Molecular characterization of Helicobacter pylori VacA induction of IL-8 in U937 cells reveals a prominent role for p38MAPK in activating transcription factor-2, cAMP response element binding protein, and NF-kappaB activation.
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Transcriptional mechanisms and protein kinase signaling mediate organic dust induction of IL-8 expression in lung epithelial and THP-1 cellsUse of VacA as a Vaccine AntigenAn Overview of Helicobacter pylori VacA Toxin BiologyHelicobacter pylori infection: An overview of bacterial virulence factors and pathogenesisHelicobacter pylori and interleukin-8 in gastric cancerThe Effect of Probiotic Plus Prebiotic Supplementation on the Tolerance and Efficacy of Helicobacter Pylori Eradication Quadruple Therapy: a Randomized Prospective Double Blind Controlled TrialHelicobacter pylori induces ERK-dependent formation of a phospho-c-Fos c-Jun activator protein-1 complex that causes apoptosis in macrophagesInterleukin-8 is the single most up-regulated gene in whole genome profiling of H. pylori exposed gastric epithelial cells.Gene expression profiling in gastric mucosa from Helicobacter pylori-infected and uninfected patients undergoing chronic superficial gastritisHeme oxygenase-1 dysregulates macrophage polarization and the immune response to Helicobacter pylori.Normalization of the levels of inflammatory molecules in Mycobacterium smegmatis-infected U937 cells by fibrate pretreatmentAssociation between Helicobacter pylori genotypes and severity of chronic gastritis, peptic ulcer disease and gastric mucosal interleukin-8 levels: Evidence from a study in the Middle EastA genome-wide small interfering RNA (siRNA) screen reveals nuclear factor-κB (NF-κB)-independent regulators of NOD2-induced interleukin-8 (IL-8) secretionHelicobacter bilis gamma-glutamyltranspeptidase enhances inflammatory stress response via oxidative stress in colon epithelial cells.Helicobacter pylori protein JHP0290 binds to multiple cell types and induces macrophage apoptosis via tumor necrosis factor (TNF)-dependent and independent pathwaysthe versatility of the Helicobacter pylori vacuolating cytotoxin vacA in signal transduction and molecular crosstalk.Transcription of Interleukin-8: How Altered Regulation Can Affect Cystic Fibrosis Lung Disease.Is helicobacter pylori infection a risk factor for coronary heart disease?Helicobacter pylori VacA-induced inhibition of GSK3 through the PI3K/Akt signaling pathwayEarly secreted antigenic target of 6 kDa (ESAT-6) protein of Mycobacterium tuberculosis induces interleukin-8 (IL-8) expression in lung epithelial cells via protein kinase signaling and reactive oxygen speciesHelicobacter pylori immunology and vaccines.Inflammation, immunity, and vaccines for Helicobacter pylori.Human and Helicobacter pylori Interactions Determine the Outcome of Gastric Diseases.Remodeling the host environment: modulation of the gastric epithelium by the Helicobacter pylori vacuolating toxin (VacA).Suppressive action of acetate on interleukin-8 production via tubulin-α acetylation.Hemoglobin receptor protein from Porphyromonas gingivalis induces interleukin-8 production in human gingival epithelial cells through stimulation of the mitogen-activated protein kinase and NF-κB signal transduction pathways.Pleiotropic cytotoxicity of VacA toxin in host cells and its impact on immunotherapyDiphenyleneiodonium inhibits the activation of mitogen-activated protein kinases and the expression of monocyte chemoattractant protein-1 in Helicobacter pylori-infected gastric epithelial AGS cells.Differential effects of multiplicity of infection on Helicobacter pylori-induced signaling pathways and interleukin-8 gene transcription.Helicobacter pylori VacA activates NF-kappaB in T cells via the classical but not alternative pathway.Helicobacter pylori-induced interleukin-12 p40 expression.A Tale of Two Toxins: Helicobacter Pylori CagA and VacA Modulate Host Pathways that Impact Disease.Specific NFkappaB subunit activation and kinetics of cytokine induction in adenoviral keratitis.Anti-inflammatory properties of gastric-derived Lactobacillus plantarum XB7 in the context of Helicobacter pylori infection.Helicobacter pylori-induced inhibition of vascular endothelial cell functions: a role for VacA-dependent nitric oxide reduction.Classical Swine Fever Virus Infection and Its NS4A Protein Expression Induce IL-8 Production through MAVS Signaling Pathway in Swine Umbilical Vein Endothelial Cells.
P2860
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P2860
Molecular characterization of Helicobacter pylori VacA induction of IL-8 in U937 cells reveals a prominent role for p38MAPK in activating transcription factor-2, cAMP response element binding protein, and NF-kappaB activation.
description
2008 nî lūn-bûn
@nan
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
2008年论文
@zh
2008年论文
@zh-cn
name
Molecular characterization of ...... ein, and NF-kappaB activation.
@ast
Molecular characterization of ...... ein, and NF-kappaB activation.
@en
type
label
Molecular characterization of ...... ein, and NF-kappaB activation.
@ast
Molecular characterization of ...... ein, and NF-kappaB activation.
@en
prefLabel
Molecular characterization of ...... ein, and NF-kappaB activation.
@ast
Molecular characterization of ...... ein, and NF-kappaB activation.
@en
P2093
P2860
P1476
Molecular characterization of ...... ein, and NF-kappaB activation.
@en
P2093
Asish K Mukhopadhyay
Eiki Yamasaki
Hajime Isomoto
Hisao Kurazono
Junzo Hisatsune
Kinnosuke Yahiro
Masaaki Nakayama
Naofumi Mukaida
P2860
P304
P356
10.4049/JIMMUNOL.180.7.5017
P407
P577
2008-04-01T00:00:00Z