Bortezomib attenuates HIF-1- but not HIF-2-mediated transcriptional activation

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Hypoxia inducible factor-1 alpha and multiple myeloma.Newcastle disease virus degrades HIF-1α through proteasomal pathways independent of VHL and p53.Expression of hypoxia inducible factor 1α and 2α and its association with vitamin C level in thyroid lesions.

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Bortezomib attenuates HIF-1- but not HIF-2-mediated transcriptional activation

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http://www.wikidata.org/entity/Q36085646

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

@zh-hk

2015年論文

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2015年論文

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2015年论文

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2015年论文

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name

Bortezomib attenuates HIF-1- but not HIF-2-mediated transcriptional activation

@ast

Bortezomib attenuates HIF-1- but not HIF-2-mediated transcriptional activation

@en

type

Item

label

Bortezomib attenuates HIF-1- but not HIF-2-mediated transcriptional activation

@ast

Bortezomib attenuates HIF-1- but not HIF-2-mediated transcriptional activation

@en

prefLabel

Bortezomib attenuates HIF-1- but not HIF-2-mediated transcriptional activation

@ast

Bortezomib attenuates HIF-1- but not HIF-2-mediated transcriptional activation

@en

P1476

Bortezomib attenuates HIF-1- but not HIF-2-mediated transcriptional activation

@en

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Eric J Stanbridge

http://www.w3.org/2001/XMLSchema#string

Noraini Abd-Aziz

http://www.w3.org/2001/XMLSchema#string

Norazizah Shafee

http://www.w3.org/2001/XMLSchema#string

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Endothelial PAS domain protein 1 (EPAS1), a transcription factor selectively expressed in endothelial cells

A novel bHLH-PAS factor with close sequence similarity to hypoxia-inducible factor 1alpha regulates the VEGF expression and is potentially involved in lung and vascular development

HIF1α and HIF2α: sibling rivalry in hypoxic tumour growth and progression

The Expression and Distribution of the Hypoxia-Inducible Factors HIF-1α and HIF-2α in Normal Human Tissues, Cancers, and Tumor-Associated Macrophages

Hypoxia-inducible factor-1 deficiency results in dysregulated erythropoiesis signaling and iron homeostasis in mouse development

Differential roles of hypoxia-inducible factor 1alpha (HIF-1alpha) and HIF-2alpha in hypoxic gene regulation

Bortezomib as the first proteasome inhibitor anticancer drug: current status and future perspectives.

The N-terminal transactivation domain confers target gene specificity of hypoxia-inducible factors HIF-1alpha and HIF-2alpha

Bortezomib: proteasome inhibition as an effective anticancer therapy.

Transcriptional control of the tumor- and hypoxia-marker carbonic anhydrase 9: A one transcription factor (HIF-1) show?

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2192-2196

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scholarly article

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10.3892/OL.2015.3545

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2015-07-29T00:00:00Z

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282458215

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26622817

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4579903

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Bortezomib attenuates HIF-1- but not HIF-2-mediated transcriptional activation

about

P2860

P2860

Bortezomib attenuates HIF-1- but not HIF-2-mediated transcriptional activation

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P932