Th-17 cell activation in response to high salt following acute kidney injury is associated with progressive fibrosis and attenuated by AT-1R antagonism.
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Immune Imbalances in Non-Alcoholic Fatty Liver Disease: From General Biomarkers and Neutrophils to Interleukin-17 Axis Activation and New Therapeutic TargetsHydrodynamic Isotonic Fluid Delivery Ameliorates Moderate-to-Severe Ischemia-Reperfusion Injury in Rat Kidneys.Human adipose stromal cell therapy improves survival and reduces renal inflammation and capillary rarefaction in acute kidney injury.Endothelial colony-forming cells ameliorate endothelial dysfunction via secreted factors following ischemia-reperfusion injury.Vitamin D deficiency contributes to vascular damage in sustained ischemic acute kidney injury.Elementary immunology: Na+ as a regulator of immunity.IL-17 mediates neutrophil infiltration and renal fibrosis following recovery from ischemia reperfusion: compensatory role of natural killer cells in athymic rats.Antithrombin III prevents progression of chronic kidney disease following experimental ischaemic-reperfusion injury.Th17 cells contribute to pulmonary fibrosis and inflammation during chronic kidney disease progression following acute ischemia.Early Phase Mast Cell Activation Determines the Chronic Outcome of Renal Ischemia-Reperfusion Injury.Increased Perfusion Pressure Drives Renal T-Cell Infiltration in the Dahl Salt-Sensitive Rat.CCR2 contributes to the recruitment of monocytes and leads to kidney inflammation and fibrosis development.The angiotensin II/AT1 receptor pathway mediates malaria-induced acute kidney injuryET receptor activation contributes to T cell accumulation in the kidney following ischemia-reperfusion injury
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Th-17 cell activation in response to high salt following acute kidney injury is associated with progressive fibrosis and attenuated by AT-1R antagonism.
description
2015 nî lūn-bûn
@nan
2015年の論文
@ja
2015年論文
@yue
2015年論文
@zh-hant
2015年論文
@zh-hk
2015年論文
@zh-mo
2015年論文
@zh-tw
2015年论文
@wuu
2015年论文
@zh
2015年论文
@zh-cn
name
Th-17 cell activation in respo ...... ttenuated by AT-1R antagonism.
@ast
Th-17 cell activation in respo ...... ttenuated by AT-1R antagonism.
@en
type
label
Th-17 cell activation in respo ...... ttenuated by AT-1R antagonism.
@ast
Th-17 cell activation in respo ...... ttenuated by AT-1R antagonism.
@en
prefLabel
Th-17 cell activation in respo ...... ttenuated by AT-1R antagonism.
@ast
Th-17 cell activation in respo ...... ttenuated by AT-1R antagonism.
@en
P2093
P2860
P356
P1433
P1476
Th-17 cell activation in respo ...... ttenuated by AT-1R antagonism.
@en
P2093
Carlie M Ivancic
David P Basile
Jason A Collett
Jaymin B Patel
Purvi Mehrotra
P2860
P2888
P304
P356
10.1038/KI.2015.200
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P577
2015-07-22T00:00:00Z