A PhoP-repressed gene promotes Salmonella typhimurium invasion of epithelial cells.
about
Crossing the line: selection and evolution of virulence traitsIdentification of CsrC and characterization of its role in epithelial cell invasion in Salmonella enterica serovar TyphimuriumType III protein secretion systems in bacterial pathogens of animals and plantsThe pleiotropic two-component regulatory system PhoP-PhoQRole of SPI-1 secreted effectors in acute bovine response to Salmonella enterica Serovar Typhimurium: a systems biology analysis approachAn image classification approach to analyze the suppression of plant immunity by the human pathogen Salmonella Typhimurium.Genome-wide screen of Salmonella genes expressed during infection in pigs, using in vivo expression technologyA model of Salmonella colitis with features of diarrhea in SLC11A1 wild-type mice.Genome-wide analysis of the PreA/PreB (QseB/QseC) regulon of Salmonella enterica serovar Typhimurium.Comparative proteomic analysis of the PhoP regulon in Salmonella enterica serovar Typhi versus TyphimuriumMolecular basis of the interaction of Salmonella with the intestinal mucosa.Salmonella pathogenicity island 1-independent induction of apoptosis in infected macrophages by Salmonella enterica serotype typhimurium.Curcumin increases the pathogenicity of Salmonella enterica serovar Typhimurium in murine model.CD4+ T cells and toll-like receptors recognize Salmonella antigens expressed in bacterial surface organelles.Environmental regulation of Salmonella typhi invasion-defective mutantsCell-contact-stimulated formation of filamentous appendages by Salmonella typhimurium does not depend on the type III secretion system encoded by Salmonella pathogenicity island 1.Physical limitations on Salmonella typhi entry into cultured human intestinal epithelial cells.Induction of Salmonella pathogenicity island 1 under different growth conditions can affect Salmonella-host cell interactions in vitroInhibition of Salmonella typhimurium invasion by host cell expression of secreted bacterial invasion proteinsElimination of persistent vaccine bacteria of Salmonella enterica serovar Typhimurium in the guts of immunized mice by inducible expression of truncated YncESignal transduction pathway mediated by the novel regulator LoiA for low oxygen tension induced Salmonella Typhimurium invasion.Microarray analysis and motif detection reveal new targets of the Salmonella enterica serovar Typhimurium HilA regulatory protein, including hilA itself.A HilA-independent pathway to Salmonella typhimurium invasion gene transcription.InvF is required for expression of genes encoding proteins secreted by the SPI1 type III secretion apparatus in Salmonella typhimurium.Genetic analysis of assembly of the Salmonella enterica serovar Typhimurium type III secretion-associated needle complex.Hha is a negative modulator of transcription of hilA, the Salmonella enterica serovar Typhimurium invasion gene transcriptional activatorDifferential regulation of Salmonella typhimurium type III secreted proteins by pathogenicity island 1 (SPI-1)-encoded transcriptional activators InvF and hilA.Salmonella typhimurium virulence genes are induced upon bacterial invasion into phagocytic and nonphagocytic cells.Identification of a putative Salmonella enterica serotype typhimurium host range factor with homology to IpaH and YopM by signature-tagged mutagenesisTranscriptional organization and function of invasion genes within Salmonella enterica serovar Typhimurium pathogenicity island 1, including the prgH, prgI, prgJ, prgK, orgA, orgB, and orgC genes.The response regulator PhoP is important for survival under conditions of macrophage-induced stress and virulence in Yersinia pestis.Salmonella enterica serovar typhimurium invasion is repressed in the presence of bile.Salmonella enterica serovar typhimurium waaP mutants show increased susceptibility to polymyxin and loss of virulence In vivoIdentification of Legionella pneumophila rcp, a pagP-like gene that confers resistance to cationic antimicrobial peptides and promotes intracellular infection.In vivo genetic analysis indicates that PhoP-PhoQ and the Salmonella pathogenicity island 2 type III secretion system contribute independently to Salmonella enterica serovar Typhimurium virulence.Conservation of Salmonella infection mechanisms in plants and animalsThe flagellar regulator fliT represses Salmonella pathogenicity island 1 through flhDC and fliZ.Pathways leading from BarA/SirA to motility and virulence gene expression in Salmonella.Salmonella enterica serovar Typhimurium skills to succeed in the host: virulence and regulation.Macrophage-inducible expression of a model antigen in Salmonella typhimurium enhances immunogenicity.
P2860
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P2860
A PhoP-repressed gene promotes Salmonella typhimurium invasion of epithelial cells.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年論文
@yue
1993年論文
@zh-hant
1993年論文
@zh-hk
1993年論文
@zh-mo
1993年論文
@zh-tw
1993年论文
@wuu
1993年论文
@zh
1993年论文
@zh-cn
name
A PhoP-repressed gene promotes Salmonella typhimurium invasion of epithelial cells.
@ast
A PhoP-repressed gene promotes Salmonella typhimurium invasion of epithelial cells.
@en
type
label
A PhoP-repressed gene promotes Salmonella typhimurium invasion of epithelial cells.
@ast
A PhoP-repressed gene promotes Salmonella typhimurium invasion of epithelial cells.
@en
prefLabel
A PhoP-repressed gene promotes Salmonella typhimurium invasion of epithelial cells.
@ast
A PhoP-repressed gene promotes Salmonella typhimurium invasion of epithelial cells.
@en
P2860
P1476
A PhoP-repressed gene promotes Salmonella typhimurium invasion of epithelial cells.
@en
P2093
P2860
P304
P356
10.1128/JB.175.14.4475-4484.1993
P407
P577
1993-07-01T00:00:00Z