Overexpression of FGFR2 contributes to inherent resistance to MET inhibitors in MET-amplified patient-derived gastric cancer xenografts.

about

Cell-Free DNA Provides a Good Representation of the Tumor Genome Despite Its Biased Fragmentation Patterns.

P2860

Q36238197-99115178-678A-47F6-AEE3-8BA241E7F51E

P2860

Overexpression of FGFR2 contributes to inherent resistance to MET inhibitors in MET-amplified patient-derived gastric cancer xenografts.

Item

http://www.wikidata.org/entity/Q36085827

description

2015 nî lūn-bûn

@nan

2015年の論文

@ja

2015年論文

@yue

2015年論文

@zh-hant

2015年論文

@zh-hk

2015年論文

@zh-mo

2015年論文

@zh-tw

2015年论文

@wuu

2015年论文

@zh

2015年论文

@zh-cn

name

Overexpression of FGFR2 contri ...... ved gastric cancer xenografts.

@ast

Overexpression of FGFR2 contri ...... ved gastric cancer xenografts.

@en

type

Item

label

Overexpression of FGFR2 contri ...... ved gastric cancer xenografts.

@ast

Overexpression of FGFR2 contri ...... ved gastric cancer xenografts.

@en

prefLabel

Overexpression of FGFR2 contri ...... ved gastric cancer xenografts.

@ast

Overexpression of FGFR2 contri ...... ved gastric cancer xenografts.

@en

P1476

Overexpression of FGFR2 contri ...... ved gastric cancer xenografts.

@en

P2093

Heng Ma

http://www.w3.org/2001/XMLSchema#string

Kai Liu

http://www.w3.org/2001/XMLSchema#string

Meirong Zhu

http://www.w3.org/2001/XMLSchema#string

Xilin Song

http://www.w3.org/2001/XMLSchema#string

P2860

A family of transmembrane proteins with homology to the MET-hepatocyte growth factor receptor

Targeting receptor tyrosine kinases in gastric cancer

Cytoreductive antitumor activity of PF-2341066, a novel inhibitor of anaplastic lymphoma kinase and c-Met, in experimental models of anaplastic large-cell lymphoma.

Factors underlying sensitivity of cancers to small-molecule kinase inhibitors

A novel SND1-BRAF fusion confers resistance to c-Met inhibitor PF-04217903 in GTL16 cells through [corrected] MAPK activation

Patterns of cancer incidence, mortality, and prevalence across five continents: defining priorities to reduce cancer disparities in different geographic regions of the world

Activation of HER family members in gastric carcinoma cells mediates resistance to MET inhibition.

Treatment strategies in gastric cancer.

Gain of ALK gene copy number may predict lack of benefit from anti-EGFR treatment in patients with advanced colorectal cancer and RAS-RAF-PI3KCA wild-type status

Trend in gastric cancer: 35 years of surgical experience in Japan.

P304

2003-2008

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.3892/OL.2015.3601

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2015-08-12T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P5875

282519640

http://www.w3.org/2001/XMLSchema#string

P698

26622787

http://www.w3.org/2001/XMLSchema#string

P921

overexpression

xenograft

P932

4579967

http://www.w3.org/2001/XMLSchema#string

Overexpression of FGFR2 contributes to inherent resistance to MET inhibitors in MET-amplified patient-derived gastric cancer xenografts.

about

P2860

P2860

Overexpression of FGFR2 contributes to inherent resistance to MET inhibitors in MET-amplified patient-derived gastric cancer xenografts.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P921

P932

P356

P1433

P1476

P2093

P2860

P304

P31

P356

P433

P478

P577

P5875

P698

P921

P932