Mechanism for p38α-mediated experimental autoimmune encephalomyelitis. - Wikidata - awgldk - TriplyDB

Mechanism for p38α-mediated experimental autoimmune encephalomyelitis.

Mechanism for p38α-mediated experimental autoimmune encephalomyelitis.

http://www.wikidata.org/entity/Q36097933

about

MK2 and Fas receptor contribute to the severity of CNS demyelination.p38α controls self-renewal and fate decision of neurosphere-forming cells in adult hippocampus A Natural Variant of the T Cell Receptor-Signaling Molecule Vav1 Reduces Both Effector T Cell Functions and Susceptibility to Neuroinflammation.The p38α mitogen-activated protein kinase is a key regulator of myelination and remyelination in the CNS.Sex-specific control of central nervous system autoimmunity by p38 mitogen-activated protein kinase signaling in myeloid cells.Genetic and Pharmacological Inhibition of p38α Improves Locomotor Recovery after Spinal Cord Injury.The emerging role of p38 mitogen-activated protein kinase in multiple sclerosis and its models.Roles of p38α mitogen-activated protein kinase in mouse models of inflammatory diseases and cancer.Bidirectional role of IL-6 signal in pathogenesis of lung fibrosis.The Pyrazolobenzothiazine Core as a New Chemotype of p38 Alpha Mitogen-Activated Protein Kinase Inhibitors.An optimized and versatile synthesis to pyridinylimidazole-type p38α mitogen activated protein kinase inhibitors.The p38 mitogen-activated protein kinase cascade modulates T helper type 17 differentiation and functionality in multiple sclerosis.Alleviation of Microglial Activation Induced by p38 MAPK/MK2/PGE2 Axis by Capsaicin: Potential Involvement of other than TRPV1 Mechanism/s.Central Administration of 5Z-7-Oxozeaenol Protects Experimental Autoimmune Encephalomyelitis Mice by Inhibiting Microglia Activation.Identifying characteristic miRNAs-genes and risk pathways of multiple sclerosis based on bioinformatics analysis.Administration of SB203580, a p38 MAPK Inhibitor, Reduced the Expression of MMP9, and Relieved Neurologic Severity in the Experimental Autoimmune Neuritis (EAN) in Rats.Stress-Activated Protein Kinases in Spinal Cord Injury: Focus on Roles of p38.p38 Mitogen-activated protein kinase accelerates emphysema in mouse model of chronic obstructive pulmonary disease.

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Mechanism for p38α-mediated experimental autoimmune encephalomyelitis.

http://www.wikidata.org/entity/Q36097933

description

2012 nî lūn-bûn

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2012年の論文

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2012年論文

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2012年論文

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2012年論文

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2012年論文

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2012年论文

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2012年论文

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2012年论文

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name

Mechanism for p38α-mediated experimental autoimmune encephalomyelitis.

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Mechanism for p38α-mediated experimental autoimmune encephalomyelitis.

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type

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Mechanism for p38α-mediated experimental autoimmune encephalomyelitis.

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Mechanism for p38α-mediated experimental autoimmune encephalomyelitis.

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prefLabel

Mechanism for p38α-mediated experimental autoimmune encephalomyelitis.

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Mechanism for p38α-mediated experimental autoimmune encephalomyelitis.

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JBC.M111.338541

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Journal of Biological Chemistry

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Mechanism for p38α-mediated experimental autoimmune encephalomyelitis.

@en

P2093

Akira Sakairi

http://www.w3.org/2001/XMLSchema#string

Akiyoshi Fukamizu

http://www.w3.org/2001/XMLSchema#string

Hirofumi Matsunaga

http://www.w3.org/2001/XMLSchema#string

Jundal Kim

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Junji Ishida

http://www.w3.org/2001/XMLSchema#string

Kana Namiki

http://www.w3.org/2001/XMLSchema#string

Kazuya Murata

http://www.w3.org/2001/XMLSchema#string

Kensuke Tanaka

http://www.w3.org/2001/XMLSchema#string

Kento Yoshioka

http://www.w3.org/2001/XMLSchema#string

Manabu Shigetomi

http://www.w3.org/2001/XMLSchema#string

P2860

Novel p19 protein engages IL-12p40 to form a cytokine, IL-23, with biological activities similar as well as distinct from IL-12

Mutual regulation of c-Jun and ATF2 by transcriptional activation and subcellular localization

Characterization of the structure and function of a new mitogen-activated protein kinase (p38beta)

Cross-family dimerization of transcription factors Fos/Jun and ATF/CREB alters DNA binding specificity

Loss of a gp130 cardiac muscle cell survival pathway is a critical event in the onset of heart failure during biomechanical stress

Mammalian MAP kinase signalling cascades

Requirement for p38alpha in erythropoietin expression: a role for stress kinases in erythropoiesis

Characterization of the structure and function of the fourth member of p38 group mitogen-activated protein kinases, p38delta

IL-23 drives a pathogenic T cell population that induces autoimmune inflammation

Interleukin-23 rather than interleukin-12 is the critical cytokine for autoimmune inflammation of the brain

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24228-24238

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10.1074/JBC.M111.338541

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22637476

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