Genetic inactivation of mitochondria-targeted redox enzyme p66ShcA preserves neuronal viability and mitochondrial integrity in response to oxidative challenges.
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Inflammation, Iron, Energy Failure, and Oxidative Stress in the Pathogenesis of Multiple Sclerosisp53 and mitochondrial function in neurons.Mitochondrial dysfunction and neurodegeneration in multiple sclerosisDL0410, a novel dual cholinesterase inhibitor, protects mouse brains against Aβ-induced neuronal damage via the Akt/JNK signaling pathway.Mitochondrial dysfunction in psychiatric and neurological diseases: cause(s), consequence(s), and implications of antioxidant therapy.Mechanisms of white matter damage in multiple sclerosis.
P2860
Genetic inactivation of mitochondria-targeted redox enzyme p66ShcA preserves neuronal viability and mitochondrial integrity in response to oxidative challenges.
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2012 nî lūn-bûn
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2012年の論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年論文
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2012年论文
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name
Genetic inactivation of mitoch ...... ponse to oxidative challenges.
@ast
Genetic inactivation of mitoch ...... ponse to oxidative challenges.
@en
type
label
Genetic inactivation of mitoch ...... ponse to oxidative challenges.
@ast
Genetic inactivation of mitoch ...... ponse to oxidative challenges.
@en
prefLabel
Genetic inactivation of mitoch ...... ponse to oxidative challenges.
@ast
Genetic inactivation of mitoch ...... ponse to oxidative challenges.
@en
P2093
P2860
P356
P1476
Genetic inactivation of mitoch ...... ponse to oxidative challenges.
@en
P2093
Dennis Bourdette
Michael Forte
P2860
P356
10.3389/FPHYS.2012.00285
P577
2012-07-20T00:00:00Z