosis Canonical and noncanonical g-protein signaling helps coordinate actin dynamics to promote macrophage phagocytosis of zymosan PU.1 serves a critical role in the innate defense against Aspergillus fumigatus via dendritic cell-associated C-type lectin receptor-1 and toll-like receptors-2 and 4 in THP-1-derived macrophages Inflammation and immunity in the pathogenesis of pulmonary arterial hypertension Understanding the mechanisms controlling Leishmania amazonensis infection in vitro: the role of LTB4 derived from human neutrophils Prostaglandin E2 reduces Toll-like receptor 4 expression in alveolar macrophages by inhibition of translation.Myeloid-derived suppressor cells impair alveolar macrophages through PD-1 receptor ligation during Pneumocystis pneumonia.Leukotriene B4-mediated sterile inflammation promotes susceptibility to sepsis in a mouse model of type 1 diabetes PU.1 is involved in the immune response to Aspergillus fumigatus through upregulating Dectin-1 expression.Opposing roles of LTB4 and PGE2 in regulating the inflammasome-dependent scorpion venom-induced mortality.Reduced BMPR2 expression induces GM-CSF translation and macrophage recruitment in humans and mice to exacerbate pulmonary hypertension.Leukotriene B4 enhances the generation of proinflammatory microRNAs to promote MyD88-dependent macrophage activation Immunomodulation of Fungal β-Glucan in Host Defense Signaling by Dectin-1.Low-Dose Oxygen Enhances Macrophage-Derived Bacterial Clearance following Cigarette Smoke Exposure.Biochemical and immunological characterization of a novel monoclonal antibody against mouse leukotriene B4 receptor 1.Targeting leukotriene B4 in inflammation.Too much of a good thing: How modulating LTB4 actions restore host defense in homeostasis or disease.The Interaction of Human Pathogenic Fungi With C-Type Lectin Receptors.Macrophage-derived LTB4 promotes abscess formation and clearance of Staphylococcus aureus skin infection in mice Excessive localized leukotriene B4 levels dictate poor skin host defense in diabetic mice

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Macrophage dectin-1 expression is controlled by leukotriene B4 via a GM-CSF/PU.1 axis.

http://www.wikidata.org/entity/Q36083704

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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name

Macrophage dectin-1 expression is controlled by leukotriene B4 via a GM-CSF/PU.1 axis.

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Macrophage dectin-1 expression is controlled by leukotriene B4 via a GM-CSF/PU.1 axis.

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Macrophage dectin-1 expression is controlled by leukotriene B4 via a GM-CSF/PU.1 axis.

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Macrophage dectin-1 expression is controlled by leukotriene B4 via a GM-CSF/PU.1 axis.

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prefLabel

Macrophage dectin-1 expression is controlled by leukotriene B4 via a GM-CSF/PU.1 axis.

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Macrophage dectin-1 expression is controlled by leukotriene B4 via a GM-CSF/PU.1 axis.

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JIMMUNOL.1200257

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Journal of Immunology

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Macrophage dectin-1 expression is controlled by leukotriene B4 via a GM-CSF/PU.1 axis.

@en

P2093

C Henrique Serezani

http://www.w3.org/2001/XMLSchema#string

John J Osterholzer

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Latima Collins

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Mariana Morato-Marques

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Steve Kane

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P2860

PU.1 and interferon consensus sequence-binding protein regulate the myeloid expression of the human Toll-like receptor 4 gene

Immune recognition. A new receptor for beta-glucans

Dectin-1: a signalling non-TLR pattern-recognition receptor

Dectin-1 mediates the biological effects of beta-glucans

Syk-dependent cytokine induction by Dectin-1 reveals a novel pattern recognition pathway for C type lectins

Dectin-1 is a major beta-glucan receptor on macrophages

Pathogen recognition by the innate immune system

PPARγ controls Dectin-1 expression required for host antifungal defense against Candida albicans.

Downregulation of PU.1 leads to decreased expression of Dectin-1 in alveolar macrophages during Pneumocystis pneumonia.

The role of Ets family transcription factor PU.1 in hematopoietic cell differentiation, proliferation and apoptosis.

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906-915

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10.4049/JIMMUNOL.1200257

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2

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189

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Marc Peters-Golden

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2012-06-13T00:00:00Z

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225305321

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3392366

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