Productive human immunodeficiency virus type 1 infection in peripheral blood predominantly takes place in CD4/CD8 double-negative T lymphocytes.
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Double-negative T cells during HIV/SIV infections: potential pinch hitters in the T-cell lineupHumanized mice recapitulate key features of HIV-1 infection: a novel concept using long-acting anti-retroviral drugs for treating HIV-1Profound depletion of HIV-1 transcription in patients initiating antiretroviral therapy during acute infectionIn vivo CD8+ T-cell suppression of siv viremia is not mediated by CTL clearance of productively infected cells.Cytomegalovirus replication in semen is associated with higher levels of proviral HIV DNA and CD4+ T cell activation during antiretroviral treatment.A Novel Single-Cell FISH-Flow Assay Identifies Effector Memory CD4+ T cells as a Major Niche for HIV-1 Transcription in HIV-Infected Patients.Differences in HIV burden and immune activation within the gut of HIV-positive patients receiving suppressive antiretroviral therapyEffect of raltegravir-containing intensification on HIV burden and T-cell activation in multiple gut sites of HIV-positive adults on suppressive antiretroviral therapyQuantifying the turnover of transcriptional subclasses of HIV-1-infected cellsDistribution of HIV-1 infection in different T lymphocyte subsets: antiretroviral therapy-naïve vs. experienced patients.Gag-positive reservoir cells are susceptible to HIV-specific cytotoxic T lymphocyte mediated clearance in vitro and can be detected in vivo [corrected]Structured observations reveal slow HIV-1 CTL escape.Antigen-dependent and -independent mechanisms of T and B cell hyperactivation during chronic HIV-1 infectionMedroxyprogesterone acetate increases HIV-1 infection of unstimulated peripheral blood mononuclear cells in vitro.Gut commensal microbes do not represent a dominant antigenic source for continuous CD4+ T-cell activation during HIV-1 infection.HIV-1 replication activates CD4+ T cells with specificities for persistent herpes virusesHIV persistence in the setting of antiretroviral therapy: when, where and how does HIV hide?Immune Activation at Sites of HIV/TB Co-Infection Contributes to the Pathogenesis of HIV-1 Disease.Nef promotes evasion of human immunodeficiency virus type 1-infected cells from the CTLA-4-mediated inhibition of T-cell activation.HIV-1 pathogenesis: the virus.A Subset of CD4/CD8 Double-Negative T Cells Expresses HIV Proteins in Patients on Antiretroviral Therapy.Cell-associated HIV RNA: a dynamic biomarker of viral persistence.HIV rebounds from latently infected cells, rather than from continuing low-level replicationBiphasic decay kinetics suggest progressive slowing in turnover of latently HIV-1 infected cells during antiretroviral therapy.Modification of the Abbott RealTime assay for detection of HIV-1 plasma RNA viral loads less than one copy per milliliter.Dynamics of viral replication in blood and lymphoid tissues during SIVmac251 infection of macaques.Where the wild things are: pathogenesis of SIV infection in African nonhuman primate hosts.HIV load testing with small samples of whole blood.Productive HIV-1 infection is enriched in CD4(-)CD8(-) double negative (DN) T cells at pleural sites of dual infection with HIV and Mycobacterium tuberculosis.The Alphabet Soup of HIV Reservoir Markers.Analysis of multiple cell reservoirs expressing unspliced HIV-1 gag-pol mRNA in patients on antiretroviral therapyCombining Cell and Gene Therapy in an Effort to Eradicate HIV.The well-tempered SIV infection: Pathogenesis of SIV infection in natural hosts in the wild, with emphasis on virus transmission and early events post-infection that may contribute to protection from disease progression.Minor Contribution of Chimeric Host-HIV Readthrough Transcripts to the Level of HIV Cell-Associated gag RNA.Identification and characterization of HIV-1 latent viral reservoirs in peripheral blood.Double Negative (DN) [CD3⁺CD4⁻CD8⁻] T cells correlate with disease progression during HIV infection.Cohort profile: the Swiss HIV Cohort study.What do we measure when we measure cell-associated HIV RNA.Beyond the replication-competent HIV reservoir: transcription and translation-competent reservoirs.
P2860
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P2860
Productive human immunodeficiency virus type 1 infection in peripheral blood predominantly takes place in CD4/CD8 double-negative T lymphocytes.
description
2007 nî lūn-bûn
@nan
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
2007年论文
@zh
2007年论文
@zh-cn
name
Productive human immunodeficie ...... double-negative T lymphocytes.
@ast
Productive human immunodeficie ...... double-negative T lymphocytes.
@en
type
label
Productive human immunodeficie ...... double-negative T lymphocytes.
@ast
Productive human immunodeficie ...... double-negative T lymphocytes.
@en
prefLabel
Productive human immunodeficie ...... double-negative T lymphocytes.
@ast
Productive human immunodeficie ...... double-negative T lymphocytes.
@en
P2093
P2860
P356
P1433
P1476
Productive human immunodeficie ...... double-negative T lymphocytes.
@en
P2093
Barbara Niederöst
Marek Fischer
Philipp Kaiser
Rainer Weber
P2860
P304
P356
10.1128/JVI.00492-07
P407
P577
2007-07-03T00:00:00Z