about
Brain injury following cardiac arrest: pathophysiology for neurocritical careDeletion in the N-terminal half of olfactomedin 1 modifies its interaction with synaptic proteins and causes brain dystrophy and abnormal behavior in mice.Mitochondrial dysfunction induced by frataxin deficiency is associated with cellular senescence and abnormal calcium metabolismPotential targets for protecting against hippocampal cell apoptosis after transient cerebral ischemia-reperfusion injury in aged ratsChronic enrichment of hepatic endoplasmic reticulum-mitochondria contact leads to mitochondrial dysfunction in obesity.Acute focal brain damage alters mitochondrial dynamics and autophagy in axotomized neurons.Analyses of the mitochondrial mutations in the Chinese patients with sporadic Creutzfeldt-Jakob diseaseThe role of glycogen synthase kinase-3β in glioma cell apoptosis induced by remifentanil.Pharmacological stimulation of sigma-1 receptors has neurorestorative effects in experimental parkinsonism.CR6-interacting factor 1 is a key regulator in Aβ-induced mitochondrial disruption and pathogenesis of Alzheimer's disease.Molecular Targets of Cannabidiol in Neurological DisordersKetogenic diets, mitochondria, and neurological diseases.A γ-Secretase Independent Role for Presenilin in Calcium Homeostasis Impacts Mitochondrial Function and Morphology in Caenorhabditis elegans.Novel targets for mitochondrial medicine.PINK1 and Parkin cooperatively protect neurons against constitutively active TRP channel-induced retinal degeneration in Drosophila.Nutraceuticals against Neurodegeneration: A Mechanistic Insight.cAMP and mitochondria.Changes in mitochondrial function are pivotal in neurodegenerative and psychiatric disorders: how important is BDNF?Function and characteristics of PINK1 in mitochondria.Unravelling mitochondrial pathways to Parkinson's disease.Perturbed mitochondrial Ca2+ signals as causes or consequences of mitophagy induction.Fission and fusion of the neuronal endoplasmic reticulum.Neuronal calcium signaling: function and dysfunction.Calcium signaling in Parkinson's disease.Cockayne syndrome: Clinical features, model systems and pathways.Mitochondrial fission and fusion in secondary brain damage after CNS insults.PINK1 in the limelight: multiple functions of an eclectic protein in human health and disease.A possible role of transglutaminase 2 in the nucleus of INS-1E and of cells of human pancreatic isletsNCLs and ER: A stressful relationship.The role of Ca2+ signaling in Parkinson's disease.Olesoxime suppresses calpain activation and mutant huntingtin fragmentation in the BACHD rat.Calcium uptake and cytochrome c release from normal and ischemic brain mitochondria.Antioxidant-mediated reversal of oxidative damage in mouse modeling of complex I inhibition.Primary cerebral and cerebellar astrocytes display differential sensitivity to extracellular sodium with significant effects on apoptosis.SK channel activation is neuroprotective in conditions of enhanced ER-mitochondrial coupling.Mitochondrial Targeting in Neurodegeneration: A Heme PerspectiveT-type Calcium Channels Determine the Vulnerability of Dopaminergic Neurons to Mitochondrial Stress in Familial Parkinson Disease
P2860
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P2860
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
Mitochondrial Ca(2+) and neurodegeneration
@ast
Mitochondrial Ca(2+) and neurodegeneration
@en
type
label
Mitochondrial Ca(2+) and neurodegeneration
@ast
Mitochondrial Ca(2+) and neurodegeneration
@en
prefLabel
Mitochondrial Ca(2+) and neurodegeneration
@ast
Mitochondrial Ca(2+) and neurodegeneration
@en
P2860
P921
P1433
P1476
Mitochondrial Ca(2+) and neurodegeneration
@en
P2093
Denis Ottolini
P2860
P356
10.1016/J.CECA.2012.04.015
P5008
P577
2012-05-18T00:00:00Z