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Gene therapy for chronic obstructive pulmonary disease: twilight or triumph?

Gene therapy for chronic obstructive pulmonary disease: twilight or triumph?

http://www.wikidata.org/entity/Q36099456

about

Barriers to inhaled gene therapy of obstructive lung diseases: A review Expert opinion in biological therapy: update on developments in lung gene transfer.Formulation of pH responsive peptides as inhalable dry powders for pulmonary delivery of nucleic acids.

P2860

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P2860

Gene therapy for chronic obstructive pulmonary disease: twilight or triumph?

http://www.wikidata.org/entity/Q36099456

description

2005 nî lūn-bûn

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

@zh-hk

2005年論文

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2005年論文

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2005年论文

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2005年论文

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2005年论文

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name

Gene therapy for chronic obstructive pulmonary disease: twilight or triumph?

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Gene therapy for chronic obstructive pulmonary disease: twilight or triumph?

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type

label

Gene therapy for chronic obstructive pulmonary disease: twilight or triumph?

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Gene therapy for chronic obstructive pulmonary disease: twilight or triumph?

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prefLabel

Gene therapy for chronic obstructive pulmonary disease: twilight or triumph?

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Gene therapy for chronic obstructive pulmonary disease: twilight or triumph?

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14712598.5.3.333

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Expert Opinion on Biological Therapy

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Gene therapy for chronic obstructive pulmonary disease: twilight or triumph?

@en

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R Al-Jamal

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W A H Wallace

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P2860

Association of IL-1beta and IL-1 receptor antagonist haplotypes with rate of decline in lung function in smokers

Increased metalloproteinase activity, oxidant production, and emphysema in surfactant protein D gene-inactivated mice

The role of matrix metalloproteinase polymorphisms in the rate of decline in lung function

The impact of smoking cessation on respiratory symptoms, lung function, airway hyperresponsiveness and inflammation

The degradation of human endothelial cell-derived perlecan and release of bound basic fibroblast growth factor by stromelysin, collagenase, plasmin, and heparanases

Matrix metalloproteinase degradation of extracellular matrix: biological consequences

PPAR-gamma agonists inhibit production of monocyte inflammatory cytokines

Cloning of the human alpha 2-macroglobulin gene and detection of mutations in two functional domains: the bait region and the thiolester site.

Bone marrow-derived cells contribute to lung regeneration after elastase-induced pulmonary emphysema.

The centrilobular form of hypertrophic emphysema and its relation to chronic bronchitis.

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333-346

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scholarly article

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10.1517/14712598.5.3.333

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3

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David J Harrison

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2005-03-01T00:00:00Z

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15833071

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P921

chronic obstructive pulmonary disease