about
Overview of Alzheimer's Disease and Some Therapeutic Approaches Targeting Aβ by Using Several Synthetic and Herbal CompoundsCaenorhabditis elegans as a model organism to study APP functionDestabilizing protein polymorphisms in the genetic background direct phenotypic expression of mutant SOD1 toxicityWidespread protein aggregation as an inherent part of aging in C. elegansC. elegans expressing human β2-microglobulin: a novel model for studying the relationship between the molecular assembly and the toxic phenotypeTILLING is an effective reverse genetics technique for Caenorhabditis elegans.The Caenorhabditis elegans A beta 1-42 model of Alzheimer disease predominantly expresses A beta 3-42.Chronic activation of the epithelial immune system of the fruit fly's salivary glands has a negative effect on organismal growth and induces a peculiar set of target genesAscidians: an invertebrate chordate model to study Alzheimer's disease pathogenesis.Towards a membrane proteome in Drosophila: a method for the isolation of plasma membrane.Oleuropein aglycone protects transgenic C. elegans strains expressing Aβ42 by reducing plaque load and motor deficitNeurotoxic effects induced by the Drosophila amyloid-beta peptide suggest a conserved toxic function.Multigram synthesis and in vivo efficacy studies of a novel multitarget anti-Alzheimer's compound.Roles for ROS and hydrogen sulfide in the longevity response to germline loss in Caenorhabditis elegans.A Caenorhabditis elegans genetic-interaction map wiggles into view.Scorpion Venom Heat-Resistant Peptide Protects Transgenic Caenorhabditis elegans from β-Amyloid Toxicity.Analysis of amyloid precursor protein function in Drosophila melanogaster.Overcoming translational barriers impeding development of Alzheimer's disease modifying therapies.Investigating heart-specific toxicity of amyloidogenic immunoglobulin light chains: A lesson from C. elegansSpecific recognition of biologically active amyloid-β oligomers by a new surface plasmon resonance-based immunoassay and an in vivo assay in Caenorhabditis elegans.Less is More: Reducing Tau Ameliorates Seizures in Epilepsy ModelsHeat shock treatment reduces beta amyloid toxicity in vivo by diminishing oligomers.Copper ions influence the toxicity of β-amyloid(1-42) in a concentration-dependent manner in a Caenorhabditis elegans model of Alzheimer's disease.Phospholipase D functional ablation has a protective effect in an Alzheimer's disease Caenorhabditis elegans model.Synaptic defects associated with s-inclusion body myositis are prevented by copper.Phenolic Compounds Characteristic of the Mediterranean Diet in Mitigating Microglia-Mediated Neuroinflammation
P2860
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P2860
description
2005 nî lūn-bûn
@nan
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
2005年论文
@zh
2005年论文
@zh-cn
name
Invertebrate models of Alzheimer's disease.
@ast
Invertebrate models of Alzheimer's disease.
@en
type
label
Invertebrate models of Alzheimer's disease.
@ast
Invertebrate models of Alzheimer's disease.
@en
prefLabel
Invertebrate models of Alzheimer's disease.
@ast
Invertebrate models of Alzheimer's disease.
@en
P2860
P1476
Invertebrate models of Alzheimer's disease.
@en
P2093
P2860
P304
P356
10.1111/J.1601-183X.2004.00105.X
P407
P577
2005-04-01T00:00:00Z