The α6 nicotinic acetylcholine receptor subunit influences ethanol-induced sedation.
about
The β2 nicotinic acetylcholine receptor subunit differentially influences ethanol behavioral effects in the mouse.mRNA and microRNA analysis reveals modulation of biochemical pathways related to addiction in the ventral tegmental area of methamphetamine self-administering ratsMechanisms and genetic factors underlying co-use of nicotine and alcohol or other drugs of abuse.Neuronal nicotinic acetylcholine receptors: common molecular substrates of nicotine and alcohol dependence.Potential substrates for nicotine and alcohol interactions: a focus on the mesocorticolimbic dopamine system.Nicotinic acetylcholine receptors containing α6 subunits contribute to alcohol reward-related behaviours.Nicotinic acetylcholine receptors containing the α4 subunit modulate alcohol rewardNo evidence of a role of the β4 subunit of the nicotinic acetylcholine receptor in alcohol-related behaviors.Nicotine Enhances the Hypnotic and Hypothermic Effects of Alcohol in the MouseExposure to nicotine increases nicotinic acetylcholine receptor density in the reward pathway and binge ethanol consumption in C57BL/6J adolescent female mice.Effect of Lowering the Dose of Varenicline on Alcohol Self-administration in Drinkers With Alcohol Use Disorders.Nicotinic acetylcholine receptors containing the α6 subunit contribute to ethanol activation of ventral tegmental area dopaminergic neurons.Genes and Alcohol Consumption: Studies with Mutant Mice.Neuronal nicotinic acetylcholine receptors are important targets for alcohol reward and dependence.Modulation of ethanol reward sensitivity by nicotinic acetylcholine receptors containing the α6 subunit.Neuronal Deletion of Kmt2a/Mll1 Histone Methyltransferase in Ventral Striatum is Associated with Defective Spike-Timing-Dependent Striatal Synaptic Plasticity, Altered Response to Dopaminergic Drugs, and Increased Anxiety.Varenicline modulates ethanol and saccharin consumption in adolescent male and female C57BL/6J mice.α6β2 nicotinic acetylcholine receptors influence locomotor activity and ethanol consumption.α6 subunit-containing nicotinic receptors mediate low-dose ethanol effects on ventral tegmental area neurons and ethanol reward.The influence of adolescent nicotine exposure on ethanol intake and brain gene expression.
P2860
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P2860
The α6 nicotinic acetylcholine receptor subunit influences ethanol-induced sedation.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年論文
@yue
2012年論文
@zh-hant
2012年論文
@zh-hk
2012年論文
@zh-mo
2012年論文
@zh-tw
2012年论文
@wuu
2012年论文
@zh
2012年论文
@zh-cn
name
The α6 nicotinic acetylcholine receptor subunit influences ethanol-induced sedation.
@ast
The α6 nicotinic acetylcholine receptor subunit influences ethanol-induced sedation.
@en
type
label
The α6 nicotinic acetylcholine receptor subunit influences ethanol-induced sedation.
@ast
The α6 nicotinic acetylcholine receptor subunit influences ethanol-induced sedation.
@en
prefLabel
The α6 nicotinic acetylcholine receptor subunit influences ethanol-induced sedation.
@ast
The α6 nicotinic acetylcholine receptor subunit influences ethanol-induced sedation.
@en
P2093
P2860
P1433
P1476
The α6 nicotinic acetylcholine receptor subunit influences ethanol-induced sedation.
@en
P2093
Jill H Miyamoto
Marissa A Ehringer
Nicole R Hoft
Ryan J Cox
P2860
P304
P356
10.1016/J.ALCOHOL.2012.03.001
P577
2012-05-07T00:00:00Z